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Physiology Exam V > Gastric Motility and Secretions > Flashcards

Gastric Motility and Secretions Flashcards

1
Q

What are the 3 basic movemnts of the stomach

A

storing
churning
emptying

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2
Q

Receptive relaxation

A

vasovagal reflex
initiated by swallowing
mediated by NO

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3
Q

gastric accommination

A

stomach relaxation in response to gastric filling

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4
Q

what is gastric accommodation reglatued by

A

ENS

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5
Q

What hormones impaire gastric emptying

A

secretin, CCK, gastrin

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6
Q

what NT impair gastric emptying

A

Ach, 5-HT

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7
Q

what NT promote gastric empything

A

NO, VIP

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8
Q

what are key neurotransmitters involved in emetic resposne

A

serotonin, dopamine, Ach, histamine

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9
Q

Pyloric stneosis

A

narrowing of pyloric sphincter

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10
Q

what does pyloric stenosis cause

A

impaired motility of stomach which can cause GERD and vomiting

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11
Q

Secretions of parietal cell

A

HCl, intrinsic factor

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12
Q

function of Hcl

A

protein digestion, sterilization, absorption, prevent bacterial/fungal growth, promote bile and pancreatic enzyme flow

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13
Q

function of intrinsic factor

A

B12 absorption

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14
Q

secretion of chief cell

A

pepsinogen

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15
Q

function of pepsinogen

A

protein digestion

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16
Q

secretion of ECL

A

histamine

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17
Q

function of histamine

A

promote HCl secretion

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18
Q

secretion of G cell

A

gastrin

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19
Q

funciton of gastrin

A

promote HCl secretion

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20
Q

secretion of D cell

A

somatostatin

21
Q

functino of somatostatin

A

suppress HCl secretion

22
Q

secretion of nerve cell

A

ACH

23
Q

functino of Ach

A

promote mucus, bicarb, and HCl secretion

24
Q

secretion of superficial epithelial cell

A

mucus, bicarb

25
Q

how does the paracrine pathway regulate acid secretion ins tomach

A

histamine promote acid secretion

prostglandin and somatostatin decrease acid secretion

26
Q

how does endocrine pathway regulate acid secretion in stomach

A

gastrin promotes acid secretion

27
Q

how does neuronal pathway regulate acid secretion in stomach

A

Ach promotes acid secretion

28
Q

what negative feedback affect does somatostatin have

A

inhibits release of gastrin from G cells

29
Q

what indiirectly acts on ECL cells

A

gastrin, Ach

30
Q

what acts on neighboring parietal celsl

A

histamine

31
Q

regulators of interdigestive phase

A

of pariteal cells, body weight, presence of food in stomach

32
Q

what is pH in interdigestive phase

A

3-6

33
Q

what happens during cephalic phase

A

30% of acid secretion

34
Q

regulators of cephalic phase

A

sigh/smell of food triggers vagal response

35
Q

what happens during gastric phase

A

50-60% of gastric secretion

36
Q

regulation of gastric phase

A

distension of stomach, protein products in stomach trigger vagal response

37
Q

what is hapepning during intestinal phase

A

10% of gastric secertion

38
Q

regulators of intestinal phase

A

presence of peptide in duodenum,

absorption of amino acids in duodenum

39
Q

role of mucus in stomach

A

protective factor
lowers pH when you get closer to lumen
forms protective barrier

40
Q

what are 2 key stimuli for mucus secretion

A

Ach, Prostaglandin both increase secretion

41
Q

components of mucus

A

mucin, phosphlipid, electrolyte, water, bicarb

42
Q

Role of secretin

A

decreases acid release from parietal cells
increases pepsinogen release from chief cells
delay gastric emptying

43
Q

What is gastrisis

A

infiltration of inflammatory cells without break in mucosal barrier

44
Q

what can gastritis be caused by

A

NSAIDS, H. pylori

45
Q

H pylori infection

A

bacteria colonize luminal surface of gastric epithelium causing inflammation and ulceration
damage done because of immune system putting up response to H. pylori

46
Q

how is H. pylori protected from stomach acid

A

urease

47
Q

peptic ulcers

A

inflammation that breaks through mucosal lining

can lead to perforation of GI tissue, hemorrhage, cancer

48
Q

zollinger-ellison syndrome

A

high serum gastrin lvels

causes tumor in duodenum/pancreas

Physiology Exam V (14 decks)

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