ITE - Cardiology Flashcards

Educational objectives for missed questions

1
Q

Chronic venous insufficiency: Clues, Dx

A

Dx: clinical diagnosis with/without venous duplex u/s
clues: edema, dilated veins, thin/hyperpigmented skin, ulceration.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Hypertrophic cardiomyopathy: Clues, Dx.

A

Clues: ejection murmur at LSB (outflow obstruction), late systolic murmur at apex (MR), abnormal ECG
Dx: TT echo w/ asymmetric LVH and septal thickening

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Peripheral vascular disease: Dx.

A

Dx: resting ABI < 0.9 (1.0-1.4 is normal), exercise ABI with >30mmhg or 20% drop. ABI > 1.4 uninterpretable and needs toe ABI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

RIght ventricular infarction: Dx.

A

Sxs: hypotension, JVD, clear lungs
ECG: V1, V4R

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Torsades de Pointes, drug-induced: Dx

A

Dx: Use of QTc prolonging meds
Ex: ondansetron, amio, quinolones, antifungals, antipsychotics, triptans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Wolff-Parkinson-White pre-excitation: Dx

A

Dx: symptomatic AVRT with preexcitation (delta wave)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Resynchronization therapy: Indications

A

HF w/ EF < 35%, NYHA class II-IV despite GDMT, sinus rhythm, LBBB with QRS > 150

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

AC for mechanical prosthetic valve

A

Lifelong AC and antiplatelet therapy. Goal INR 2.5, with risk factors or ball/cage valve INR 3.0, bioprosthesis INR 2.5

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Bradycardia: Tx

A

Pacemaker for symptomatic and asymptomatic with high risk conduction abnormalities or heart block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Mitral stenosis: Ausculation

A

Loud S1, increased pulmonic component of S2, opening snap, diastolic rumble, low pitched murmur at apex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

STEMI + signs of heart failure or shock

A

Emergent PCI, temporize with IABP or LVAD, reduce afterload, avoid beta blockers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

STEMI + V-Fib

A

EP consult and ICD, predischarge

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Atrial myxoma: Tx

A

Surgical removal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Cardiogenic shock: Tx

A

Early/Aggressive fluids with initial bolus of 30mL/kg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

ICD: indications

A

HF w/ EF<35%, NYHA class II-III despite GDMT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

PVCs: Tx

A

Reasurance

If high risk or > 10% of beats affected: Beta blocker or CC blocker, then antiarrhythmics, then ablation

17
Q

PVD: Tx

A

Tx: Similar to CAD. Smoking cessation, DM control, HLD control. Aspirin, supervised exercise training (most effective way to improve fxnl status)

18
Q

Claudication: Tx

A

cilostazol for claudication

19
Q

Unstable angina: Tx

A

Tx: relieve pain, early risk stratification (TIMI risk score), if high risk –> PCI, if low –> medical management

20
Q

NYHA class I

A

?

21
Q

NYHA class II

A

?

22
Q

NYHA class III

A

?

23
Q

NYHA class IV

A

?

24
Q

Risk factors ranked

A

?

25
Q

ECG abnormalities that contraindicate stress ECG:

A

ST segment changes, preexcitation, LBBB, VPCs, digitalis effect

26
Q

digitalis effect:

A

?

27
Q

Contra’s for adenosine (for single-photon emission CT)

A

reactive airway disease with active wheezing

28
Q

Red flag murmurs

A

???diastolic or continuous murmur, cardiac symptoms (chest pain, dyspnea, syncope), or abnormalities on examination (clicks, abnormal S2, abnormal pulses)

29
Q

Benign murmurs do what with standing

A

decrease

30
Q

HCM, hemodynamics that affect

A

increases with decreased venous return

31
Q

Maneuver to decrease venous return

A

standing

valsalva

32
Q

MV prolapse, hemodynamics that affect

A

decrease venous return will move earlier in systole, increase intensity