Lecture 8, 9 And 10 Flashcards

1
Q

What is a tendon

A

Organization of Collagen fibres
• Connects muscle to bone
• Transfer force from muscles
into skeletal system
• Excellent tensile properties

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2
Q

Enthesis

A

• Junction between a
tendon and a bone
• Fibrocartilage

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3
Q

Myotendinous
Junction (MTJ)

A

• Connection between
tendon and muscle
• Susceptible for
injury

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4
Q

Tendon
Stress-Strain Curve

A

• Relationship between
stress and deformation
of tendons is the same
as for ligaments

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5
Q

Adaptation to Training in tendons

A

Tendons adapt to training by increasing cross-sectional area

Tendon loading every 2-3 days; problematic to jumping sport not too much time for recovery; problematic to jumping sport not too much time for recovery

Compared to muscles, it takes longer time to gain tendon strength

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6
Q

Tendon Injury Types

A

Overuse tendon injuries
• Enthesopathy
• Tendinopathy
Acute tendon injuries
• Direct trauma
• Rupture

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7
Q

Enthesopathy

A

• Injury or disorder affecting the enthesis
• Cause/mechanism
• Overuse (most common); repetitive loading
• Trauma (direct blow)
• Characterized by inflammation,
degeneration, or calcification of the
attachment point
• Pain and dysfunction

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8
Q

Enthesopathy:
Diagnosis and
Treatment

A

• History
• Inspection & Palpation
• Imaging (rarely used): US, MRI
Treatment
• Rest from offending activity
• Pain control: Ice, NSAIDS
• Orthoses
• Physiotherapy: progressive strength training
• Mild cases (4-6 weeks); moderate to severe
cases (3-6 months)

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9
Q

mechanism of tendinopathy

A

Repetitive tensile (or compressive) loading (e.g.,
sprinting, jumping, changing direction) → repetitive
microtraumas
• Inadequate recovery between loadings

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10
Q

Tendon Pathology:
Cook–Purdam Model

A

• Reactive tendinopathy
• Non-inflammatory, structural
changes & thickening of stressed
tendon area
• Tendon disrepair
• Worsening tendon pathology,
tendon structure becomes
disorganized
• Degenerative tendinopathy
• Chronic stage

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11
Q

when athlete does jump training and there is insufficient recivery it results in tendinopathy true or false

A

True

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12
Q

healthy tendon is highly organized and when we start to feel pain it becomes disorganized , the structure does not act like a healthy tendon anymore true or false

A

True

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13
Q

Intrinsic risk factors for tendinopathy

A

• Older age
• Male sex
• Menopause
• Genetics
• Systemic conditions
• Medications
• Biomechanics
• Previous injury

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14
Q

Extrinsic risk factors in tendinopathy

A

• Training load
• Spikes in loads
• Periods of deconditioning
• Biomechanical change

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15
Q

What does biomechanics mean when referring to intrinsic risk factors to tendinopathy

A

biomechanists refer to how you land, directional changes and landing

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16
Q

What is bio mechanical change when referring to extrinsic risk factors with tendinopathy

A

biomechanical change; change in movement patterns, different tendons engaged and leads to tendinopathy

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17
Q

Diagnosis of tendon injury

A

• History: Symptoms often progress
• First pain after exercise
• Then pain at the start of
an activity
• Finally pain both during
and after activity
• Physical Examination
• Palpation → tenderness
• Imaging: US, MRI

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18
Q

Management of tendinopathy

A

Education of patients
Load monitoring
Pain monitoring
Exercise based progressive
rehabilitation program

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19
Q

What is the stages of exercise based rehabilitation programs for lower limb tendinopathy

A

Stage 1 Isometric exercises: every day, multiple times a day (relieves pain)

Stage 2 Isotonic & Heavy slow resistance exercises, alternate days
(improves tendon stiffness and strength), continue doing isometric exercises

Stage 3 Increase in speed and energy storage exercises: single jumps
continue doing isometric and isotonic exercises

Stage 4 Energy storage and release & Sport specific exercises: repeated jumps
(This replaces stage 3) direction changes, continue doing isometric and isotonic exercises

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20
Q

Rotator Cuff
Tendinopathy
And associated pain

A

-Subacromial pain syndrome
(SPS)
-Rotator-cuff related shoulder
pain

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21
Q

Rotator Cuff Tendinopathy Diagnosis

A

• History
• Inspection, palpation, ROM, Pain provocation tests
• Imaging
• Treatment
• Progressive exercise therapy 6-12 weeks
• Surgery and rehabilitation 6-7 months

