Johnston ECG Rhythms and Disturbances Flashcards

1
Q

What is a normal axis considered?

A
  • AVF positive and lead 1 positive
  • bottom right quadrant
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2
Q

Left axis?

A
  • Positive lead 1
  • Negative AVF
  • Top right quadrant
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3
Q

Right axis?

A
  • negaitve lead 1
  • positive AVF
  • bottom left quadrant
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4
Q

rate rhythm axis

A
  • 100 bpm
  • Left axis
    • positive lead 1 negative AVF
  • sinus rhythm tachy
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5
Q

Symptoms of arrhythmia?

A
  • Palpitations such as skipping, pounding, irregular
  • Lightheadedness
  • Syncope
  • Chest pain
  • Dyspnea
  • Sudden death
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6
Q

Etiology for arrhythhmia?

A
  • Stress
  • Ischemia
  • Hypoxia
  • Metabolic acidosis
  • Infection
  • Inflammation
  • Cardiomyopathy
  • Electrolyte imbalance
  • Drugs
  • Htn
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7
Q

What is sinus tachycardia?

A
  • physiologic/pathologic process
  • can be caused by emotion, anxiety, fear, drugs, hyperthyroidism, fever, pregnancy, anemia, CHF
  • Hypovolemia
  • Treat underlying cause
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8
Q

what is this

A

normal sinus rhythm with physiologic sinus arrhythmia

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9
Q

what is this and what is rate

A

all are sinus tachycardia

  • 138 bpm
  • 160 bpm
  • 140 bpm
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10
Q

What is considered bradycardia?

A

<60bpm

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11
Q

Where are you going to see sinus bradycardia?

A
  • Normal people
  • Healthy athlete
  • Physiologic component to sleep, fright, carotid sinus massage/hypersensitivity
  • Obstructive jaundice
  • Sliding hiatal hernia
  • Valsalva maneuver
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12
Q

What type of drugs can cause bradycardia?

A

Beta blockers such as propanolol and metoprolol

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13
Q

Medical conditions/situations associated with bradycardia? (labs)

A
  • Acute inferior MI (increased vagal tone, N/V)
  • Ischemia
  • decreased pO2
  • Increased pCO2
  • Decrease PH
  • Increase BP
  • Sick Sinus Syndrome
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14
Q

what is this?

A

sinus bradycardia

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15
Q

How do you treat sinus bradycardia?

A
  • Depends on clinical setting and cause
    • it may not need to be treated
  • Depends on hemodynamics
  • Use Atropine
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16
Q

What is automaticity?

A
  • Property of cardiac cell to depolarize spontaneously during phase 4 action potential leads to generation of an impulse
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17
Q

What is the significance of premature atrial contractions?

A
  • can feel it or see it on ECG but it is of no hemodynamic significance
    • unless it turns into a lot of irregular beats or causes distress
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18
Q

When can you see a PAC?

A
  • Absence of significant heart disease
  • associated with stress alcohol tobacco coffee COPD and CAD
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19
Q

what is this?

A

PAC

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20
Q

what is this?

A

non conductive PAC

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21
Q

Different types of PAC

A
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22
Q

Treatment of PACs?

A
  • treat cause
  • Beta adrenergic antagonist
    • Metoprolol 25-50 mg BID-TID
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23
Q

What is paroxysmal atrial tachycardia?

A
  • Sudden heart rate greater than 100 BPM
  • look for irritable focus P wave
  • This one is about 150-200 bpm
24
Q
A

PAT with AV block

  • think digitalis excess or toxicity
  • ratio of 2:1 P:QRS
25
Q

What is multifocal atrial tachycardia?

A
  • 3 or more P waves
  • PR interval varies
  • Irregular ventricular rhythm
  • Atrial rate >00 bmp
  • Associated with an underlying pulmonary disease
26
Q

What is this?

A

MAT

3 or more different types of atrial contractions (look at P )

27
Q

what is this?

A
  • Multifocal Atrial Tachy
  • biphasic
  • decreased amplitude
  • inverted (#4)
28
Q

How do you treat MAT?

A
  • Ca channel blocker
  • Diltiazem
  • Verapamil
  • Mg SO4
  • amiodarone
  • digitalis isn’t helpful and DC cardioconversion isn’t effective
29
Q

What is A fib?

