MDD Psychological models Flashcards

1
Q

What is persistent depressive disorder (fka dysthymic)?

A

milder, chronic depression
for at least 2 years
without remission >2 months

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2
Q

MDD symptoms

4 types

A

emotional= sadness/ numbing +- anxiety, anger, agitation
cognitive= negative self-view, guilt/ self-blame, pessimism/ hopelessness
motivation= trouble starting, physical inertia, indecisiveness
somatic= appetite, sleep, fatigue, libido, hypochondriac
single episode/ recurrent

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3
Q

MDD diagnostic criteria

A
at least 5 symptoms
nearly daily
for at least 2 weeks
daily depressed mood (for most of the day) +//
daily diminished interest/ pleasure (")
may include psychotic features
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4
Q

Harre+ Finlay-Jones (1986)

emotional talk in history

A
accidie= boredom, disgust with fulfilling one's religious duties
melancholy= black bile, gloomy, clever men
mopishness= impaired senses, idleness in those born to work
20thC= MDD= accidie+ mopishness= linked to unfulfilling work
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5
Q

Kessler+ Bromet (2013)

A

higher lifetime prevalence in richer countries
median AOO= early adulthood
course= often chronic- recurrent
women lifetime risk roughly double mens (worldwide)
separated/ divorced have higher rates of MDD than married
prevalence decreases with age

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6
Q

Behavioural aetiology

A

Mair+ Seligman (1976)= learned helplessness (perceived uncontrollability of aversive stimuli)
Support:
Hiroto (1974)= non-depressed stopped trying to stop a noise when previously unsuccessful
Lewisohn (1979)= reduced rewards
Constantino (2012)= negative spiral of reduced social rewards

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7
Q

Behavioural Rx

A
operant conditioning (test uncontrollability)
classical (learn non-depressive association to stimuli)
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8
Q

Behavioural evaluation

A

lab created motivational deficits seen in depression

behavioural Rx rarely used alone

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9
Q

Cognitive aetiology

Who created the attributional reformulation of learned helplessness?

A

Abramson, Seligman and Teasdale (1978)
internal (personal failing)
stable (will persist over time)
global (will persist over different situations)

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10
Q

Depressive attributional style (Beck)

Cognitive aetiology

A

Negative cognitive triad
-pessimism of self, world+ future
(stems from our childhood/ schemata/ how we evaluate)

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11
Q
Schemata
what is it?
what's it for?
what's it triggered by?
what does it determine?
A

unspoken/ inflexible assumptions/ beliefs
for screening/ discriminating/ processing stimuli
triggered by negative life events
determine content of cognitions+ affective processes

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12
Q

CBT 4 stages

A

education= teach relationships between cognition, emotion and behaviour

behavioural= reinforce/ reality test, pleasant event scheduling (increase engagement/ activity)

cognitive rehearsal= develop coping strategies (detecting thoughts/ bias) and correct dysfunctional thinking

behavioural hypothesis testing= test validity of negative assumptions

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13
Q

Carter+ Garber (2011) longitudinal study predicting MDD onset

A

cognitions ?cause/consequence of MDD
predicted by high negative cognitions (even with low interpersonal stress) or increased stress (regardless of cognitions)

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14
Q

Hiroto (1974)

A

non-depressed volunteers stopped trying to stop a loud noise when their past efforts were unsuccessful
(support for learned helplessness theory)

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15
Q

Lewishon (1979) and Constantino (2012)

A

further support for learned helplessness theory (reduced rewards)

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16
Q

Cognitive aetiology (5)

A
Negative cognitive triad
Learned helplessness
Schemata
Cognitive biases
Negative automatic thoughts (steady train of unpleasant ideas pop into head)
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17
Q

Moore+ Fresco (2012)

A

depressed people have less illusion of control

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18
Q

Roshanhaei-Moghaddam (2014)

A

MDD patients show relatively similar effects for CBT+ psychopharmacology Rx

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19
Q

Freud’s psychodynamic aetiology of MDD (1917)

A

defence mounted by the ego against the intra-psychic conflict
reaction to loss (real/symbolic/ imagined)- regression to oral stage of dependency
unconscious anger turned upon self- despair/ self-hatred

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20
Q

Blatt+ Homann (1992) core assumptions of modern psychodynamic theories of MDD

A

rooted in early losses
wound reactivated by a recent blow
regression to stage of help/hopelessness
ambivalent feelings= central unconscious conflict
overly seek self-esteem from others
ensuing dependency has a relationship function

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21
Q

Psychodynamic Rx= traditional psychoanalysis

A
uncover childhood roots of MDD
explore ambivalent feelings towards lost object
-free association
-dream analysis
-analysis of resistance/ transferance
IPT
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22
Q

Klerman (2004) IPT

A

Interpersonal psychotherapy (IPT) focuses on

  • present problems> past
  • MDD use in relationships
  • core problem+ possible solutions
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23
Q

Psychodynamic evaluation

Bornstein (1992)