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22
Q

Other Treatments for tendinopathy

A

• Shock wave therapy, laser, and
ultrasound
• Medications
• Injectable therapies
• Passive treatments
• Experimental treatments
• Surgery

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23
Q

prevention of tendinopathy

A

• Progressive training
• Enough rest
• Education of athletes and coaches
• Correct movement technique
• Offseason continue strength training

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24
Q

Main reason for tendon rupture

A

tendon rupture Main reason is tendinopathy and the structural changes associated, common
In older adults

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25
Q

Tendon
Rupture

A

• Acute rupture of a normal and healthy tendon is rare
• Commonly occur in athletes and recreational exercisers aged
30-50 years

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26
Q

True or false within Tendon
Rupture it is caused by Eccentric force generation in Mid-tendon area causing a Partial or Complete
rupture

A

True

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27
Q

Achillies tendon rupture Mechanism

A

• Strong contracture of the
lower leg musculature,
with simultaneous
extension
• ECCENTRIC LOADING of
the tendon

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28
Q

Diagnosis TENDON RUPTURE

A

Acute, intense pain
Audible ‘SNAP’
Reduced power in plantar flexion
‘Gap’ in the tendon tissue
Bruise and swelling
Ultrasound/MRI

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29
Q

Treatment and Rehabilitation for tendon rupture

A

Conservative (exercise program interventions) vs. Surgical repair
(end-to-end suture)
Cast
Rehabilitation
Return to Sport

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30
Q

Patellar Tendon Rupture: Treatment

A

• Partial rupture
• Conservative treatment: Cast 4 weeks, physiotherapy &
progressive training
• Surgery
• RTS 4-6 weeks
• Complete rupture
• Surgery
• End-to-end repair or Transsosseus repair
• Tendon reconstruction (severe degenerative tissue changes)
• Post-operative rehabilitation
• Running after 6-15 months
• RTS after 8-18 months

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31
Q

prevention of tendon rupture

A

• Strengthening exercises when approached with tendon tightness
• Heavy lifting (healthy tendons need heavy lifting)
• stay active
• Proper recovery

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32
Q

Prevention of shoulder
problems in overhead sports: caused us to use what program

A

OSTRC
Shoulder Program
28% decrease in problems

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33
Q

Exercises to treat tendinopathy (2)

A

Single jump, load tendons but don’t release energy ( increase in speed and energy storage)
And then
Repeated jumps, energy storage and release as well as sport specific exercises

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34
Q

Muscle function

A

Generate power

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35
Q

Muscle actions

A
  • isometric; during contraction, muscle length does not change, static muscle generates force
  • isotonic; tension,contraction and change in muscle length
  • concentric; shortening of muscle; tension is present
  • eccentric; lengthening of muscle when muscle is extended
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36
Q

Muscle adaptation to training

A

-rapid response to training, changes in strength within a few weeks
- neural factors (early stages), changes are due to new role adaptation improvements in muscle strength to neuromuscular adaptation
- muscular factors (prolonged training)
- muscle fibres increase their cross sectional area (hypertrophy)

37
Q

Muscle injury types

A
  1. Direct muscle injury;contusion and laceration
  2. Indirect muscle injury disorder; muscle strain, fatigue- induced muscle disorder, DOMS, Neuromuscular muscle disorders
  3. Other; muscle cramps, chronic compartment syndrome
38
Q

What is muscle contusion common in

A

Contact sport, caused by a direct blow

39
Q

Muscle contusion

A

Muscle bruise (hematoma)
- caused by external force
- contact sports, team ball sports
- most common site is the quadriceps
- mild, moderate or severe

40
Q

Muscle contusion diagnosis and treatment

A
  • history (mechanism), physical examination (introspection, palpitation, function tests (active ROM)
  • Imaging
  • Progressive physiotherapy; gentle active and passive pain free muscle stretching, stretching, isometric strength, functional and sport specific movements once ROM achieved
  • RTS for mild contusion (5-7 days), RTS for moderate and severe (4-6 weeks)
41
Q

Compression is important in the first few days with having a muscle contusion TRUE OR FALSE

42
Q

Muscle contusion complications

A
  • acute compartment syndrome
  • myostitis ossificans- formation of bone inside muscle tissue, rare
  • muscle fibrosis- scar tissue not functioning properly limited range of motion due to improper rehab
  • chronic pain and weakness- improper rehab
  • recurrent injury
43
Q

