Lesson 3.1 - Panc Path (part 1) Flashcards

1
Q

GDA vs CBD location - which is more anterior

A

GDA more anterior

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2
Q

when might serum amylase increase? (4)

A
  • pancreatitis
  • obstruction intestinal
  • pseudocyst
  • peptic ulcer disease
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3
Q

when does serum amylase decrease (2)

A

hepatitis

cirrhosis

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4
Q

in episodes of acute pancreatitis, which stays increased longer urine or serum amylase?

A

urine amylase

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5
Q

what stays elevated the longest?

A

serum lipase

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6
Q

elevated serum lipase assoc with (6)

A
  • pancreatitis
  • obstruction panc duct
  • carcinoma
  • cirrhosis
  • acute cholecystitis
  • severe renal disease
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7
Q

what cases indicate elevated glucose

A
  • diabetes mellitus
  • chronic liver disease
  • over activity of endocrine glands
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8
Q

glucose decreases with

A

tumours of islets of langerhands in the pancreas

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9
Q

t/f increased echogenicity in the pac is not always a result of fatty replacement

A

true

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10
Q

panc normal variant size (1)

A

pseudomass - larger head bulging to right of GDA

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11
Q

what area of the pancreas (and size) may indicate acute pancreatitis

A

if the body exceeds 3 cm AP

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12
Q

what size should the panc duct measure

A

3 mm or less

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13
Q

congenital anomalies (3)

A
  • pancreatic divisum
  • annular pancreas
  • partial agenesis
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14
Q

what is the most common pancreas variant

A

pancreatic divisum

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15
Q

describe annular panc

A

duodenum encircled by a ring of pancreatic tissue - may constrict duo

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16
Q

when do we use doppler (3)

A
  • any mass
  • suspected pancreatitis
  • if splenic vein does not appear anechoic and is over distended
17
Q

are pancreatic enzymes increased or decreased with acute pancreatitis

18
Q

what are the causes/risk factors of acute pancreatitis?

A

gallstones (40%)

alcoholism (40%)

19
Q

what should you do if you suspect acute pancreatitis? (3)

A

evaluate the biliary tree for gallstones, choledochlithiasis and obstruction

20
Q

other causes of acute pancreatitis (9)

A

choledocholithiasis

  • biliary sludge
  • neoplasm
  • infection
  • toxins
  • drugs
  • genetic
  • traumatic
  • iatrogenic factors
21
Q

steps for diagnosing acute pancreatitis

A

amylase and lipase elevated on blood test*

pt sent for imaging (US/ CECT)

22
Q

sono findings of pancreatitis

A

-panc tissue hypoechoic / enlarged
-surrounding/
potential spaces (lesser sac, anterior pararenal spaces, trans mesacolon)
-biliary system (GB stones, choledocalithasis, dilated ducts)

23
Q

the most common and useful finding to diagnose pancreatitis

A

pac assoc inflammation:
Hypoechoic or anechoic collections that conform to the retroperitoneal space
Ascites

24
Q

*local complications of pancreatitis (4)

A

fluid collection (40%)
pseudocysts (fluid collection >6 wks)
abscess
necrosis

25
vascular complications associated with pancreatitis (3)
hemorrhage venous thrombosis pseudoaneurysms
26
define pseudocyst and why isn't it a true cyst
fluid collection persists > 6wks | not true cyst /cystic neoplasm because no epithelial lining
27
t/f pseudocyst comprise most of the cystic lesions of the pancreas
true (more common in chronic pancreatitis)
28
How must we confirm its a pseudocyst (2)
clinical history and/or evidence of acute/chronic pancreatitis
29
pseudocyst sono appearance
range in appearance | - purely cystic or mural irregularity, septations , interal echo debris
30
what might a pseudocyst look like
cystic neoplasm
31
significant pancreatic necrosis defined as
parenchyma >3cm AP or | involving more than 30% of the panc
32
how is necrosis treated
antibiotics
33
t/f necrosis can be diagnosed with US
``` false CECT (contrast enhanced CT) ```
34
main modalities in diagnosing acute pancreatitis
CECT | abdo US
35
US ROLE! (3)
- detect gallstones, choledocholithiasis and/or signs of obstruction - diagnose acute pancreatitis - treatment: guidance in drainage of infected pseudocyst