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SRNAHealthAssessment > FINAL ASSESSMENT > Flashcards

FINAL ASSESSMENT Flashcards

1
Q

Difficult intubation

A

External anatomic features
• ↓ Head/neck movement (atlanto-occipitaljoint)
• Jaw movement (temporo-mandibular joint), mouth opening, and subluxation of the mandible
• Receding mandible
• Protruding maxillary incisors
• Obesity

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2
Q

Other Predictors

A 
Thyromental distance
• Sternomental distance
• Visualization of the oropharyngeal
structures
• Anterior tilt of the larynx
• Radiographic assessment
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3
Q

Mouth opening predictors of difficult intubation

A

A mouth opening (distance between incisors) limited to 3.5 cm or less will contribute to difficult intubation

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4
Q

Other predictors of difficult intubation incision

A

Protruding maxillary incisors can interfere with

laryngoscope placement and ETT passage

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5
Q

BAG mask ventilation MOANS

A 
Mask seal
Obese
Age
No teeth
Snores or stiff
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6
Q

LEMON Laryngoscopy and intubation

A 
L: Look externally
➢ E: Evaluate 3-3 (3 fingers between teeth, 3
fingers chin-neck to thyroid notch)
➢ M: Mallampati class
➢ O: Obstruction
➢ N: Neck mobility
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7
Q

Predictors of a Difficult Airway

A 
➢ High Mallampati Classification
➢ Small mouth opening
➢ Prominent Incisors
➢ Thyromental Distance <6 cm
➢ Decreased neck extension
➢ Neck Circumference
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8
Q

Predictors of Difficult Face Mask Ventilation

A 
➢ Age >55 y.o.
➢ BMI >26-30 kg/m2
➢ Beard
➢ Snoring
➢ Lack of teeth
➢ Mallampati III or IV
➢ Limited mandibular protrusion
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9
Q

What is the Single most important predictor for both Difficult mask ventilation and difficult intubation

A

Limited mandibular protrusion

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10
Q

Predictors of Impossible Face Mask Ventilation

MBONM

A 
➢ Male
➢ Beard
➢ Obstructive Sleep Apnea
➢ Mallampatie III or IV
➢ Neck radiation changes
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11
Q

Awake fiberoptic intubation can be performed

A

without atlanto-occipital extension

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12
Q

What can be left in place with awake fiberoptic intubation

A

Any head and neck stabilizing device can be left in place to prevent movement of c-spine

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13
Q

Awake intubation should be the technique of choice when?

A

if there is any reason to believe that maintaining a patent airway after induction of anesthesia may be difficult

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14
Q

Tracheal intubation in patients with an

A

unstable neck should be done with extreme caution.

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15
Q

Avoid movement that can

A

cause spinal cord compression and damage

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16
Q

Most conservative approach when difficult airway is
known or suspected
➢ Be careful

A

Awake intubation
➢ Must explain to the patient and coach through the
procedure
–> with sedatives

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17
Q

Topical anesthesia is the

A

KEY to successful awake intubation

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18
Q

During awake intubation, important to use

A

Important to use glycopyrrolate to dry mucous

membranes prior to topical LA (at least 20 min before)

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19
Q

To numb airway

A

➢ Nebulized LA, LA swish and swallow, LA spray
(hurricane spray), bilateral lingual nerve block, superior
laryngeal nerve block, transtracheal LA injection

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20
Q

Awake vs sleep intubation

A

Consider presence of at least 3 factors predictive of difficult or impossible to mask ventilate

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21
Q

COPD can lead to

A

Possible right-sided failure, cor pulmonale
• Peripheral edema
• Increased hepatojugular reflux

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22
Q

Can lead to cor pulmonale

A

COPD

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23
Q

Chronic Instrinsic pulmonary disorder In late stages, signs

A

signs of right ventricular failure/cor pulmonale

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24
Q

Treat cor pulmonale

A

– diuretics, dig, oxygen

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25
Q

Interpreting pulmonary funcitons: CLASS

A

FEV/FVC = <0.8 (80%)

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26
Q

Restrictive FEV/FVC

A

Both reduced, ratio is normal or high

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27
Q

Obstructive FEV/FVC

A

Low ratio , less than 70%

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28
Q

FEF25–75%:

A

forced expiratory flow over the middle one-half of the

FVC

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29
Q

FEF 25-75% is the

A

Average flow from the point at which 25% of the FVC has been exhaled to the point at which 75% of the FVC has been exhale