Muscle strain

A
  • tensile forces
  • usually close to myotendinous junction
  • hamstrings,quads, gastrocnemius
  • pop,bump,swelling
  • pain on active contraction and passive stretch reduced contraction strength, decreased ROM and loss of function
44
Q

Myotendinous junction

A
  • common injury site
  • region where muscle fibres fibres connect to tendon
  • transfer of contraction force from muscle to tendon and skeletal system
  • high stress during jumping and sprinting
45
Q

Clinical grading of muscle strains

A
  1. Mild, “few fibre” injury, minimal loss of strength and motion
  2. Moderate, tissue damage decreased ability to contract and decreased ROM
  3. Severe complete tear, complete loss of muscle function
46
Q

Two types of hamstring rupture

A

Type 1- Sprinting related hamstring strain (bicep femoris)
Type 2- Stretching-related hamstring strain (semimembranosus)

47
Q

Type 1 diagnosis

A

-history, physical examination; mechanism, palpation and spinal examination
- imaging MRI

48
Q

Type 2 diagnosis

A
  • history and physical examination mechanism, palpitation and spinal examination
    -MRI
49
Q

Neuromuscular muscle disorder

A
  • spinal or spinal nerve-related
  • neuromuscular end plate-related
50
Q

Spinal or spinal nerve related

A
  • nerve damage in the spine, nerve root plexus lead to increased muscle tone, pain, tinging, numbness, weakness
51
Q

Neuromuscular endplate related

A
  • muscle fatigue leads to dysfunctional neuromuscular control leading to increased alpha motor neuron activity leading to increased muscle tone, over contraction
52
Q

Assessment of hamstring injury should include a thorough

A
  • spinal examination, lumbar spine, pelvis, sacrum
  • neural testing
  • biomechanical evaluation
53
Q

Muscle injury healing phases

A
  1. Destructive phase (Hemostasis and inflammation)
  2. Repair phase (proliferation)
  3. Maturation phase (remodelling)
54
Q

Muscle strain rehab if a patient can do jogging 2 days after injury can return to sport after

55
Q

Muscle strength rehab if it takes more than 5 days return to sport after a

56
Q

Muscle strain rehab

A
  • mobilization
  • progressive strengthening
  • functional exercises (running programs)
  • other body region exercises
57
Q

Prevention of hamstring strains

A

Stretching
Nordic eccentric exercise

58
Q

Hip and core exercise programs prevents running related overuse injuries T or F

59
Q

Run RCT aim

A
  • aim investigate which intervention was effective in prevention of running related injuries (hip and core exercise program)
60
Q

Nordic hamstring exercise

A
  • used in high risk sports
61
Q

DOMS is related to what structure

62
Q

Overuse injury types

A

Chronic injuries that develop over time due to repetitive strain

63
Q

Overuse bone injury

A

Stress fractures, apophysitis, ostetits

64
Q

Overuse tendon injury

A
  • tendinopathy
65
Q

Overuse joint/ligament injury

A
  • labrum overuse
  • ligament degeneration
  • synovitis
66
Q

Overuse muscle/fascia injuries

A
  • DOMS
  • Fasciitis
  • chronic compartment syndrome
67
Q

Low back pain in adult athlete prevalence

A

Lifetime prevalence in athletes can lead to time loss from sport and reduced quality of life

68
Q

adult athletes non specific low back pain

A
  • caused by intrinsic (spine structure, past injury) and extrinsic (technique and stress) factors
69
Q

Degenerative disc disease

A
  • symptoms; stiffness, reduced ROM, radiating pain
  • treatment- physiotherapy focusing on core strength and flexibility (conservative treatment)
70
Q

Avulsion

A
  • tendon or ligament pulls a piece of bone off attachment point
  • common sites- ASIS, ischial tuberosity
  • causes- common in high risk sports
  • symptoms- sudden pain, popping sound, swelling
  • treatment- conservative (physiotherapy), or surgical
71
Q

Ankle sprain (3 types)

A
  1. Lateral ankle sprain
  2. Medial ankle sprain
  3. High ankle sprain
72
Q

Lateral Ankle Sprain:

A

Mechanism: Excessive inversion or supination.
Involved Structures: ATF, PTF, CF ligaments.
Grades:
Grade I: Partial rupture of one ligament.
Grade II: Complete rupture of one ligament or partial rupture of two.
Grade III: Complete rupture of two or more ligaments.
Treatment: Bracing, taping, progressive exercise.

73
Q

Medial Ankle Sprain:

A

Mechanism: Excessive eversion.
Involved Structures: Deltoid ligament, sometimes malleolar fractures or syndesmosis.
Treatment: Bracing, arch support, functional treatment, physiotherapy (longer recovery compared to lateral sprains).

