Eye posterior segment Flashcards

1
Q

describe what happens to vitreous with age

A

collagen forms bundles. Floaters, flashes, hemorrhage, curtain/veil as vitreous detaches from retina

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2
Q

arterial supply of eye

A

internal carotid branches to ophthalmic a branches into central retinal (no redundancy) and ciliary arteries (choroidal, lots of redundancy).

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3
Q

how do you test for vascular obstruction

A

flourescein dye injection (arteries in 10 seconds, veins in another 5 seconds)

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4
Q

where do photoreceptors and RPE cells get blood flow from?

A

they are avascular. supply comes from choroidal circulation

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5
Q

artery vs vein occlusion

A

artery=cherry red spot (opacification). vein=hemorrhage

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6
Q

in what diseases do we see neovascularization

A

prolific diabetic retinopathy=retinal

wet macular degeneration=choroidal

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7
Q

NPDR: when is it seen? characteristic findings?

A

early DR. No neovascularization. Microaneurysms, cotton wool spots. Increased vasc. permeability leads to edema and lipid deposits (hard exudates)

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8
Q

PDR: when is it seen? what can it lead to? tx?

A

later stage DR. neovascularization of disc and elsewhere. Can lead to hemorrhage. tx: anti-VEGF and laser burn outer retina

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9
Q

what is the macula used for

A

reading and seeing faces

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10
Q

progression of ARMD. tx?

A

dry (non-exudative, non-neovascular) then wet. Anti-VEGF is tx

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11
Q

what are the four descriptors of the optic nerve

A

cup, color, contour, margins

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12
Q

blood supply to optic nerve

A

mostly by posterior ciliary arteries

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13
Q

optic neuritis is often linked to

A

MS

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14
Q

papilledema: pathophys? complaints?

A

bilateral disc swelling secondary to increased intra-cranial pressure. Transient (seconds) vision loss. NO APD b/c its bilateral

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15
Q

Giant cell arteritis: pathology? complications?

A

intimal proliferation leading to obstruction of CRA

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