Integumentary Flashcards

1
Q

role of lymphatic system

A

drainage/sanitation
one way system to transport lymph w proteins/water/fatty/acids/cellular components through lymph nodes, thymus, bone marrow, spleen, tonsils, Small intestine
- remove waste, excess fluid
- alert immune system
- return fluid and plasma proteins to blood

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2
Q

lymphatic system anatomy small to large

A

lymph capillary
pre collectors
collectors
lymph nodes
lymphatic trunks
R lymphatic duct and thoracic duct

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3
Q

lymph formation

A

precollectors fill with low interstitial pressure
interstitial pressure increases with filling, inlet valves open w Pressure
fill lymph vessel w fluid, lowering pressure and close valves
valves open to precollector and fluid flows onward

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4
Q

transport capacity

A

amount of fluid the system can move at maximum intensity
normally works at 10% capacity

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5
Q

functional reserve

A

difference between transport capacity and amount of fluid being transported at rest

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6
Q

lymphatic load

A

amount of fluid being transported at rest

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7
Q

dynamic insufficiency

A

caused by immobility, CHF, sprained ankle, increased LL

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8
Q

mechanical insufficiency

A

caused by damage to lymphatic system making it unable to handle increased LL
eg lymphedema
also surgery, infection, trauma causing reduced TC

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9
Q

causes of lymphedema

mechanical cause within lymphatic system

A

TC dropping below LL
causes accumulation of fluid in subcutaneous tissue
risk factors include: excess weight, arm infection/injury

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10
Q

diagnosis of lymphedema

primary/secondary

A

primary: from abnormally developed lymphatic system
secondary: results from known injury

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11
Q

clinical presentation of lymphedema

A

slow progression
mild warmth
no color change
painless
full/heavy feeling
pitting edema
asymmetrical limbs

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12
Q

differences between lymphedema and general edema

A

risk factors
other diagnoses
Stemmer sign: + for lymphedema
acute injury is general
general chronic edema comes with skin changes, achy pain, progressing through day

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13
Q

stages of lymphedema

A

0: no edema, - stemmer
1: soft pitting edema, reversible w elevation, increase w activity, - stemmer
2: spontaneously irreversible, edema progressing to nonpitting brawny edema, not reversed w elevation, + stemmer, fibrosclerotic tissue, frequent infection/skin changes
3: lymphostatic elephantiasis, severe brawny edema not reversed w elevation, + stemmer, fibrosclerotic tissue, frequent infection, skin changes

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14
Q

Stemmer sign

A

+ if skin cannot be pinched and lifted at base of fingers or toes

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15
Q

lymphedema treatment

A

depends on cause, which should be addressed
medications

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16
Q

cellulitis

A

bacterial skin infection with open wound

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17
Q

cellulitis s/s

A

red, edema, tender, pain, warm, blister, fever, headache, chills, weakness, red streaks

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18
Q

cellulitis diagnosis

A

blood tests, skin culture

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19
Q

cellulitis treatment

A

antibiotics (oral/topical/IV)
wound dressing
pain meds
surgery

20
Q

complications of cellulitis

A

extensive tissue damage, gangrene, blood infection causing sepsis, amputation, deat

21
Q

prevention of cellulitis

A

hygiene, protect dry skin, protective footwear, wound prevention

22
Q

function of the epidermis

A

skin integrity
physical barrier to pathogens
protect against cellular fluid loss

23
Q

function of the dermis

A

tensile strengh/support
retain moisture, blood O2

24
Q

normal wound healing timeline

A

1: inflammatory 1-10 days
2: proliferative 3-21 days
3: maturation 7 days - 2 years

25
Q

factors affecting wound healing

A

age
comorbidities
edema
bad wound care
infectionlifestyle
stress
medications

26
Q

what happens when healing phases are interrupted?

A

1: chronic inflammation cycle can occur
2: delay healing resulting in chronic wound
3: scar tissue remodeling, only 80% strength of normal tissue

27
Q

ideal wound healing environment

A

moist
breathable barrier
controlled exudate for peri wound integrity
change bandage when leakage

28
Q

wound characteristics to take note off

A

location
size
shape
edges
tunneling
base compared to sides
periwound area
pain

29
Q

serous drainage

A

clear/watery
inflammatory/proliferative phases

30
Q

sanguineous drainage

A

red/watery
inflammatory/proliferative phases

31
Q

serosanguineous drainage

A

clear/pink/watery
inflammatory/proliferative phases

32
Q

seropurulent drainage

A

cloudly/opaque/yellow/waterly
early warning sign of infection, abn finding

33
Q

purulent drainage

A

yellow/green/thick
wound infection, abn finding

34
Q

necrotic tissue types

A

slough: easily removed moist/stringy white or yellow tissue attached to wound
eschar: hard dehydrated tissue adhered to healthy tissue
gangrene: death of tissue due to interrupted blood dlow
hyperkeratosis: white callus, firm/soggy texture

35
Q

primary intention wound closure

A

surgeon closes edges by approximating with flue/stitches, etc
opening called dehiscence

36
Q

secondary intention wound closure

A

wound left open to heal on its own
new tissue laying down in wound bed closes eventually
heal deepest to superficial

37
Q

tertiary intention wound closure

A

delayed primary
secondary intention fails and it is closed surgically
may be purposefully delayed due to infection, closed once healed

38
Q

abn findings in wound healing

A

changing: color, odor
persistent edema
necrotic tissue
tunneling
infection
ridge at wound edge
hypertrophic scarring

39
Q

venous ulcer

A

edema, pain in dependent position, cyanotic, pigmentation of skin, exudate

40
Q

arterial ulcer

A

poor pulse, severe pain, cool to touch, trophic changes to skin, deep ulcers, black gangrenous skin

41
Q

diabetic ulcer

A

diminished pulse
sensory loss
ulcer present
may develop gangrene

42
Q

pressure ulcer

A

pain if sensation intact
color change
warm if infection/fever
necrotic tissue
over bony prominences
can be gangrene

43
Q

Braden Scale

A

predict pressure sore risk
use sensory perception, moisture, activity, mobility, nutrition, friction/shear

44
Q

stages of pressure ulcers

A

1: intact skin, nonblanchable redness
2: partial thickness tissue loss, no slough
3: full thickness tissue loss, visible structures underneath
4: full thickness w exposed bone, slough or eschar
unstageable: full thickness, covered in slough
deep tissue injury: purple area of discolored skin due to soft tissue damage

45
Q

risk factors for diabetic ulcers

A

poor footwear/deformity/foot trauma
non compliance w dx management
lack of protective sensation
skin changes
Hx of amputation
alc/tobacco
immobile
advanced age
weakened immune
vascular disease

46
Q

wagner diabetic ulcer grades

A

0: no open lesion, may be preulcerative
1: superficial ulcer, not subcutaneous
2: deep ulcer into subcutaneous, may expose structures
3: deep ulcer w abscess/bone infection
4: gangrene –> maybe amputation
5: gangrene w deep tissue injury –> definitely amputation