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30
Q

VC, TLC, RV in restrictive

A

Decreased

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31
Q

Signs of EARLY asthma attack

A

Alteration of expiratory plateau on capnography

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32
Q

Adventitious lung sounds

A

Crackles (rales)
■ Wheezes
■ Friction rubs
■ Rhonchi

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33
Q

What is crackles

– Late = pneumonia, CHF, atelectasis

A

Series of individual clinking or popping noises in an area

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34
Q

May sound like hairs rubbing together, Velcro, or a

crumpling piece of cellophane

A

Crackles

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35
Q

Sounds are caused by opening of small airways or alveoli that have been collapsed or decreased in volume during expiration because of fluid, inflammatory exudate

A

Crackles

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36
Q

Heard in both phases of resp

A

Crackles

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37
Q

Chronic bronchitis and crackles.

A

– Early inspiratory/expiratory crackles =

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38
Q

Fine vs. coarse
Fine Crackles =
–

A

brief, discontinuous, popping lung sounds that

are high-pitched

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39
Q

– Wood burning in a fireplace

A

Fine crackles

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40
Q

Coarse Crackles =

A

discontinuous, brief, popping lung sounds

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41
Q

Compared to fine crackles , coarse =

A

louder, lower in pitch, last longer

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42
Q

Bubbling sound, rolling strands of hair between fingers near ears?

A

Fine crackles

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43
Q

Can be inspiratory or expiratory

A

Wheezes

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44
Q

■ High-pitched continuous sounds that are generated by airflow through narrowed airways

A

Wheezes

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45
Q

Caused by airflow obstruction from edema, smooth muscle constriction, secretions

A

Wheezes

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46
Q

Low-pitched wheezes

A

– Continuous in both phases

– Snoring, gurgling, rattle-like quality

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47
Q

■ Occur in bronchi!

A

Rhonchu

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48
Q

■ Most commonly used to describe sounds generated by secretions in airways, usually clear after coughing

A

Rhonchi

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49
Q

Can also be used to describe coarse crackles from airway secretions

A

Rhonchi

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50
Q

Sound generated by inflamed or roughened pleural surfaces rubbing against each other during respiration (both phases)

A

Pleural rub

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51
Q

■ Series of creaky or rasping sounds heard during inspiration & expiration

A

Pleural rub

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52
Q

– Sounds like cat purring, walking on fresh snow

A

Pleural rub

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53
Q

Caused by inflammatory disease: pneumonia or pulmonary infarction

A

Pleural rub

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54
Q

– Not cleared by coughing

A

Pleural rub

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55
Q

– Localized, may be transient

A

Pleural rub

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56
Q

Pleural rub stops when_______if it continues.

A

holding breath, if it continues, it might be a pericardial friction rub (pericarditis!)

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57
Q

Stridor mostly

A

Inspiratory

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58
Q

Stridor associated with

A

Epiglottis
Foreign body
Laryngeal edema and croup

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59
Q 
Lung Sounds (B, V, BV) 
– Vesicular
A

– inspiration/expiratory ratio of 3 to 1 or I:E of 3:1

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60
Q

■ Over most of both lungs (V, B, BV)

A

Vesicular

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61
Q

■ Inspiratory sounds last longer then expiratory (V, B, BV)

A

Vesicular

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62
Q

■ NORMAL Soft sound with relatively low pitch (B, V, BV)

A

Vesicular

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63
Q

Mixture of the pitch of the bronchial breath sounds
heard near the trachea and the alveoli with the vesicular
sound (B, V, BV)

A

Bronchovesicular

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64
Q

Inspiratory & expiratory almost equal (V, B, BV)

A

Bronchovesicular

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65
Q

– Medium intensity(B, V, BV)

– Medium pitch

A

Bronchovesicular

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66
Q

– Heard between scapula & in 1st & 2nd interspaces

anteriorly (B, V, BV)

A

Bronchovesicular

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67
Q

Abnormal in the lung periphery and may indicate an

early infiltrate or partial atelectasis (B, V, BV)

A

Bronchovesicular

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68
Q

– Hollow, tubular(B, V, BV)

A

Bronchial

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69
Q

– Expiratory sounds last longer than inspiratory (1:3)(B, V, BV)

A

Bronchial

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70
Q 
Loud intensity (B, V, BV) 
– High pitch
A

Bronchial

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71
Q

Normal over trachea (B, V, BV)

A

Bronchial

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72
Q 
Bronchial breath sounds other than close to the trachea
may indicate (B, V, BV)
A

pneumonia, atelectasis, pleural effusions

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73
Q

Distinct pause between I/E (B, V, BV)