A
  • Atrial quivering with a rate of 350-600 bpm
  • Undulating baselines, no discernible P waves
  • Irregular PR intervals
  • Irregularly irregular
30
Q

what is this

A
  • A fib
  • can’t make out any P waves at the arrows, just see undulation of the baseline
31
Q

what is seen?

A

a fib

32
Q

What has a typical “saw tooth pattern”

A
  • Atrial flutter: 250-350 bpm
  • Seen best on leads II, III, AVF, and V
33
Q

What is paroxysmal junctional tachycardia?

A
  • 150-250 bpm
  • P wave may be lost in QRS, inverted before or after each QRS
  • Comes from the junction UP to the atrium (that is why P wave is inverted)
34
Q

what is happening?

A

P wave is buried in QRS or ST segment, it is a paraxysmal junctional tachycardia as it is irritable junctional focus paces rapid

35
Q

what is this?

A
  • QRS is narrow and rate is around 200
  • Junctional tachycardia
36
Q

What is this?

A
  • Paroxysmal Supraventricular Tachycardia
  • includes PAT and PJT
37
Q

What causes Premature Ventricular Contractions (PVC’s)

A
  • normal heart
  • CAD MI HF MI Hypoxia
  • Valvular heart disease
  • Congenital heart disease
  • Cardiomyopathy
  • Acid base imbalance
  • Hyperthyroid
38
Q

ECG characteristics of PVC’s?

A
  • premature bizarre wide QRS
  • No preceding P wave, may produce a retrograde P wave in ST segment
  • ST-T wave moves opposite direction of QRS
  • Usually full compensatory pause
39
Q

what is this

A

PVC

40
Q

what is happening?

A
  • multiple PVC’s
  • QRS is elevated ST segment is down with a notch in it, possibly a P wave
41
Q

what is this?

A

run of PVC’s

Run >3 is considred Ventricular Tachycardia for at least 30 seconds or more

42
Q

what is this?

A
  • wide QRS
  • ST segments inverted
  • Multifocal premature ventricular contractions
    • hemodynamically unstable
43
Q

What is ventricular premature contraction with R on T phenomenon?

A
  • The beat is hitting during the ventricular repolarization
    • can lead to v tach or v fib
    • watch patient closely
44
Q

What is an Accelerated idioventricular rhythm?

A
  • Good sign of reperfusion, indicates that the thrombolytic agent is working
  • Do not treat just leave alone
45
Q

How do you treat PVC’s?

A
  • If stable no treatments
  • If sx in setting of ACS use Metoprolol
  • If unstable Amiodarone, lidocaine, Procainamide
46
Q

What is V tach?

A
  • 3+ consecutive bizarre QRS complexes
  • Ventricular rate is 120-200
  • Usually regular with a wide ARS
  • P wave is lost and if it is seen it has no relation to QRS
  • Lasts longer than 30 seconds
47
Q

what is this?

A
  • Paroxysmal Ventricular Tachycardia beating around 150-250
  • Wide QRS
  • ST going opposite direction
  • Suddenly occurred
48
Q

what is happening?

A
  • Runs of V tach
  • first QRS is inverted ST elevated
  • 4 QRS elevations in a row
  • end of strip is more runs of v tach
  • Multifocal (from different sites, they don’t all look alike)
49
Q

what is happening?

A

from one foci, they all look alike

V tach

50
Q

what is this?

A
  • Wide QRS 200+bpm
  • Premature Ventricular contraction but sustained so it is Vtach
51
Q

What is Torsades de Pointes?

A
  • Twisting of the points
  • QRS swings from positive to negative
  • May be inherited from Prolonged QT
  • Could be acquired by meds, electrolyte imbalances, alcohol
52
Q

what is the diagnosis? what can happen later on?

A

This is Torsades de Pointes, this can lead to V tach

53
Q

How do you treat torsades?

A
  • MgSO4
  • Overdrive pacing
  • Isoproternol
  • Get off what might have induced the arrhythmia
54
Q

What is Ventricular fibrillation?

A
  • Disorganized depolarization
  • Not an effective pump
55
Q

what is this?

A

v fib

56
Q

what is this?

A

vfib

differentiate from artifact, if patient is stable and talking probably not v fib