74
Q

High Ankle Sprain (Syndesmosis):

A

Mechanism: Forced external rotation. Excessive
Involved Structures: Syndesmotic ligaments connecting tibia and fibula.
Treatment: Walking boot (partial rupture, conservative treatment, 2 weeks) or surgery for severe cases.(complete rupture )
Complications: High re-injury rate, risk of chronic instability, and potential arthritis.

75
Q

Difference in Back Pain between Youth and Adult Athletes

A

Youth: More likely due to growth plate injuries, apophysitis.
Adults: Degenerative conditions (e.g., DDD), higher risk of chronic pain.

76
Q

characteristics of over use injury Gradual Onset:

A

Athletes may not notice symptoms initially; the injury develops over time.
Chronic in Nature: These injuries are persistent and may worsen without intervention.
Alternative Names:
Chronic injury
Repetitive strain injury (RSI)
Cumulative trauma disorder
Sports disease

77
Q

Growth plate= growth cartiliage T or F

78
Q

Epiphysis located vs where apophysis located and are they both resistant to receptive loading

A

At the end of the bone

Site where tendons/ligaments attach the bone

No they are less resistant to repetitive loading leading to increased risk of injury

79
Q

Apophysitis definition

A

Apophysitis: A traction injury that occurs when there is repeated stress on the apophysis (the growth plate at the site where tendons and ligaments attach to bones).

80
Q

Apophysitis cause and symptoms

A

Causes: Repetitive motion or stress on the growth plates during periods of rapid growth.
Symptoms: Pain, swelling, and tenderness at the affected site.

81
Q

Common Types of Apophysitis:

A

Sever’s Disease:
Location: At the heel bone (calcaneus).
Age group: Common in children aged 8-14 years.
Symptoms: Pain in the heel, especially during running or jumping.

Osgood-Schlatter Disease:
Location: At the tibial tuberosity (just below the knee cap).
Age group: Typically affects children aged 10-15 years.
Symptoms: Knee pain, particularly during activities that involve running, jumping, or kneeling.

Little League Elbow:
Location: At the medial epicondyle of the elbow.
Age group: Affects children between 8-15 years, often baseball players.
Symptoms: Pain on the inside of the elbow, especially during throwing motions

82
Q

treatment of synovitis of knee is load modification, physiotherapy and nsaids treatment of chondromalacia patella is correcting valgus movement true or false

83
Q

Plantar Fasciitis: def, cause, symptoms

A

Definition: Inflammation of the plantar fascia,
Causes: Overuse or repetitive stress (e.g., running, prolonged standing).
Improper footwear lacking proper arch support. Biomechanical issues like flat feet or high arches.
Symptoms: Heel pain, especially with the first steps in the morning or after periods of inactivity.
Treatment: Conservative Treatment: Rest and load reduction. Proper footwear or orthotics for support.
Stretching exercises

84
Q

Risk Factors for Overuse Injury: intrinsic

A

Intrinsic Factors:
Previous Injury
Malalignment: Structural imbalances in the body.
Leg Length Discrepancy
Muscle Imbalance or Weakness
Lack of Flexibility
Sex: Certain injuries may affect men and women differently.
Body Composition
Genetic Factors

85
Q

Risk Factors for Overuse Injury: Extrinsic Factors:

A

Extrinsic Factors:
Training Load Errors: Increasing intensity or volume too quickly.
Surfaces: Hard or uneven training surfaces.
Shoes: Inadequate or worn-out footwear.
Equipment: Improper or poorly fitting gear.
Environmental Conditions: Extreme temperatures or poor weather conditions.
Inadequate Nutrition

86
Q

Osteochondral Fractures and Chondral Injuries:

A

Definition: Damage to the cartilage and underlying bone, usually associated with ankle sprains.
Symptoms: Recurrent pain, stiffness, or joint locking.
Diagnosis: MRI to assess the extent of cartilage and bone damage.
Treatment: Conservative: Cast/brace for 6-8 weeks, followed by rehabilitation.
Surgery: May be required if symptoms persist or damage is extensive.
Complication: Can lead to ankle joint osteoarthritis if not treated properly.

87
Q

Sprained ankle complications

A
  1. Increased risk for recurrent injury
  2. Unstable joint (chronic ankle instability)
  3. Ankle joint osteoarthritis
  4. Persistent pain
88
Q

Treatment of chronic ankle instability

A
  • brace
  • balance and strength
  • surgery