Long-term Memory Flashcards

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1
Q

declarative memory

A

the conscious recall of events and detail

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2
Q

subdivisions of declarative memory

A

semantic memory
episodic memory

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3
Q

semantic memory

A

knowledge about the world

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4
Q

episodic memory

A

recollection of content and context of own events

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5
Q

what is semanticisation?

A

when some degree of semantic memory is required for episodic memory

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6
Q

non-declarative memory

A

unconscious recall of movement sequences which cannot be explicitly retrieved

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7
Q

subdivisions of non-declarative memory

A

procedural memory
priming
conditioning

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8
Q

procedural memory

A

performing actions and skills over gradual learning

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9
Q

priming

A

prior encounters with similar stimuli can influence the processing of later stimuli

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10
Q

semantic dementia

A

degeneration affecting the anterior temporal lobe, resulting in impaired semantic memory

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11
Q

impaired episodic memory

A

semantic knowledge is retained but memories cannot be related to real life (not episodic)

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12
Q

how is explicit and implicit memory different in encoding?

A
  • explicit information is processed top-down as it can be reorganised
  • implicit information is bottom-up as it is encoded in the same way it was perceived
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13
Q

how is explicit and implicit memory different in retrieval?

A
  • explicit memory requires conscious recollection of prior experience
  • implicit memory is not dependent on conscious memory that experience is being used
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14
Q

causes of amnesia

A
  • acute virus infection
  • PD
  • brain resection as epilepsy treatment
  • physical accident
  • korsakoff syndrome
  • psychological disorders
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15
Q

what type of amnesia did patient HM experience?

A

anterograde amnesia

caused by bilateral medial-temporal lobe resection

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16
Q

anterograde amnesia

A

forgets everything new after a few minutes

17
Q

retrograde amnesia

A

loss of memory for information learned in the past

18
Q

in anterograde amnesia, _____ ______ is still possible but there is no ________ _______

A

implicit learning

explicit memory

19
Q

disconnecting implicit memory

A

patient JK had impaired implicit memory after developing PD

20
Q

how was patient JK impaired?

A

could not perform usual procedural tasks but could still recall explicit events (declarative)

21
Q

neural circuits for explicit memory

A
  • medial temporal region
    - hippocampus
    • amygdala
    • entorhinal cortex
    • parahippocampal cortex
  • frontal cortex (dorso and ventro)
  • connections between fronto-temporal regions
22
Q

neural circuits for implicit memory

A
  • basal ganglia
  • ventral thalamus
  • substantia nigra
  • premotor cortex
23
Q

what is dementia?

A

a neurological disorder caused by progressive cell death

24
Q

how is dementia characterised?

A

by memory and another impairment:
- aphasia (language)
- apraxia (motor memory)
- agnosia (sensory memory)
- executive function/abstract thinking

25
Q

degenerative dementia

A

have a degree of genetic cause, from cortical (AD) and subcortical (PD) areas

26
Q

nondegenerative dementia

A

heterogenous group with diverse origin, KS, vascular dementias, infectious dementia

27
Q

memory impairments in AD

A
  • difficulty learning or retaining information
  • information retrieval deficits
  • episodic memory impairments
  • declarative memory more than procedural
28
Q

language impairments in AD

A
  • list-generation deficits
  • word finding and verbal fluency difficulties
  • less complex sentence structure
  • language and auditory comprehension intact
  • executive function impairments
29
Q

cortical dementia in AD

A
  • cell loss in the entorhinal cortex explains early memory problems
  • limbic cortex, inferior temporal cortex, and posterior parietal cortex
  • spared primary sensory and motor areas
30
Q

how can consolidation be evidenced?

A

in forgetting curves

where memory traces are most vulnerable shortly after learning

31
Q

theories on forgetting

A
  • decay (fading of memory trace) is dependent on the time elapsed
  • interference (memory traces are disrupted) occurs as a result of interpolated events
32
Q

cognitive neuroscience has found two stages to memory consolidation:

A
  1. temporary memory storage in the hippocampus to create traces
  2. transferred to cortex, which involves distributing involvement to several areas
33
Q

consolidation and sleep

A

fewer memories formed (during sleep) led to less disruption of the consolidation process