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Telementry Technician > Final Part 3 > Flashcards

Final Part 3 Flashcards

1
Q

Originates in the ventricles below the bundle of HIS

A

ventricular arrhythmias

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2
Q

occurs when electrical impulses depolarize the myocardium using a different pathway from normal impulses

A

ventricular arrhythmias

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3
Q

the atrium does not depolarizes and atrial kick is lost decreasing CO

A

ventricular arrhythmias

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4
Q

may be benign but are potentially lethal because they are ultimately responsible for CO

A

ventricular arrhythmias

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5
Q

List the Dysrhythmias that orginate in the ventricles

A
  1. PVC - Premature Ventricular Contractions
  2. Ventricular Tachycardia
  3. Ventricular Fibrillation
  4. Ventricular Escapes Complexes and Rhyrhms
  5. Ventricular Asystole
  6. Artificial Pacemaker Rhythm
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6
Q

Explain the Origin of Ventricular arrhythmias

A
  1. Ventricular arrhythmias originate in the ventricles below the bundle of HIS
  2. They occur when electrical impulses depolarize the myocardium, using a different pathway from normal impulses
  3. the atrium does not depolarizes and atrial kick is lost decreasing CO
  4. May be benign but are potentially lethal because they are ultimately responsible for CO
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7
Q

CO

A

cardiac output

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8
Q

single ectopic impulse resulting from an irritable focus in either ventricle

A

The Etiology of Premature Ventricular Contractions

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9
Q

Causes may include myocardial ischemia, increased sympathetic tone, hypoxia, idiopathic causes, acid-base disturbances, electrolyte imbalances, or as a normal variation of the ECG

A

The Etiology of Premature Ventricular Contractions

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10
Q

May occur in patterns such as bigeminy, trigeminy, or quadgeminy, couplets and triplets

A

The Etiology of Premature Ventricular Contractions

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11
Q

occurs when blood flow to your heart is reduced, preventing it from receiving enough oxygen.

A

myocardial ischemia

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12
Q

The reduced blood flow is usually the result of a partial or complete blockage of your heart’s arteries (coronary arteries)

A

myocardial ischemia

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13
Q

the condition of a muscle when the tone is maintained predominately by impulses from the sympathetic nervous system and these impulses have increased`

A

increased sympathetic tone

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14
Q

deficiency in the amount of oxygen reaching the tissues

A

hypoxia

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15
Q

relating to or denoting any disease or condition that arises spontaneously for which the cause is unknown

A

idiopathic causes

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16
Q

acidosis and alkalosis refer to physiologic process that cause accumulation of loss of acid and or alkali; blood pH may or may not be abnormal. Acidemia and alkalemia refer to an abnormally acidic (pH 7045)

A

acid-base disturbances

17
Q

serum concentrations of an electrolyte that are wither higher or lower than normal

A

electrolyte imbalances

18
Q

state the etiology of Premature Ventricular Contraction

A
  1. Single ectopic (occurring in an abnormal position or place; displaced) impulse resulting from an irritable focus in either ventricle
  2. Causes may include hypoxia, electrolyte imbalances, acid-base disturbances, myocardial ischemia, increased sympathetic tone, idiopathic causes, or as a normal variation of ECG
  3. May occur in patterns such as; bigeminy, trigeminy, quadgeminy, couplets, and triplets
19
Q

Malignant PVCs—more htan 6/minute, R on T phenomenon, couplets or runs of ventricular tachycardia, multifocal PVCs, or PVCs associated with chest pain

A

clinical significance of premature ventricular contraction

20
Q

ventricles do not adequately fill, causing decreased cardiac output

A

clinical significance of premature ventricular contractions

21
Q

Explain the clinical significance of Premature Ventricular Contractions

A

Malignangt PVCs
- More than 6 minute, R on T phenomenon, couplets or runs of ventricular tachycardia, multifocal PVCs, or PVCs associated with chest pain
Ventricles do not adequately fill, causing decreased cardiac output

22
Q

Explain treatment of Premature Ventricular Contractions

A
  1. Non-malignant PVCs do not usually require treatment in patients without a cardiac history
  2. Cardiac patient with nonmalignant PVCs administer oxygen and establish IV access
23
Q

Under rules of interpretation what is the explain the rate , rhythm, etc of PVCs

A

PVCs are dysrhythmias that originate in the ventricles. The Rules of interpretation for PVCs are:
Rate —underlying rhythm
Rhythm —interrupts the regular underlying rhythmn
Pacemaker site- ventricle
P waves - none
QRS -0.12 seconds bizzare

24
Q

An extra ventricular contraction consist of

A
  • an abnormally wide and bizarre QRS complex that originates in an ectopic pacemaker in the ventricles
  • it occurs earlier than the expected beat of the unerling rhythm
25
Q

Premature Ventricular Contraction usually is followed by a

A

compensatory pause

26
Q

Why does a full compensatory pause follow a PVC

A

A full compensatory pause follows a PVC because the SA Node is not prematurely depolarized by the PVC.

27
Q

How does the T wave look in a PVC

A

T wave often tall and the opposite direction of QRS

28
Q

What does unifocal PVC’s arise from

A

Unifocal PVC’s arise from the same ectopic pacemaker site and have the same morphology

29
Q

What does multifocal PVC’s arise from

A

Multifocal PVC’s arise from different sites and different morphologies (shapes)

30
Q

Name the frequency and pattern of PVC’s

A 
Infrequent - 5/min
Isolated - occur singly
Group - groups of two or more ,salvos (simultaneous or successive discharge)
Two - paired/couplet
Three >-run of VT
Repetitive- bigeminy, trigeminy
31
Q

An arrhythmia originating in an ectopic pacemaker in the ventricles whose rate is 110-250/min, and the QRS is wide and bizzare

A

Ventricular Tachycardia

32
Q

What are the rules of interpretation of Ventricular Tachycardia

A
  1. Rate - 100-250
  2. Rhythm - usually regular
  3. Pacemaker Site - ventricle
  4. P waves - If present, it is not associated with QRS complex
  5. PRI - none
  6. QRS - >0.12 seconds, bizzare
33
Q

3 or more ventricular complexes in succession at a rate of >100 and may appear monomorphic or polymorphic

A

The Etiology of Ventricular Tachycardia

34
Q

Perfusing patient administer oxygen and establish IV access. Consider immediate synchronized cardio inversion starting at 100 for hemodynamically unstable patients

A

treatment for V tach

Telementry Technician (16 decks)

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