HIIT Flashcards

1
Q
A
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2
Q

Describe HIIT

A

High-Intensity Interval Training
- Near maximal effort
- Elicits 80% max HR
- Rest Periods

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3
Q

Define MICT

A

Moderate Intensity Continuous Training:
- Exercise performed continuous manner
- Lower intensity than HIIT

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4
Q

Describe SIT

A

Sprint Interval Training
- VO2max Intensity
- All-out effort
- REst periods

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5
Q

Which skeletal muscle adaptations occur from interval training?

A
  • Mitochondrial response: enzyme levels; electron transport chain complexes
  • Changes each fiber type
  • Muscle capillary density
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6
Q

What regulates substrate metabolism during submaximal exercise?

A
  • skeletal muscle mitochondrial density
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7
Q

What does an increase in mitochondrial content promote?

A
  • Increase reliance on fat oxidation
  • Proportional decrease in carb oxidation
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8
Q

Which techniques are used for investigating the effects of exercise on skeletal muscle mitochondria?

A
  • Changes in signaling proteins
  • Gene Expression
  • Mitochondrial protein synthesis rate
  • Enzyme content
  • Volume of mitochondria (microscope)
  • Oxidative Phosphorylation Capacity
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9
Q

How quickly can Citrate Synthase Activity increase after HIIT or SIT sessions?

A
  • 24hr after 1 session
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10
Q

How much does Citrate Synthase and Cytochrome C Oxidase increase by following 6-7 sessions of HIIT or SIT?

A
  • 25-35%
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11
Q

What happens when exercise duration and intensity are held constant regarding mitochondrial content?

A
  • Plateau after 5 days of training
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12
Q

What happens to mitochondrial content when intensity continues to increase progressively?

A
  • Rises for at least several weeks
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13
Q

What does evidence suggest about exercise intensity?

A

Cellular stress occurs in proportion to exercise intensity:
- Greater metabolic response to high-intensity exercise compared to moderate

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14
Q

Explain the study that shows superior mitochondrial adaptation from interval compared to continuous

A
  • Counterweighted, single-leg cycling
  • study adaptations in same subject
  • 10 young men
  • 6 sessions HIIT 1 leg
  • 6 session MICT other leg
  • Matched total work
  • 2 weeks
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15
Q

Describe the differences between MICT and HIIT when matched for work

A
  • Skeletal muscle capillarization greater in MICT
  • Skeletal mitochondrial density greater for HIIT
  • VO2max greater for HIIT
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16
Q

What can exercise training do for individuals with type 2 diabetes? What is not known about it?

A
  • Improve glycaemic control
  • Unknown optimal training regimen
17
Q

What is the acute response of the pancreas to exercise bouts?

A
  • Decrease Insulin Secretion
  • Increase Glucagon Secretion
18
Q

What is the acute response of the liver to exercise bouts?

A
  • Increase in Glucose Release
19
Q

What is the acute response of adipose tissue to exercise bouts?

A
  • Increase in Triacylglycerol Breakdown
  • Increase in NEFA Release
20
Q

How does HIT compare to MICT with glycaemic improvement?

A

HIT
- Confer superior glycaemic improvement with lower time commitment

21
Q

How do athletes achieve training in a low carbohydrate state?

A
  • Fast overnight: reduce glycogen content/lowers carb available
  • Training 2/day: depletes muscle glycogen from first bout
  • Sleep Low: both above; reduced muscle glycogen by training, reduced liver glycogen by fasting
22
Q

Why would someone train in low carb availability?

A

Type 2 Diabetes
- Superior Glycaemic improvements
- High-intensity after meal
- low-intensity fasted conditions

23
Q

what are the benefits of strength training for glycemic control?

A
  • promotes skeletal muscle hypertrophy and increases strength,
  • more powerful contractions fuelled by glycolysis and supported by higher lactate dehydrogenase content
24
Q

what are the benefits of aerobic training for glycemic control?

A
  • enhances cardiovascular function
  • promotes skeletal muscle mitochondrial biogenesis
  • improving exercise endurance
25
which type of training helps with glycemic control?
all training types improve skeletal muscle glucose transport and glycogen synthesis capacity, expanding glycogen stores and improving glycaemic control