A

Bronchial

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74
Q

Vital Capacity in obstructive

A

Normal or decreased

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75
Q

TLC in obstructive

A

Normal or increased

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76
Q

RV in obstructive

A

Increase

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77
Q

FEV/FVC

A

Decreased

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78
Q

Maximum midexpiratory flow rate

A

Decreased

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79
Q

Maximum breathing capacity

A

Decreased

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80
Q

Primary Survey

A 
 A – Airway with C-spine
 B – Breathing
 C – circulation with hemorrhage control
 D – disability
 E – Exposure / Environment
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81
Q

Secondary Survery

A 
A – allergies
M – medications
P – past medical history
L – last meal
E – events/environment related to injury
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82
Q

Even in smokers with no chronic lung

disease

A

smoking increases carboxyhemoglobin levels, decreases

ciliary function, increases sputum production, stimulates CV system

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83
Q

Smoke-free interval of

A

12-18 hours shows significant declines in carboxyhgb & normalization of oxygen-HGB dissociation curve

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84
Q

CO Hb < 15-20

A

Headache, dizzins and occasional confusion

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85
Q

CO Hb 20-40

A

N/V, disorientation and visual impairment

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86
Q

CO Hb 40-60

A

Agitation, combativeness, hallucinations, coma, and shock

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87
Q

CO Hb > 60

A

Death

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88
Q

Early complications

A 
 Carbon monoxide poisoning
 Airway obstruction
 Pulmonary edema
 1-5 days post-injury
 ARDS
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89
Q

Late complications

A

 Pneumonia
 Atelectasis
 Pulmonary emboli

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90
Q

Tension pneumothorax

A

Immediate threats to life=>Tension pneumothorax

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91
Q

Pneumo and CXR

A

No time for CXR!

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92
Q

Tension PNeumo Treatment:

A

14 gauge angiocatheter at 2nd intercostal midclavicular line or 4th intercostal midaxillary line

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93
Q

Anesthesia Considerations for Burns

A

No easy way to induce – slow & steady

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94
Q

Hypovolemia and/or depleted catecholamines

A

lead to hypotension with all agents

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95
Q

Burns and succ

A

No succinylcholine after 1st 24 hrs, and for up to 2

yrs

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96
Q

Burn pts and NDNMB

A

are resistant to nondepolarizers

Up to 5X normal dose!

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97
Q

Opioid requirements and BURNS

A

increased

98
Q

Regional and

A

is an option unless electric burn

99
Q

Cardiac Tamponade

A

 Beck’s triad
 Electrical alternans
 May need to drain under local before general
induction

100
Q

Beck’s Triad

A

JVD, Hypotension, Muffled heart sounds

101
Q

C-collar

A

Manual in-line stabilization – 2 people
No traction
2nd person will stabilize both shoulders
Remove anterior C-collar

102
Q

C-Collar 1 person

A

1 person stabilizes and aligns head in neutral position

WITHOUT cephalad traction

103
Q

Greater than 1 MAC

A

increase CBF , • Ketamine, > 1 MAC volatiles

104
Q

Normal ICP =

A

5-15 mm Hg

105
Q

ICP waveform Lundberg A wave

A

 “plateau waves”

Worst

106
Q

ICP waveform Lundberg B wave

A

Sharp, brief

107
Q

ICP wavefrom C wave

A

Benign

108
Q

o CBF and CBV

A

1mm Hg increase = 2ml /100g/min increase in CBF

109
Q

o PaO2 below 50 =

A

vasodilation = increase in CBF

110
Q

Increase ICP Meds:

A

mannitol, Lasix, corticosteroids, acetazolamide,

111
Q

Central neurogenic hyperventilation –

A

spontaneous and severe, PaCO2 may decrease to less than 20 mm Hg

112
Q

Related to

A

cerebral thrombosis, embolism, or closed head injury

113
Q

Ataxic breathing (Biot’s breathing)

A

completely random pattern of tidal volumes related to disruption of medullary neural pathways by trauma, hemorrhage, or compression by tumors

114
Q

Apenustic breathing –

A

prolonged end-inspiratory pauses maintained for as long as 30 seconds, related to lesions in the pons or basilar artery infarct

115
Q

Cheyne-Stokes –

A

breaths of progressively increasing and then decreasing tidal volume (crescendo-decrescendo pattern), followed by periods of apnea lasting 15-20 seconds related to basal ganglia or cerebral hemispheres brain injury

