Lecture 6 Flashcards

1
Q

Broca’s area

A

Non fluent speech (speech if slow and broken)–> broken words, hard time with speech production (broken, not fluid) BUT they can understand fine

Motor issues

Frontal lobe

Bad Mother Fucker
(broca, motor, frontal)

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2
Q

Wernicke’s Area

A

Fluent (speech is normal and excessive but makes NO sense)

Sensory

Temporal lobe

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3
Q

Broca and Wernicke’s on a map

A

Broca in front of motor area, wernickes after motor, near auditory corte

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4
Q

Receptive vs expressive aphasia

A

Receptive: Wernicke’s (they can speak fine, but doesn’t make sense)

Expressive: Brocas (can express language, can’t understand)

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5
Q

Definition of learning

A

acquisition of new information

Refers to the process by which experiences change our nervous system and our behavior

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6
Q

Stages of learning

A

Stage 1: Echoic memory, sensory information
- information first processed through our senses
- < 1 second

Stage 2: short term memory, meaningful and salient information
- < 1 minute
- Can support via repitition or chunking (7 +/- 2 rule – you can remember 7 things plus or minus 2)

Stage 3: Long term memory
- consolidation: short term memories are converted into long term memories
- Can be retrieved across a lifetime
- increases retrieval (ie rehearsal) = strengthening of memory
- involves hippocampus

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7
Q

Observational learning

A

AKA Social learning theory

Process of learning by watching the behaviors of Models –> ex. daughter watches mom put on lipstick, she puts on lipstick. How mom reacts (positively or negatively) will influence if daughter does that again

Occurs via operant coniditoning and vicarious conidtioning
–> vicarious: watching sister get in trouble, won’t do what she did

Modes: either prosocial models (prompts engagement in helpful and healthy ways) OR antisocial modeling (prompts others to engage in aggressive/unhealthy bx)

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8
Q

Observational learning: more likely to mimic models who…

A

positive perception (well liked, high status)

shared traits

stand out (mimic someone who stands out because they are creative, different, etc.)

Familiarity (want to be like someone who doesn’t stand out, going back to individuals they know, ex. abuse cycle)

Self-efficacy and mimicry: going to mimic things you can do (things that are in reach)

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9
Q

Observational learning: social media and videogames

A

Social media: branding psychology, trying to create the things above that you are more likely to mimic. The power of influencers; they have high status, they highlight their shared traits (just like you), etc.

Violence in videogames: people watch violent images, so you retain them and then produce them

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10
Q

Three main brain arteries

A

anterior cerebral artery

middle cerebral artery

posterior cerebral artery

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11
Q

Middle Cerebral Artery Strokes (MCA) are _____ of strokes

A

90%

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12
Q

Middle Cerebral Artery Strokes (MCA)

A

Largest of the brain arteries

Supplies most of the outer surface of the frontal, parietal, temporal lobes and the basal ganglia.
* Including pre-central (sensory) and postcentral
(motor) gyrus

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13
Q

MCA Stroke Symptoms

A

Contralateral weaknesses and sensory loss in upper extremities
–> remember: left effects right

Loss of visual field

Left MCA stroke: speech deficits
- brocas
- wernickes

Right MCA stroke: neglect and poor movitation
- flat prosody
- ex. neglect of left side, won’t notice if left art stuck in door

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14
Q

Anterior cerebral artery (ACA) stroke – general

A

Less common (left ACA more common than R ACA)

Feeds deep structures in brain, frontal, parietal, corupus callosum, and bottom of cerebrum

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15
Q

Anterior cerebral artery (ACA) stroke – symptoms

A

Contralateral motry and sensory loss in lower extremeties

poor gait and coordination (clumsy)

slowed initiation (abulia) –> takes longer to do things

flat affect

urinary incontinence

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16
Q

ACA vs MCA strokes

A

ACA: contralateral lower extremity deficits

MCA: contralateral upper extremity and face deficits

17
Q

Depression and strokes treatment

KNOW

A

Post-Stroke Depression Psychosis Treatment = Early Psychopharmacologic treatment is KEY

18
Q

Depression and strokes: depression

A

GET THEM ON MEDS, therapy not enough

  • Post stroke depression = 1/3 of survivors
  • 6x ↑ risk of depression 2-3 years post stroke
  • More common in L frontal and basal ganglia strokes
  • adversely effects functional recovery
  • ↑ Risk Factors = Premorbid depression & Social
    isolation post stroke
19
Q

Psychiatric Considerations Post-Stroke: Anxiety

A

1/4 meet GAD criteria post stroke

less common

20
Q

Psychiatric Considerations Post-Stroke: Psychosis

A

More common in right-temporo-parietal-occipito
area lesions, seizures, and subcortical atrophy

Pseudobulbar Affect (episodes of sudden uncontrollable and inappropriate laughing or crying) = 10-15% post stroke patients

Hypomanic symptoms = 1%

21
Q

If you suspect a stroke, BE FAST

KNOW

A
22
Q

Supplements: deficiency vitamin D

A

Correlated with depression/negative emotions

Concurrent use with anti-depressants supportive –> good depression treatment

the more melanin your skin has, the harder it is to synthezize vit d

Nearly 40-50% of men and women in the Denver metro area are deficient in vitamin D

23
Q

Supplements: magnesium deficiency

A

w/stress can increase agitation, anxiety, sleeplessness, headaches, and apathy

Can treat restless leg syndrome

Slow response time to reach steady state via oral supplementation (30+ weeks) –> if you are VERY defficient, might need infusion

24
Q

Supplements: Omega-3 Fatty Oils

A

Add on treatment for depression (strong evidence good with therapy)

For ADHD, some evidence could be helpful

25
Q

Anorexia: heritability

A

Hereditary: 58-76% in twin studies

26
Q

Anorexia: impact on brain

A

Assocaited with:
- loss of gray and white matter in the brain
- enlarged ventricles and widened sulci (shirnkage of brain tissue)
- inhibited emotional facial expression despite reporting similar or more intense emotions (saying they are really depressed but not showing it)
- tissue loss can be reversed with successful treatment!

27
Q

Anorexia: Starvation study

A

Starvation study:
* 6 months ate at 50% of baseline
* Loss 25% body weight
* Demonstrated preoccupation w/food, ritualistic eating, erratic mood, impaired cognition, slowed eating/lingering
* Post-study = complained of fat on their abdomens and legs

28
Q

Anorexia: Excessive exercise study

A
  • starved mice run on wheel more than well-feed mice –> food seeking?
29
Q

Anorexia: gender differences research

A

Gender differences:
* women ate less post-fast than men

30
Q

Research hypothesis on anorexia

A

restricted food access = starvation = anorexia?

31
Q

Anorexia treatment

A

CBT, increasing eating speed, stimulation of ACC