L9: Cardiac Arrhythmias Flashcards

1
Q

fast heart rate >100bpm

A

tachycardia

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2
Q

causes of tachycardia

A

increased body temp.
sympathetic stimulation
toxic heart conditions (weakened myocardium)

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3
Q

tachycardia: increased body temperature

A

HR increases by 10 bpm per 1F
or
increases by 18 per 1C

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4
Q

tachycardia: sympathetic stimulation

A

severe blood loss or shock

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5
Q

slow heart rate <60bpm

A

bradycardia

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6
Q

causes of bradycardia

A

athletic heart
vagal stimulation
extremely sensitive carotid baroreceptors in carotid sinus syndrome

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7
Q

respiratory sinus arrhythmia

A

ability to change heart rate thru inspiration/expiration cycles

thus increases/decreases number of impulses sent via the sympathetic/vagal

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8
Q

exercise induced tachycardia

A

endogenously mediated tachycardia
increased HR,CO
decreased filling time but SV does not drop

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9
Q

pathologically mediated tachycardia

A

increased HR

decreased CO

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10
Q

what factor drops in pathological tachycardia and why?

A

cardiac output
mean arterial pressure decreases
sympathetic activated after thus not able to compensate

thus no muscles stimulated to increase venous return

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11
Q

sudden cessation of P waves

A

sinoatrial block

atria standstill

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12
Q

sinoatrial block

A

ventricles pick up new rhythm
QRS is slowed but not altered
P wave is missing

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13
Q

causes of atrioventricular block

A

ischemia of AV/bundles
compression of AV by scar tissue
inflammation of AV
extreme stimulation of heart by vagus

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14
Q

PR interval increases greater than 0.2 seconds

A

1st degree incomplete atrioventricular heart block

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15
Q

PR interval increases 0.25 to 0.45 seconds

A

2nd degree incomplete atrioventricular heart block

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16
Q

results of 2nd degree atrioventricular block

A

missing beats of ventricles

2:1 rhythms develop

17
Q

P wave and QRS complex are no longer related

A

complete atrioventricular block

18
Q

in atrioventricular blocks the AV node may cease conduction

A

ventricles may not beat again for 5 to 30 seconds

19
Q

ventricular escape

A

resuming of ventricular beats due to purkinje fibers acting as ectopic pacemaker

20
Q

in a complete atrioventricular block patients may…..

A

faint due to lack of blood to brain

21
Q

stokes-adams syndrome

A

fainting spells caused by complete atrioventricular blocks

22
Q

electrical alternans

A

partial intraventricular block
an alternation in amplitude of P, QRS, or T

no missing waves

23
Q

contraction occurring before it should

A
premature contraction
caused by: an ectopic focus
local ischemia
calcified plaques
irritation of conduction system or nodes
24
Q

heart suddenly beats rapidly, ranging from seconds to hours or even days

A

paroxysmal tachycardia

begins and ends suddenly
returning to normal at conclusion

25
Q

the slow-twitching of individual muscle fibers in a heart chamber

A

fibrillation

26
Q

occur when the length of the pathway remains normal but the conduction velocity of the impulse is slowed

A

circus mvt

27
Q

causes of circus mvts

A
blockage of purkinje system
ischemia
high K+ levels
response to epinephrine
response to repetitive electrical stimulation
28
Q

basis for fibrillation

A

occurs as a result of circus mvts

29
Q

why does normal ventricle depolarization die out in circus mvts and fibrillation?

A

because a portion of the heart is already in refractory period and cannot respond to a second stimulus

30
Q

3 different conditions resulting in circus mvts

A

the pathway is too long – dilated heart

length of pathway is constant but velocity has slowed

refractory period has become greatly shortened

31
Q

cardiac fibrous skeleton

A

ventricle and atria fibers are separated by this

allows each chamber to exhibit fibrillation independently of each other

32
Q

causes of atrial fibrillation

A

enlargement of atria – How?

  • -valve lesions
  • -inadequate emptying of ventricles
33
Q

how does inadequate emptying of ventricles enlarge the atria?

A

causes blood to back up into the atria