Extradural haemorrhage (A&E) Flashcards

1
Q

Define extradural haemorrhage.

A

Bleeding between skull & dura mater

AKA epidural haemorrhage

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2
Q

Which demographic does extradural haemorrhage usually happen in? (2)

A
  • M>F
  • 20-30y
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3
Q

What usually causes extradural haemorrhage, and where?

A
  • usually caused by ‘low-impact trauma’ - head injury (blow to the head, fall)
  • most commonly an arterial bleed - rupture of middle meningeal artery
  • pterion is common site of rupture - thinnest part of skull where middle meningeal artery lies
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4
Q

What can extradural haemorrhage lead to? (2)

A
  • expanding haematoma
    • –> uncus of temporal lobe herniates around the tentorium cerebelli and patient develops a fixed and dilated pupil due to compression of the parasympathetic fibres of CN III
  • brain herniation (coning)
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5
Q

What is the classic presentation of extradural haemorrhage?

A

Presents with sudden onset soon after injury following a brief lucid interval:

  1. initial loss of consciousness after head injury
  2. temporary consciousness recovery + return to normal neurological function (lucid interval)
  3. neurological status declines again - due to haematoma expansion
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6
Q

What might we see on examination of extradural haemorrhage? (5)

A
  • contralateral neurological deficits
  • headache
  • reduced GCS –> confusion
  • fixed and dilated pupils
  • Cushing’s reflex - hypertension and bradycardia
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7
Q

Where are neurological deficits seen in extradural haemorrhage?

A

Contralaterally

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8
Q

What signs of raised ICP can we see in extradural haemorrhage? (2)

A
  • headache
  • confusion (decreased GCS)
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9
Q

What nerve can be compressed in extradural haemorrhage and what can we see?

A

Compression of parasympathetic fibres of CN III –> fixed, dilated pupils

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10
Q

What is the Cushing’s reflex in extradural haemorrhage?

A

Hypertension and bradycardia

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11
Q

What are some risk factors for extradural haemorrhage? (3)

A
  • low-impact trauma
  • assault
  • sporting injury
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12
Q

What is the first-line investigation for extradural haemorrhage?

A

Non-contrast head CT

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13
Q

What would you see on non-contrast head CT in extradural haemorrhage?

A
  • hyperdense (bright) biconvex lesion (lemon-shaped) limited by suture lines of skull
    • think EPIdural = PIE = LEMON
  • 95% of cases are supratentorial, of which 60% are in temporoparietal region
  • check for secondary features of mass effect on CT scan e.g. midline shift or subfalcine/uncal herniation - may require urgent neurosurgical intervention
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14
Q

What are some differential diagnoses for extradural haemorrhage? (7)

A
  • subdural haemorrhage
  • subarachnoid haemorrhage
  • intracerebral haemorrhage
  • delirium
  • meningitis
  • intracranial mass
  • migraine
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15
Q

What is the management plan for extradural haemorrhage? (6)

A
  • conservative monitoring
  • BP reduction - IV mannitol
  • severe: burr holes to evacuate haematoma + craniotomy
  • stop anticoagulation/antiplatelets
  • can give reversal agents
  • prophylactic anticonvulsants
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16
Q

Why is extradural haemorrhage an emergency?

A

Haematoma expansion can lead to brain herniation and death

17
Q

What is the definitive treatment for extradural haemorrhage?

A

Craniotomy and haematoma evacuation (burr holes)

18
Q

What do we do with extradural haemorrhage patients who have no neurological deficit?

A

Cautious clinical and radiological observation

19
Q

What else do we need to manage alongside extradural haemorrhage?

A

ICP

20
Q

How do we prevent haematoma expansion in extradural haemorrhage?

A

Anticoagulant reversal

21
Q

What are some complications of extradural haemorrhage? (3)

A
  • herniation of the brain and permanent coma
  • normal pressure hydrocephalus –> weakness, headaches, incontinence, difficulty walking
  • paralysis or loss of sensation (which began at time of injury)
22
Q

Describe the prognosis of extradural haemorrhage.

A

In patients with no other associated brain injury, early decompression is associated with good neurological outcomes, including full recovery