Exam 2 pulmonary part 2 Flashcards

1
Q

Pathophysiology of Asthma

A

Intermittent and reversible airflow obstruction affecting airways only, not alveoli

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2
Q

Etiology of Asthma

A
  • Inflammation:
    Caused by cold air, dry air, specific allergens, general irritants, microorganisms, aspirin
  • Hyper-responsiveness:
    caused by exercise, URI, GERD, unknown reasons
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3
Q

Asthma severity: Mild Intermittent- Class I

A
  • Sx less than twice a week
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4
Q

Asthma severity: Mild Persistent- Class II

A

Sx more than twice a week, but not daily

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5
Q

Asthma severity: Moderate Persistent- Class III

A

Daily sx, with exacerbations twice a week

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6
Q

Asthma severity: Severe Persistent- Class IV

A

Sx occur continually with frequent exacerbations

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7
Q

Drug therapy for Asthma (Not the specific names): Preventive therapy (controller drugs)

A
  • Change airway responsiveness to prevent asthma attacks

- Used every day, regardless of symptoms

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8
Q

Drug therapy for Asthma (Not the specific names): Rescue Drugs

A

Actually stop attack once it has started

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9
Q

Albuterol; Salmetrol

A
  • Short- and long-acting beta2 agonists
  • Bronchodilator
  • Main rescue inhaler (Albuterol)
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10
Q

Ipratroprium bromide

A
  • Cholinergic antagonists

- Bronchodilator

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11
Q

theophylline

A
  • Methylxanthines

- Bronchodilator

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12
Q

Fluticasone, prednisone

A
  • Corticosteroids

- Anti-inflammatory agents

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13
Q

nedocromil

A
  • Cromones

- Anti-inflammatory agents

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14
Q

montelukast

A
  • Leukotriene modifiers

- Anti-inflammatory agents

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15
Q

Status Asthmaticus

A

Severe, life-threatening, acute episode of airway obstruction

Intensifies once it begins, often does not respond to common therapy

Patient can develop pneumothorax and cardiac/respiratory arrest

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16
Q

Chronic Obstructive Pulmonary Disease (COPD)

A
  • Characterized by bronchospasm and dyspnea

- Tissue damage not reversible; increases in severity, eventually leads to respiratory failure

17
Q

Emphysema

A
  • Loss of lung elasticity and hyperinflation of lung
  • Dyspnea
    need for increased respiratory rate
  • Air trapping
    caused by loss of elastic recoil in alveolar walls, overstretching and enlargement of alveoli into bullae, collapse of small airways (bronchioles)
18
Q

Chronic Bronchitis

A

Inflammation of bronchi and bronchioles caused by chronic exposure to irritants, especially cigarette smoke

19
Q

Chronic Bronchitis symptoms

A
  • Inflammation, vasodilation, congestion, mucosal edema, bronchospasm
  • Affects only airways, not alveoli
  • Production of large amounts of thick mucus
20
Q

Chronic Obstructive Pulmonary Disease (COPD) etiology

A
  • Cigarette Smoke
    First, second & third hand smoke
  • Advanced Age
  • Alpha1-antitrypsin (AAT) deficiency (important)
  • Exposure to air pollution
21
Q

Chronic Obstructive Pulmonary Disease (COPD) complications

A
  • Hypoxemia/tissue anoxia
  • Acidosis
  • Respiratory infections
  • Cardiac failure, especially cor pulmonale
  • Cardiac dysrhythmias
22
Q

Laboratory Assessment of COPD

A
  • ABG values for abnormal oxygenation, ventilation, acid-base status
  • Sputum samples
  • CBC
  • Hemoglobin and hematocrit (up)
  • Serum electrolytes
  • Serum AAT
  • Chest x-ray
  • Pulmonary function test
23
Q

COPD interventions

A
  • Improve oxygenation and reduce carbon dioxide retention
  • Prevent weight loss
  • Minimize anxiety
  • Improve activity tolerance
  • Prevent respiratory infection
24
Q

COPD drug therapy

A
  • Beta-adenergic agents
  • Cholinergic antagonists
  • Methylxanthines
  • Corticosteroids
  • NSAIDs
  • Mucolytics
25
Q

Chest tube Chambers: Chamber 1

A

collects fluid draining from patient

26
Q

Chest tube Chambers: Chamber 2

A

water seal prevents air from re-entering patient’s pleural space

27
Q

Chest tube Chambers: Chamber 3

A

suction control of system