Lecture 7, Hypomobility of Shoulder Flashcards

1
Q

Shoulder Stats

A

GH accounts for 12-25% of complaints
increases in frequency w/age
more female vs male

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2
Q

Who is at increased risk for shoulder symptoms?

A

repetitive and forceful work
previous neck and shoulder injuries
high demand work-place
post-stroke
spinal cord injuries

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3
Q

High risk occupations

A

manual labor
dental healthcare providers
hair stylists
overhead athletes

using 1 hand/side for job
regular keyboard use (more than 4 hours)
10% of day spent in 90° of flexion

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4
Q

Where are the symptoms coming from?

A

C-spine
Neurological
Internal organ

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5
Q

C-spine pain

A

pain with cervical ROM
pain distal to elbow

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6
Q

Internal organ pathology

A

visceral referral–refers pain to arm and shoulder

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7
Q

Bottom line for shoulder pain that is not the shoulder

A

clearn the c-spine in the presence of suspected referral
age >30 yrs, non-traumatic origin, neck pain

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8
Q

Broader areas of pain

A

C4 = up the neck, shoulder
C5 = back of the shoulder

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9
Q

Specific area of shoulder pain

A

AC pattern
subacromial pattern
GH pattern

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10
Q

Joint stability of GH is provided by

A

inert = capsule, labrum, ligament

muscles

remember that joint stability is a prereq for full active range of motion

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11
Q

Scapulohumeral rhythm

A

2 degrees of GH motion to 1 degree of scapular rotation

above 90 –> 1:1 ratio

humerus has to ER to clear greater tuberosity

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12
Q

Limitations in the ______ ________ will decrease ability to reach overhead

A

thoracic extension

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13
Q

Hypomobility can be caused by

A

RA
OA
trauma
post-immobilization
frozen shoulder/adhesive capsulitis

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14
Q

Acute phase of Arthritis

A

pain, muscle guarding
swelling deep in capsule

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15
Q

Subacute phase of Arthritis

A

capsular tightness
loss, greatest at ER, followed by abduction
pain at end of ROM
limited joint play

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16
Q

Chronic phase of arthritis

A

progressive increase in capsule tightness
loss of function, aching in deltoid

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17
Q

Definition of Adhesive Capsulitis

A
  1. ROM loss >25% in at least 2 movement planes
  2. at least 50% loss of passive ER
  3. duration of complaints >3 months
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18
Q

Pattern of ROM loss

A

ER
abduction
IR
flexion

19
Q

Risk factors for idiopathic AC

A

female
age over 40 years
having AC on one side for opposite arm involvement

20
Q

Risk factors for secondary AC

A

increased inflammation
diabetes

21
Q

Risk for traumatic AC

A

following a trauma
prolonged immobilization of GH joint

22
Q

Stage 1 of AC

A

inflammatory stage

gradual onset of pain w/movement
RC is strong
3 months duration
no adhesions or contracture at exam

23
Q

Stage 2 of AC

A

painful/freezing

more intense pain at rest, all ROM limited
PT may not be beneficial at this stage
3-9 months
aggressive synovitis at exam

24
Q

Stage 3 of AC

A

frozen

pain only during movement, compensated by scapula
atrophy of deltoid, RC, biceps, triceps
9-15 months
fibrosis at exam

25
Stage 4 of AC
thawing minimal pain, resolving pain may be showing some improvement in ROM 15-24 months complex fibrosis at exam
26
Treatment approach for AC
level of irritability often reflects the tissue's ability to accept physical stress match intervention strategies to level of irritability
27
High level of irritability AC
not ready for significant physical stress being applied to the affected tissues activity modification, manual therapy, low intensity levels for GH exercises, encourage motion at adjacent regions, limit # of visits
28
Moderate level of irritability AC
apply controlled physical stress to tissues progressive manual therapy mild stretching and strengthening activities basic functional activities
29
Low level of irritability AC
progressive physical stress to tissues stretching, manual therapy, resistive exercise, higher-demand physical activities
30
Patient Education for AC
home exercise programs are highly effective, but patient must have high levels of self-efficacy corticosteriod injections can also help
31
Common limitations with general GH joint hypomobility
inability to reach overhead, behind headm to the side, behind back dressing, grooming lifting weighted objects pain at night
32
Posture with GHJ hypomobility
scapular protraction, anterior tilt, elevated
33
Compensation and substitution w/hypomobility
scapular elevation
34
Acute phase Goals for GH joint hypomobility
maintain soft tissue and joint integrity and mobility maintain integrity and function control pain, edema, joint effusion precaution = pain that increases w/intervention contraindications = no stretching w/RA
35
Subacute goals for hypomobility
progressively incerease joint and soft tissue mobility correct faulty mechanics improve joint tracking improve muscle performance
36
Self-mobilization can be done in the _____ phase
subacute
37
Return to function phase goals GHJ hypo
progressively increase flexibility and strength prepare for functional demands
38
GH Distraction
indications = testing, initial treatment, general capsular mobility separating joint can be perpendicular to treatment plane
39
MWM
mobilization with movement 1. No pain should be produced 2. overpressure is provided to end range 3. results are expected at time of MWM 4. results are long lasting
40
What should you do if you produce pain with MWM?
change direction of the accessory movement change the amount of force technique may not be indicated
41
Indications for MWM
full orthopedic exam has been completed specific biomechanical analysis reveals loss of movement comparable signs manual therapy not contraindicated
42
Goals of MWM
increase active range of motion decrease pain at rest with active motion
43
Dosage of MWM
6 to 10 times reassess comparable sign and fatigue level repeat 2-4 more sets as tolerated