116
Q

Central neurogenic hyperventilation –

A

spontaneous and severe, PaCO2 may decrease to less than 20 mm Hg Related to cerebral thrombosis, embolism, or closed head injury

117
Q

Post hyperventilation apnea –

A

awake apnea following moderate PaCO2 decreases related to frontal lobe injury

118
Q

Brain compression and its signs and symptoms

A

(pupils, movement, posturing, etc)

119
Q

• Patients with diffuse brain dysfunction

A

above the level of the diencephalon will react with purposeful or semi purposeful movements toward the painful stimulus

120
Q

• Decorticate responses to pain includes

A

flexion of elbows, adduction of the shoulder, and extension of the knee and ankle = diencephalic dysfunction

121
Q

• Decerebrate responses include

A

extension of the elbow, internal rotation of the forearm, and leg extension = more severe brain dysfunction

122
Q

• No response =

A

pontine or medullary lesions

123
Q

Chiari malformation =

A

displacement of the cerebellum

s/s = HA extending to shoulders/arms, pain w/ coughing/ head movement, syringomyelia

124
Q

Chiari Class 4 –

A

cerebellar hypoplasia, no displacement of posterior fossa content

125
Q

Chiari Class 2 –

A

displacement of cerebellar vermis

126
Q

Chiari Class 1 –

A

displacement of cerebellar tonsil down over cervical spinal cord

127
Q

Chiari Class 3 –

A

displacement of cerebellum into occipital encephalocele

128
Q

Forces that affect cerebral blood flow

A 
CMRO2
ICP			
Drugs
CPP		
PaCO2-Pao2 tension	
Intracranial anomalies 
CO		
SNS and PNS 
Cerebral autoregulation
129
Q

Brain herniation 3 =

A

tonsillar herniation

130
Q

Brain herniation 2 =

A

transtentorial (uncal) herniation

131
Q

Brain herniation 4 =

A

bad day = transcalvarial

132
Q

Valve lesions and their corresponding murmurs

A

MS –Apex, holodiastolic decrescdeno with opening snap , tx – maintain preload, avoid tachy and acidosis

133
Q

MR –

A

pansystolic murmur

134
Q

AS –

A

systolic crescendo decrescendo

135
Q

AR –

A

early diastolic decresncdo murmur

136
Q

PS –

A

crescendo decrescendo, increases with deep inspiration

137
Q

PR –

A

decrescendo diastolic murmur

138
Q

TR –

A

pansystolic highpitched increases with inspiration

139
Q

TS –

A

diastolic

140
Q

Accentuated S1

Mild mitral stenosis

A
  • Shortened PR interval

* Leaflets have less time to drift back together; forced shut from wide distance

141
Q

Accentuated S1 which stenosis

A

Mild mitral stenosis

142
Q

Accentuated S1 and blood flow

A

• Impeded flow → prolonged diastolic pressure gradient → keeps leaflets farther apart during late diastole → loudly forced shut from far apart during systole

143
Q
  • High CO states or tachycardia

* Shortened diastole, less time to drift back together

A

Accentuated S1

144
Q

• Diminished S1

A

• Lengthened PR interval

145
Q

• More time to float back together before systole • Mitral regurgitation

A

Diminished S1

146
Q

Leaflets do not fully come together when they close •

A

Diminished S1

147
Q

Severe mitral stenosis

A

Diminished S1

148
Q

• Higher than normal ventricular pressure at end of diastole → leaflets drift together more rapidly → close from a smaller than normal distance during systole

A

Diminished S1

149
Q

• Leaflets are nearly fixed in position • Stiff left ventricle

A

Diminished S1

150
Q

Abnormalities in S2

Intensity =

A

velocity of blood flow toward valves with sudden arrest by closing valves

151
Q

Diminished S2

A

Severe AS or pulmonic stenosis → valve nearly fixed in position = Dim S2

152
Q

Accentuated S2

A

Systemic HTN/Pulmonary artery HTN → greater diastolic pressure → increased velocity of blood flow

153
Q

Abnormal splitting patterns of S2: A2 and P2

A

widened, fixed, paradoxical • Widened

Increased time interval between A2 and P2; noticeable even during expiration and becomes wider on inspiration

154
Q

Abnormal splitting patterns of S2

A

Delayed closure of pulmonic valve (RBBB)

155
Q

Occur shortly after S1 and coincide with opening of aortic/pulmonic valves

A

Ejection clicks

156
Q

Presence of aortic/pulmonic valve stenosis or dilatation of pulmonary artery or aorta Sharp, high-pitched

