Lung assessment Flashcards
Left axillary lung
U and L lobe seperation, posteriorly 3rd rib anteriorly 6th
R axillary lung
RUL runs posteriorly 3rd rib to anteriorly 6th rib but RML is mid axillary ~5th rib
Broncial lung sounds
Throat, high pitch, harsh amplitude short inspiration long exp
Bronchialvesicular sounds
Moderate pitch mixed quality = insp and exp length
Vesicular
Low pitch breezy quality soft amplitude Long insp SHORT exp (peripheral lungs
Fine crackles
Short high pitched popping during insp, not cleared with cough
Pneumonia, CHF.
If EARLY in insp COPD
Air opening small deflated passages coated with exudate
Course crackles
Low-pitch bubbling moist
Early insp to early exp
Air hitting secretions in large bronchi and trachea
Pneumonia, PE, fibrosis
Pleural rub
Low pitched dry grating
Inflamed pleural surfaces rubbing
Pleuritis
Sibilant wheezes
High pitched and musical
Expiration mainly
Air through constricted passages (swelling, secretions, tumour)
Asthma or emphysema
Sonorous wheezes
Low pitched snoring or moaning mainly during expiration
Air through constricted passages
Bronchitis, single obstructions
Resonance
Part air part solid
Normal lung
Hyper resonance
Mostly air
Lung with emphysema
Tympany
Over air, puffed out cheek or gastic bubble, pneumo
Dullness
Solid tissue
Diaphragm pleural effusion, liver
Flatness
Dense tissue like muscle bone sternum
Bronchophony test
Lung consolidation
Say 99 while auscultating posterior thorax
Soft and muffled normally, louder and more easily understood over consolidation
Egophony
Eeee outloud listening to lung fields
Sound like AAAA over consolidation
Good for pneumonia and effusion
Tactile Fremitus
Ulnar surface of hands and say 99, fremitus increased over consolidation
Whispered pectoriloquy
Pt speaking clearly if over consolidation
Get pt to whisper 1-2-3
S1
First heard sound, produced by AV closing at beginning of systole.
Intensity of S1 is dependant on
Position of mitral valve at start if systole, structure of valve leaflets, how quicky pressure rises in ventricles
S1 clinical (just the) tip
Normal S1 variations heard at apex of heart.
S1 softer at base and louder at apex
Accenuated S1
Louder than S2, occurs when mitral valve is wide open and closes quickly from:
Hyperkinetic states from increased blood velocity (fever, anemia, hyperthyroidism)
or Mitral stenosis in which leaflets are mobile but increased ventricular pressure needed to close the valve
Diminshed S1
Mitral valve not fully open from
Delayed conduction from atria to ventricles (1st degree block) - mitral starts drifting closed before closing
Mitral insufficiency (extreme calcification limits mobility)
Delayed/diminished ventricular contraction from forceful atrial contraction or into a noncompliant ventricle (severe pulmonary or systemic HTN)
Split S1
L and R ventricles contract at different times
BBB
Ventricular ectopy in which impulse starts in one ventricle, contracting it first and spreading into second
Varying S1
Mitral valve is in different position when contraction occurs
A and V bating independendtly (a-fib, AV dissociation, blocks)
S2 sounds
Closure of aortic and pulmonary valves. Closure of pulmonic is delayed by inspiration, resulting in split S2