Exam 3 Flashcards

1
Q

Upper airway consists of? U-PLNTS

A

Nasal cavities, sinuses, pharynx, tonsils, larynx

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2
Q

Lower airway consists of? TBB

A

Trachea, bronchi, bronchioles

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3
Q

Terminal alveoli consist of?

A

Air sacs and primary lobules

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4
Q

Ventilation and patho

A

Ability to move air in and out of the lungs via a pressure gradient

Airways, lungs, chest wall, diaphragm

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5
Q

Respiration and patho

A

Gas exchange of O2 and CO2 to blood and body tissues

Lungs, CV system, and peripheral tissues

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6
Q

Nerves that innervate the diaphragm

A

C3,4,5 stay alive!

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7
Q

Forced inspiration uses what muscles?

A

sternocleidomastoid, scalenes, serratus anterior, pec minor

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8
Q

Forced expiration uses what muscles?

A

Intercostals, abdominals

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9
Q

Primary difference between hypoxia and hypoxemia

A

Hypoxia is low o2 in body tissues

Hypoxemia is low o2 in the blood and can lead to hypoxia

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10
Q

Causes for hypoxia (VDHAP)

A

Ventilation/perfusion mismatch (asthma)
Decreased O2 content (high altitude)
Hypoventillation (drug overdose)
Alveolocapiliary perfusion abnormality (edema)
Pulmonary shunting (atelectasis - collapse of alveoli)

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11
Q

Cyanosis and O2 sat

A

Central O2 sat is decreased - typically caused by pulmonary shunting or respiratory disease - bluish discoloration in mucous membranes, mouth - depends on O2 sat and # circulating hemoglobin

Peripheral cyanosis O2 sat not necessarily decreased - can be caused by fear/anxiety, cold temps, heart failure or shock - due to decreased perfusion to extremities - vasoconstriction w/ decreased blood supply and perfusion

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12
Q

S&S of respiratory disease

A

Cough - productive cough w purulent sputum = infection, nonpurulent = airway irritation
Dyspnea - SOB usually indicative of hypoxemia but could be anxiety etc.
Chest pain - localized pain, pleural irritation
Cyanosis - central or peripheral
Clubbing of fingers - different from PC
Altered breathing patterns

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13
Q

Breathing patterns - apneustic

A

Gasping inspiration followed by short expiration

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14
Q

Breathing patterns - biots respiration

A

Fast deep breaths interspersed with abrupt pauses

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15
Q

Breathing patterns - Cheyne-Stokes

A

Pattern of deep breathing followed by shallow breathing or pauses in breathing

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16
Q

Breathing pattern - Crackles/rales

A

Low pitched popping, secretions in peripheral airways —> atelectasis - collapse of alveoli

17
Q

Breathing pattern - Kussmaul’s respiration

A

Distressing Dyspnea w increased respiration rate, depth of respiration (air hungry)

18
Q

Breathing patterns - lateral-costal breathing

A

Anterior chest flattening, excessive flaring of lower ribs.

19
Q

Breathing patterns - paradoxical

A

All or part of chest wall falls during inspiration, abdominal may expand during expiration

20
Q

Breathing pattern - stridor

A

Shrill, harsh should during inspiration - laryngeal obstruction (choking sound)

21
Q

Breathing pattern - wheezing

A

High pitched whistling sound w expiration —> constriction of airways

22
Q

Pathogenesis for cystic fibrosis and treatment

A

Most common inherited disease in caucasians
Blocking of Cl channel keeps Cl, Na, H2O inside —> thick mucous affects hepatic (liver), digestive, reproductive, and respiratory systems

Mucous affects lungs - cough, wheezing, repeated lung infection (pneumonia),
Digestive - bulky, foul stools - difficulty passing them
Salty skin/sweat

Active cycle of breathing technique (ACBT)
Airway clearance techniques (ACT)
Postural drainage
Positive expiratory pressure therapy (PEP)
Physical exercise may improve lung function reduce effect of thick mucous
CF vest - high-frequency cheat wall oscillation

23
Q

Atelectasis pathogenesis and PT treatment

A

Collapse of alveoli commonly related to immobility/shallow breathing (inpatient settings where patients are less mobile)

Spirometry, autosplinting, deep breathing, exercise

24
Q

Neonatal resp disease

A

Respiratory system is the last to develop, lack surfactin production

Rapid, shallow breathing

25
Pneumoconiosis - obstructive or restrictive? Examples?
Restrictive Silicosis - mesothelioma- cancer Black lung dz - scarring 50% lung vital capacity Asbestosis - mesothelioma
26
Most common COPD. Conditions
Bronchitis - excess mucous, hyperplasia of glands, central cyanosis, pulmonary edema Emphysema - destruction of elastic fibers causes alveoli to expand and not function properly, ORTHOPNEA, barrel chest Asthma - bronchoconstriction
27
Pleural effusion types
Hydrothorax - water in pleural space Hemothorax - blood in pleural space (ex TB) Empyema - infection w fibrosis Pyothorax - pus in pleural space due to infection
28
Hyperglycemia - 3P’s
Polydipsia - excessive thirst Polyphagia - excessive hunger Polyuria - frequent urination
29
Low Ca levels stimulate
Release of Ca from bone Ca reuptake in gut Ca resorption from kidney
30
Ca affected by what 2 hormones?
Parathyroid hormone (PTH) - increases Ca levels Calcitonin - Decreases Ca levels