A

some depressed people= highly dependent

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24
Q

Psychodynamic evaluation

Willoughby (2013)

A

poor parenting is a risk factor for MDD

consistent with attachment theory (Ainsworth+Bowlby, 1991)

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25
Psychodynamic evaluation- Rx depends on accepting what?
the internal logic of the psychodynamic theory
26
Psychodynamic evaluation | Lemmens et al (2011)
cognitive therapy+ IPT are successful for acute MDD | no consensus about one outperforming the other
27
Social aetiology | Holmes+ Rahe (1967)
recent life events
28
Stegena (2012): recent life events...
...carry the largest risk of MDD onset in mid-life middle aged= more vulnerable - increased responsibilities/ social ties? -hormonal (menopause increases vulnerability)
29
Brown+ Harris (1978) | Social aetiology
vulnerability in women - unsupported relationship with spouse - no job outside house - 3+ young children - no serious religious committment - loss of mother <11y
30
Social Rx
IPT
31
Whisman+ Schonbrun (2010)
couples/ marital therapy as effective as individual CT, IPT or psychopharmacology in reducing depression when discord is a problem
32
Social evaluation
attention on social/ situational factors | can't predict who becomes depressed
33
Perris (1991) inborn capacity of gathering/ processing information
genetics post-natal influences biochemical/ neurophysiological factors (eg ageing)
34
Perris (1991) active construction of the world development of cognitive schemata emergence of self identity
genetics, post natal, biochemical | culture, attachment, parental rearing, environmental
35
Perris (1991) basic dysfunctional assumptions individual vulnerability personality characteristics
culture, attachment, rearing, environment systematic perceptual/ processing errors stressful life events with idiosyncratic meaning
36
Perris (1991) | psychopathology- MDD
stressful life events with idiosyncratic meaning
37
Perris (1991) vulnerability reflects relationship between
genetic predisposition and one's history
38
Perris (1991) people are vulnerable to certain events which wouldn't affect others- depends on
personality, cognition, emotional organisation
39
Perris (1991) core of individual vulnerability is
dysfunctional self-schemata
40
Perris (1991) time dimension
``` chronological time (growth/age differences) social time (eg puberty, marriage, retirement) historical time (economic climate) ```
41
Perris (1991) earlier developmental issues have a higher risk of worsening because
biased self-verification process
42
Perris (1991) ADD (low what?) in kids correlates with depressive illness
low MAO | also increases vulnerability in depression-prone adults
43
Bowlby (cited in Perris (1991)) what determines likelihood of certain cognitive maps (working models/ self schemata)?
genetics
44
Perris (1991) earlier onset of MDD due to
conflictive home environments 1+ affected parent (heredofamilial/ biological) dysfunctional rearing from mother (dad= not significant) (psychosocial)
45
Perris (1991) abnormal biohumoral changes are state dependent or trait dependent? what do they interact with?
``` state dependent (often interact with cognitive processes) ```
46
Perris (1991) what % of patients don't respond enough to a single Rx (drugs/ CBT)?
30-40%
47
Perris (1991) individualised/ integrated Rx program needs
constant reappraisal/ self-evaluation of clinician/ therapeutic strategies
48
Stegenga (2012) different vulnerability to life events according to
age | implications for identifying/ preventing
49
Maciejewski (2001) life events triples likelihood of MDD in
women
50
Stegenga (2012) (dis)agrees with stress-vulnerability model?
agrees | - lower stress threshold
51
Stegenga (2012) methodology
self-reported questionnaire on major life events DV= MDD diagnosis in preceding 6 months wide CIs in men (caution in interpreting)
52
Stegenga (2012) sum of individual life events= same as combined effects of them?
false | combined> sum of individual
53
Abramson, Seligman+ Teasdale (1978) the old learned helplessness theory didn't distinguish between
universal/ personal general/ specific acute/ chronic
54
Abramson, Seligman+ Teasdale (1978) LHH is
learning that outcome= uncontrollable- cognitive deficit- harder to later learn that responses produce that outcome
55
Alloy+ Abramson: non-contingency is harder to perceive when
one is winning
56
Clark+ Clark (1939) a major determinant of attitudes to oneself is
comparison with others
57
Abramson (1977) self esteem only lowers when
it's just you/ personal helplessness
58
Abramson, Seligman+ Teasdale (1978) Rx
reduce catastrophising of uncontrollable outcomes | make people feel more in control
59
Harre+ Finlay-Jones (1986) people with undiagnosed DDs are
socially isolated unemployed/ menial jobs unable to keep busy- anxious/ depressed
60
Harre+ Finlay-Jones (1986) historically depression was seen as
laziness- behavioural/ emotional causes- misery negligence of moral order- in boring jobs- spiritual vice St Paul- good/ bad form of depression- God Boredom- apathy, can cause fear/ misery- misdiagnosis of DD
61
Cognitive biases
logic errors in assessing eg selective abstraction
62
Selective abstraction
focusing on negative detail