Learning about time & a theory of learning Flashcards

1
Q

associative learning & causality

A
  • All species live in a world in which they need to predict & control the world around them to survive
  • Associative learning found across animal kingdom
  • All vertebrates & invertebrates, & even in monocellular organisms such as planaria
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2
Q

mahoney & ayres (1976)

A
  • rats, tone paired with a 4/8s shock
  • forward (CS before shock), backward (after shock) or simultaneous conditioning
  • back = learn much if tone after shock
  • simultaneous = learned more
  • forward = learned most when tone before shock

high latency –> high fear

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3
Q

is correlation important for associative learning?

rescorla (1968)

A
  • 3 groups of rats given 5 tones & some shocks
    1. +ve contingency: 2 tone –> shock pairings, no more shocks
    2. zero contingency: 2 tone –> shock pairings, plus extra shocks
    3. -ve contingency: shock never paired with tone
  • +ve tone positively corr with shock & vice versa for -ve
  • 0 tone uncorr with shock
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4
Q

blocking

kamin (1969)

A
  • group 1 light conditioned with a pretrained noise (shock not surprising, predicted by pretrained noise)
  • group 2 light conditioned with a novel noise (shock not predicted, surprising)
  • less learning in group 1 - pretrained noise blocked learning about the light
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5
Q

rescorla & wagner (1972) theory

A
  • describes how much association strength increase on each trial
  • equation
  • each US can only support so much learning
  • learning stops when US no longer surprising
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6
Q

acquisition: light –> food

rescorla & wagner

A
  • gradual acquisition of associative strength V
  • Learning reaches asymptote determined by lambda
  • On each trial sum of V increases & surprise decreases
  • Learning stops when lambda = sum V
  • The max amount of learning to light will increase with lambda
  • As size of food US increases, lambda gets bigger & max amount of learning - asymptote - increases
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7
Q

Mackintosh (1976): Overshadowing

A
  • Light (CS1) and noise (CS2) were paired with shock (US)
  • Group 1: light presented alone before light-noise pairings
  • Group 2: light-noise pairings only
  • Group 1 learned to anticipate the shock from the light more than Group 2 (Noise overshadowed the light for Group 2)
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8
Q

overshadowing & blocking

competition for strength

A
  • control: light gets all associative strength
  • overshadowing: strength divided between light & noise
  • blocking: noise all strength in stage 1 so none left for light

response of light: highest to lowest = c –> o –> b

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9
Q

extinction

inhibition

A
  • light –> food then light –> nothing
  • changes in associative strength -ve
  • initiaally light still predicts the food (expected but doesnt happen)
  • omission of food is surprising
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10
Q

conditioned inhibition

A
  • light –> food
  • light + noise –> nothing
  • on l–>f trials associative strength to light goes up, learning stops
  • on l+n trials light predicts food but nothing happens
  • both l & n lose strength, associative strength goes down
  • l started +ve, ends lower
  • n started neutral, ends -ve
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11
Q

why does inhibitor mean US prediction take longer?

A

it starts -ve

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12
Q

why dont warning signals extinguish

A

even though the warning signal is presented without shock, it is accompanied by the response which is inhibitory

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13
Q

periodic timing

learning

A

learning to respond at a particular time of day

e.g. circadian

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14
Q

interval timing

learning

A

learning to respond after a particular interval of time

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15
Q

cockroaches - time

roberts (1965)

A
  • increased activity at dusk
  • remove visual cues: cycle drifted until increased activity started 15h before dusk
  • restore visualcues: gradual shift back to correct time
  • entrainment - light acts as a zeitgeber synchronising the internal clock
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16
Q

internal clock environmental or innate?

bolles & stokes (1965)

A
  • subjects born & reared under 19, 24 or 29h cycles
  • fed at regular point in own cycle (signalling few hrs after change in lighting)
  • animals on 24h cycle leaned to anticipate food
  • others didnt
17
Q

suprachiasmatic nucleus (of hypothalamus) & 24h clock

A
  • Metabolic rate appears to vary as a function of the day-night cycle
  • Lesions of the SCN abolish the circadian regularity of foraging & sleeping in the rat
  • Receives direct & indirect impulses from the visual system, which could keep circadian rhythms entrained with the real day-night cycle
18
Q

physical illness & disruptions to circadian rhythms

A
  • heart disease
  • diabetes
  • infections
  • cancer
19
Q

mental illness & disruptions to circadian

A
  • depression
  • SZ
  • bipolar illness
20
Q

weber’s law

A
  • JND when you change a stim is proportional to initial intensity/magnitude of changed stim
  • –> in absolute terms small amount is judged more accurately than large amounts
  • critical point is that % change is more important than absolute change
21
Q

scalar timing theory components

gibbon et al. (1984)

A
  • pacemaker
  • working mem
  • reference mem
  • comparator
22
Q

pacemaker

scalar timing theory

A
  • emits pulses at a roughly constant rate t (random variation)
  • when a stim is presented, a switch is operated & pulses allowed to accumulate in WM
23
Q

working mem

scalar timing theory

A
  • when reinforcement occurs, pulses stop accumulating
  • another switch allows num of pulses in working mem (N x t) to be stored in refernce mem
  • this storage not completely accurate - some mem distortion
24
Q

reference mem

scalar timing theory

A
  • K x N x t
  • K represents mem distortion (K less 1 smaller num pulses stored & vice versa for more 1)
  • error proportional to duration scalar
25
Q

comparator

scalar timing theory

A
  • on each trial animal compares num of pulses in working mem with a random value drawn from those stored in reference mem (NMx)
  • done by comparator
  • if values close, animal responds
26
Q

another stim occuring

scalar timing theory

A
  • successive num of pulses stored in WM
  • uses 1 of values in reference mem to decide when to respond
  • comparator works out how close values are (ratio rule)
  • large = dont respond
  • small = response
27
Q

problems with scalar timing theory

A
  • no physiological ev for a pacemaker
  • conditioning & timing supposedly occur at same time but controlled by diff mechanisms
28
Q

alternatives to pacemaker

problems with scalar timing theory

A
  • timing achieved by a series of oscillators with 2 states (on/off), pattern of activation used to determine exact time
  • behavoiural theory of timing
29
Q

behavioural theory of timing

alternatives to pacemaker

A
  • When the animal gets a reward, this simulates beh
  • Animal moves across an invariant series of behavioural classes in between reinforcements
  • Pulse from an internal pacemaker will change the beh from 1 class to another
  • The beh that is occurring when the next reinforcer occurs becomes a signal for that reinforcer
30
Q

conditioning & timing occuring at same time but with diff mechanisms

problems with scalar timing theory

A
  • G&B - calculate rate of reinforcement during stim & background. if CS higher than background –> conditioning
  • S&B - sim to regular conditioning but stim is assumed to change over course of its presentation, allowing animal to learn about when a reinforcer occurs