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Equine Medicine > GIT > Flashcards

GIT Flashcards

1
Q

Small colon impactions are strongly associated with infection of which organism?

A

Salmonella

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2
Q

Factors that adversely affect surgical outcome of horses with descending colon obstructive disease include:

A

Small colon’s relatively poor blood supply High concentration of collagenase High intraluminal concentration of bacteria High muscular activity Presence of particulate faeces

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3
Q

List the horses most prone to rectal tears:

A

Arabians, ponies, small breed Young

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4
Q

Once a rectal tear is suspected, the veterinarian must:

A
  1. Advise the owner a rectal tear has occurred 2. Further assess the severity of the tear 3. Explain the nature of the problem openly with the owner 4. Initiate appropriate treatment (may include referral) 5. Contact liability insurance company
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5
Q

At what position do most rectal tears occur and why?

A

25-30cm from the anus at 10-12 o’clock - thickness of muscle decreases and weak area due to penetration of terminal arteries.

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6
Q

What grade rectal tear is this and what structures are involved?

A

Grade 1 rectal tear - mucosa and submucosa

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7
Q

What grade rectal tear is this and what structures are involved?

A

Grade 2 rectal tear - musularis only

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8
Q

What grade rectal tear is this and what structures are involved?

A

This is a grade 3A rectal tear - mucosa, submucosa and muscularis are torn. Diverticulum is formed from the serosa.

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9
Q

What grade rectal tear is this and what structures are invloved?

A

Grade 3B rectal tear - mucosa, submucosa and muscularis torn. Only serosa intact. Involved the mesocolon / mesorectum / retroperitoneal space.

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10
Q

What grade rectal tear is this and what structures are involved?

A

Grade 4 - Torn through mucosa, submucosa, muscularis and serosa

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11
Q

As soon as a rectal tear is diagnosed the following first aid should be applied:

A
  1. Reduce activity of the rectum
  2. Gently remove faeces from the tear and the rectum
  3. Rectal packing
  4. Broad spectrum antibiotics
  5. Analgesia
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12
Q

What treatment would you give to grade 1-2 rectal tears?

A

Should be able to sufficiently treat with broad spectrum antibiotics and laxatives - heal in about a week.

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13
Q

How would you treat grade 3-4 rectal tears?

A

Need referral and transport (leave rectal packing in place!). Usually require surgical intervention (temporary indwelling rectal liner / colostomy / pimary repair) as well as medical management (antibiotics and laxatives).

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14
Q

What type of rectal prolapse is this? Describe what is occurring.

A

Type 1 rectal prolapse - prolapse of rectal mucosa and submucosa

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15
Q

What type of rectal prolapse is this? Describe what is occurring:

A

Type 2 rectal prolapse - prolapse of rectal mucosa and muscularis (full thickness rectum)

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16
Q

What type of rectal prolapse is this? Describe what is occurring:

A

Type 3 rectal prolapse - prolapse of full thickness rectum and peritoneal portion of the rectum or colon intussuscepted

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17
Q

What type of rectal prolapse is this? Describe what is occurring?

A

Type 4 rectal prolapse - peritoneal portion of the rectum or colon intussuscepted through the anus

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18
Q

At what length of rectal prolapse is mesenteric blood supply usually disrupted and what is the prognosis?

A

>30cm - the prognosis is terrible

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19
Q

What is the capacity of the equine stomach?

A

1.5% of body weight

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20
Q

What is the role of the mucus cells in the stomach?

A
  • secrete bi-carbonate rish mucous
  • coat and lubricate gastric surface
  • protects epithelium from acid and other chemical insult
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21
Q

What is the role of the parietal cells of the stomach?

A
  • Continuously secrete HCl
  • Activation of pepsinogen and inagtivation of ingested microorganisms
22
Q

What is the purpose of the chief cells of the stomach?

A
  • Secrete protease: pepsinogen
    • Pepsinogen activated by stomach acid into pepsin - digestion of proteins
  • In folas: secrete chymosin (rennin) - protease that coagulates milk proteins
23
Q

What is the role of G-cells in the stomach?