A

Ejection clicks

157
Q

Mid/late extra systolic heart sounds

A

Usually from systolic prolapse of mitral or tricuspid valves

158
Q

Ejection Click: Leaflets bulge abnormally from

A

ventricular side of the AV junction into the atrium during ventricular contraction

159
Q

Commonly associated with valvular regurgitation

A

Extra diastolic heart sounds: opening snap (OS), third heart sound (S3), fourth heart sound (S4), pericardial knock

160
Q

Commonly associated with valvular regurgitation

A

Extra diastolic heart sounds: opening snap (OS), third heart sound (S3), fourth heart sound (S4), pericardial knock

161
Q

early diastole following opening of AV valves during ventricular filling due to tensing of chordae tendineae during rapid filling/expansion of ventricle

A

S3 (ventricular gallop

162
Q

Normal in children and young adults, Suggests dilated ventricle (heart failure) or advanced mitral/tricuspid regurgitation in middle- aged/older

A

S3

163
Q

occurs late in diastole coinciding with contraction of the atria and flow of blood into a stiffened ventricle (CAD)

A

S4 (atrial gallop)=

164
Q

S4, S1, S2, S3 Tachycardia →

A

shortened duration of diastole, S3 and S4 coalesce = summation gallop

165
Q

Pericardial knock

A

Seen in severe constrictive pericarditis
Appears early in diastole after S2
High-pitched

166
Q

Quadruple rhythm/summation gallop

A

S4 (atrial gallop)=

167
Q

Eye opening response

A 
4 = spontaneous
3 = to verbal command,speech shout
2 = to pain (not applied to face)
1 = NO eye openin g
168
Q

Verbal response

A

5= Oriented
4= Confused converstation, but able to answer questions
3= Inappropriate responses, words discernable
2= Incomprehensive sounds or speech
1=NO verbal response

169
Q

3 areas Glasgow Coma scale measure

A

Eye opening
Verbal response
Motor response

170
Q

Motor Response

A

6=obeys commands for movement
5= purposeful movement to painful stimulus
4=Withdraws from pain
3= Abnormal flexion, decorticate posture2=
2= Extensor rigid response, decerebrate posture
1= NO motor response

