GEP (Life Support) Week 2 Flashcards

1
Q

What is the mediastinum and how is it seperated?

A

The Mediastinum: The middle compartment within your thoracic cavity, between your lungs. It contains the heart and other important structures and is sub-divided into the Superior mediastinum, posterior mediastinum, anterior mediastinum and middle mediastinum.

the superior and posterior mediastinum is seperated by the sternal edge at T4.

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2
Q

What are the boarders and content of the superior mediastinum?

A
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3
Q

What are the boarders and content of the Anterior mediastinum?

A
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4
Q

What are the boarders and content of the Posterior mediastinum?

A
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5
Q

What are the boarders and content of the Middle mediastinum?

A
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6
Q

Identify the anatomy of the heart

A

Atrioventricular Valves:
Tricuspid & Mitral

Semilunar Valves:
Pulmonary & Aortic

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7
Q

Describe the blood blow around the body

A
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8
Q

Conduction system of the heart

A
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9
Q

Describe SA node conduction

A

Phase 0 – Depolarisation
Once threshold of -40 is reached, VGCCs open. Ca influx = faster rate of depolarisation.

Phase 3 – Repolarisation
“Resets” the action potential. K channels open and efflux out of the cell.

Phase 4 – Pacemaker potential
Slow depolarisation of the pacemaker cell. When it reaches the threshold, an action potential is fired.

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10
Q

Describe myocyte conduction part 1

A
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11
Q

Describe myocyte conduction part 2

A
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12
Q

Describe the cardiac cycle and the different points?

A
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13
Q

What controls BP

A

CVS: Heart and blood vessels, Salt and water reabsorbtion
Nervous system: Sympathetic and parasympathetic
Endocrine: RAAS system, ADH, ANP

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14
Q

Name the structures related to the CVS and the receptors involved and whether it is SNS or PNS.

A
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15
Q

What are the 4 ways that SNS controls BP

A

-Chronotropic effect– SA node - Increase the rate of action potentials which increases the HR.
-Inotropic effect– Atrial/ventricular myocytes - Increase in calcium in the myocytes which increases contractility.
-Lusitropic effect - Atrial/ventricular myocytes – Speed up the relaxation of the myocytes
-Dromotropic – AV node - Increased rate of impulse through atria to ventricle to maintain cardiac output

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16
Q

Explain how chronotropic effect occurs?

A

B1 receptors – SA node - Increase the rate of action potentialswhich increases theHR.

17
Q

Explain how Inotropic effect occurs

A

B1 receptors– Atrial/ventricular myocytes - Increase in calcium inthe myocytes which increasescontractility.

18
Q

Explain how lusitopic effect occurs

A

B1 - Atrial/ventricular myocytes – Speed up the relaxationof themyocytes

19
Q

What are beta and aplha agonists and how do they work?

A
20
Q

Describe M2 receptors and parasympathetic mechanism

A

M2 receptors decreases HR by blocking the Na channels

21
Q

Describe the RAAS system

A
22
Q

What are the 3 different type of hypertensions

A

-Primary Hypertension: Pressure in arteries increases with no underlying reason, usually asymptomatic.

-Secondary Hypertension: Hypertension caused by another condition. Symptoms are usually associated with the underlying cause

-Severe hypertension: Severe elevation of arterial blood pressure (over 180/120) that may lead to end-organ damage

23
Q

What are modifiable and non-modifiable factors for hypertension

A

Non-Modifiable
Age
Gender
Race
Menopause
Type 1 Diabetes
**
Modifiable**
Smoking
BMI
Alcohol intake
Stress
Type 2 Diabetes
Sedentary lifestyle
Diet
Fats
Cholesterol
High salt intake

24
Q

What are the cause of secondary hypertension

A

Intrinsic kidney disease
Cushing’s Disease (raised cortisol)
Coarctation of the aorta
Atheromatous renal artery stenosis
Renovascular disease
Primary hyperaldosteronism
NSAID use
Pre/eclampsia

25
Q

What are the potential organ damage from hypertension?

A

Retinopathy
Aneurysms and intercranial bleeding
Coronary artery disease
Left Ventricular Hypertrophy
Atherosclerosis
Renal Failure
(CKD, nephrosclerosis, hypertensive nephropathy)

26
Q

How to diagnose hypertension and the different ranges

A
27
Q

What is the management of hypertension in patients

A
28
Q

Micras of all BP drugs given as management (CCB, ACEi etc)

A
29
Q

What is a 12 lead ECG

A

ECG Lead: A graphical description of the electrical activity of the heart that is made using the different electrodes. There are 12 Leads in a 12 lead ECG and each one represents a different direction

If depolirisation is going towards a lead it will have a positive bump, if its is repolarising towards the lead it will have a negative bump on the ecg.

1 SMALL square (1mm) = 0.04 sec (40ms)
5 SMALL squares (5mm) = 1 LARGE square = 0.2 sec (200ms)
5 LARGE squares = 1 second

30
Q

ST elevation and MI location

A

II/III/AVF = Inferior
I/AVL/V5/V6= Lateral
V1/V2= Septal
V1/V2/V3/V4= Anterior

31
Q

Describe p, qrs and st egment

A
32
Q

What are the common pathology in the ST segment of the ECG

A