A
  • Secrete gastrin - relax proximal stomach and increase contractions in distal stomach
24
Q

What are the functions of the equine stomach?

A
  • Short term storage reservoir
  • Initiation of chemical and enzymatic digestion
  • Liquefaction of food
25
Q

What are some gastric mucosal barriers?

A
  • Mucus bicarbonate layer
  • Production of endogenous prostaglandins
  • Rapid cellular restitution
  • Extensive capillary network
26
Q

Where do we see EGUS first?

A

The margo plicatus

27
Q

What is the prevalence of EGUS?

A

70-100% in racehorses

28
Q

What is the cause of EGUS?

A

Acid exposure

29
Q

What are some risk factors of EGUS?

A
  • intermittent feeding
  • exercise intensity
  • diet composition
  • transport stress
  • stall confinement
  • illness
  • NSAIDs
  • management changes
30
Q

How would you grade endoscopy of EGUS?

A
  1. intact mucosa with areas of reddening / squamous hyperkeratosis
  2. small single / multifoca lesions
  3. large single / multifocal lesions or extensive superficial lesions
  4. extensive lesions with areas of deep ulceration
31
Q

How could you classify EGUS?

A
  1. Primary squamous lesions
  2. Primary gastroduodenal lesions
  3. Stress related glansular disease
32
Q

What tests can you use to indicate EGUS?

A
  • sucrose permeability test (urine / blood sucrose)
  • faecal occult blood test
33
Q

How would you treat EGUS?

A
  • Acid suppressive therapy (histamine antagonist - ranitidine)
  • Bufferring of excess acid (antacids)
  • Mucosal protecting (sucralfate)
  • Environmental and dietary management (feed more frequently)
34
Q

How can you treat pyloric stenosis and delayed gastric emptying?

A
  • Prokinetics
  • Dietary modification
  • Surgery (Y-U pyloroplasty)
35
Q

When might you give a horse diet coke?

A

Gastric impaction by persimmons

36
Q

Approximately how long is the small intestine and what is the transtit time of ingesta through it?

A

~ 27m long

1-3hrs transit time

37
Q

What are some signs of small intestinal disease?

A
  • signs of abdominal pain
  • reduced abdominal borborigmi
  • large volume of gastric reflux
  • ultrasound examination (bunch of grapes)
38
Q

What is the normal small intestinal wall thickness and diameter?

What should it look like inside the intestine?

A
  • Wall thickness <2mm
  • Diameter <40mm
  • Contents - swirling fluid
39
Q

Onw hich side of the abdomen is the epiploid foramen?

A

The right hand side of the abdomen

40
Q

What can you see in this ultrasound inmage?

A

An intussusception

41
Q

List some causes of strangulating small intestinal obstruction:

A
  • pedunculated lipoma
  • epiploic foramen entrapment
  • herniation
  • volvulus
  • intussusception
  • adhesion
42
Q

In what age group would we see parascaris equorum?

A

Less than 6 months

43
Q

In what age group do we see lawsonia intracellularis?

A

Foals: 3-7 months

44
Q

What is the blood supply to the ventral colon?

A

ONE vessel - colic branch of the ileocaecal artery

45
Q

What is the approximate volume of the caecum?

A

30L

46
Q

How many taeniae does the caecum have?

A

4

47
Q

What are the functions of the caecum?

A
  1. Water resorption
  2. Electrolyte resorption (Na, Cl)
  3. Initiates microbial digestion of complex carbohydrates
48
Q

What are the functions of the colon?

A
  1. Microbial digestion
  2. Fluid absorption ~150L per day
49
Q

What can you give to treat for ingestion of sand?

A

Psylluim husks (pellets for prevention)

Pulse therapy! Maintain novel fibre.

50
Q

How do you diagnose nephrosplenic entrapment?

A

Ultrasound - loss of ability to identify the left kidney

Equine Medicine (14 decks)

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