171
Q

Trunk burn %

A

9%

172
Q

Back burn %

A

9%

173
Q

Legs each burn %

A

9%

174
Q

Arms each back and front burn %

A

4%

175
Q

Head front and back burn %

A

4 1/2%

176
Q

Penis or genital burn %

A

1%

177
Q

Right axis Deviation RALeftPO

A

Right axis deviation
Acute R heart strain
Left Posterior Fascicular Block

178
Q

Left Axis Deviation

A

Inferior wall MI
Left anterior fascicular Block
Left axis deviation

179
Q

Ataxic Biot breathing location

A

MEdulla

180
Q

Pattern of ataxic

A

Unpredictable sequence of breaths varying in rate and tidal volume

181
Q

Apneustic breathing location

A

Pons

182
Q

Pattern of Apneustic

A

Gasps and prolonged pauses at full inspiration

183
Q

Cheynes stokes breathing

A

Cyclic crescendo-decrescendo tidal volume pattern interrupted by apnea

184
Q

Cheynes stokes breathing pattern

A

Cerebral hemispheres

185
Q

Cheynes stokes breathing what conditions

A

CHF

186
Q

Central neurogenic hyperventilation Pattern

A

Marked hyperventilation

187
Q

Site of lesion/conditions with neurogenic hyperventilation pattern

A

Cerebral thrombolism

Embolism

188
Q

Post hyperventilation apnea breathing

A

Awake apnea following moderate decreases in PaCo2

189
Q

Location of post hyperventilation apnea

A

Frontal lobes

190
Q

Region of compression : Diencephalon

pupillary examination

A

Small pupils ;reactive to light

191
Q

Region of compression : Diencephalon

response to oculocephalic or cold caloric testing

A

normal

192
Q

Region of compression : Diencephalon

Gross motor findings

A

Purposeful semi-purposeful or DECORTICATE (flexor ) posturing

193
Q

Region of compression : Midbrain

Pupillary examination

A

Midsize pupils; UNREACTIVE To light

194
Q

Region of compression :Midbrain

response to oculocephalic or cold caloric testing

A

May be impaired

195
Q

Region of compression : midbrain

Gross motor findings

A

Decerebrate (extensor) posturing

196
Q

Region of compression : PONS or MEDULLA OBLONGATA

Pupillary examination

A

Midsize pupils: UNREACTIVE TO LIGHT

197
Q

Region of compression : PONS or MEDULLA OBLONGATA

response to oculocephalic or cold caloric testing

A

Absent

198
Q

Region of compression : PONS or MEDULLA OBLONGATA

Gross motor findings

A

No response

199
Q

Systolic ejection murmurs associated with

A

Aortic stenosis

Pulmonic Stenosis

200
Q

Aortic stenosis murmur characteristics

A

Radiates to the neck

201
Q

Pansystolic mumur is associated with

A

MR and TR

202
Q

Mitral Regurgitation murmur characteristics

A

Location at apex and radiates to Axilla

203
Q

Tricuspid Regurgiation murmur characteristics

A

Left Lower sternal border–> R Lower sternal border

204
Q

Late systolic associated with

A

MVP

205
Q

MVP murmur at

A

Apex radiates to Axilla

206
Q

Early diastolic murmur are

A

Aortic Regurgitation

Pulmonic Regurgitation

207
Q

Mid to late systolic

A

Mitral stenosis

208
Q

Mitral stenosis heard at

A

Apex

209
Q

CVA risk factors : Systemic Hypoperfusion

A

Hypotension
Hemorrhage
Cardiac arrest

210
Q

CVA risk factors : Embolism and Thromboembolism

MIS PDW

A 
Male gender
Ischemic heart disease
Smoking
Peripheral vascular disease
Diabetes Mellitus
White race
211
Q

CVA risk factors : Subarachnoid hemorrhage and Intracerebral hemorrhage

A 
Often none
HTN
Coagulopathy
Drugs
Trauma
212
Q

‘OFTEN NONE” risk factors

A

SAH

213
Q

Early phase of burn CV System

A

Increase HR,Increase PVR and SVR
Decrease CI
Decrease SV
Decrease contractility

214
Q

LATE phase of burn CV System

A 
Increase HR, Increase CI
Normal or Increase SV
Decrease SV
Decrease contractility
Decreased SO2
215
Q

Early Phase of burn: Blood

A

Increase hematocrit

216
Q

Late Phase of burn: Blood

A

Decrease Hematocrit

217
Q

Early Phase of burn: Lungs

A

Pulmonary Edmea
Bronchospasm
Bronchorrhea

218
Q

Late Phase of burn: Lungs

A

Pneumonia
ARDS
Atelectasis

219
Q

Early Phase of burn: Kidneys

A

Myoglobinuria
Oliguria
Fena <1%

220
Q

Late Phase of burn: Kidneys

A

Increase GFR

Decrease Tubular Function

221
Q

Both Early and LATE Phase of burn: Brain

A

Possible Cerebral Edema
Increase Pain response
Altered mental status

222
Q

Early Phase of Burn : Endocrine and metabolic function

A

Increase aldosterone and Cortisol

223
Q

Late Phase of Burn : Endocrine and metabolic function

A

Increase insulin resistance
Increase O2 consumption and Co2 production
Muscle catabolism

224
Q

Left axis deviation is

A

< -30 degrees

225
Q

Moderate left axis deviation

A

30-45 degrees

226
Q

Marked degrees

A

-45 - (-90 degrees)

227
Q

Broached Notched R or slurred R waves in lead

A

LBBI, aVl, V5 and V6

Occasional rS pattern in V5 and V6 or V1

228
Q

Right Axis deviation

A

>

  • 90
229
Q

Right axis deviation moderate

A

90-120 degrees

230
Q

Right axis Marked

A

120-180 degrees

231
Q

Rhythm common under anesthesia

A

Junctinal escape rhythm

232
Q

V3, V4

A

Anterior LAD

233
Q

V1, V2

A

Septal LAD

234
Q

II, III, aVF

A

Inferior RCA

235
Q

I, aVL, V5, V6

A

Lateral Circumflex only V6 is just lateral

236
Q

Axis when Lead I and AvF upright

A

normal axis

237
Q

Axis when lead I up and avF down

A

Left axis

238
Q

Axis when lead I Down, and AvF up

A

Right Axis

239
Q

Predictors of difficult video laryngoscopy

A
  • Scarring
  • Radiation
  • Masses
  • Large neck circumference
  • TMD < 6 cm
  • Limited neck mobility
  • Operator experience
240
Q

Brain Herniation 1

A

Subfalcine (cingulate) herniation

SRNAHealthAssessment (20 decks)

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