Exam 1-Thermal Relations Flashcards

1
Q

conduction

A

heat transfer between two objects (solids)

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2
Q

convection

A

heat transfer between solid and surrounding air

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3
Q

evaporation

A

heat causing H2O to go into gas phase

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4
Q

radiation

A

heat transfer

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5
Q

endothermy

A

heat is internally produced

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6
Q

ectothermy

A

refers to the influence of the environment on the internal body temperature of an animal

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7
Q

poikilothermy

A

refers to the ability of an animal to survive at a variety of internal body temperatures

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8
Q

eurythermal

A

refers to poikilotherms who can function over a wide range of internal and external temperatures

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9
Q

stenothermal

A

refers to poikilotherms who can function over a narrow range of internal and external temperatures

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10
Q

behavioral thermoregulation

poikilotherms

A

maintenance of a relatively constant internal temperature via certain behaviors

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11
Q

behavioral thermoregulation

homeotherms

A

the use of behavior to assist in the maintenance of a relatively constant internal temperature

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12
Q

expanding thermoneutral zone

A
  • in order to not have to increase metab.
  • insulation: make heat transfer more difficult [fat, pilomotor (fur), ptilomotor (feathers)]
  • vasomotor: blood vessels (hot-exposed, dilated at skin; cold-less exposed, constricted)
  • postural: taking up space (hot) vs curling up (cold)
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13
Q

Thermoreceptors

A

cold: phasic, 12-35oC
warm: phasic, 25-4oC
* mammals respond

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14
Q

(regulation of temperature)

hypothalamus

A
  • controls much of homeostasis
  • input from peripheral temp. receptors
  • directly detects temp.
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15
Q

(regulation of temperature)

posterior region

A
  • cold response: shiver (muscle contractions), restrict blood vessels, hair stands up, insulate, change environ., posture change)
  • damage: won’t be able to sense cold or do anything about it
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16
Q

(regulation of temperature)

anterior region

A
  • warmth response: blood vessels dilate, sweat, change posture, change environment, lower metabolism, less active
  • damage: can’t sense when we’ve gone above set-point, can’t make changes (always making heat, difficult to cool down)
17
Q

performance curve

A

-pejus range: things go to pieces (avalanche) doubtful that the individual will survive (enzymes moving so fast that they’re not really binding)

18
Q

chronic physiological response

A
  • poikilotherm’s recent thermal history will determine its acute response to temp. changes
  • acclimatization
  • acclimation
19
Q

chronic metabolism-temperature curve

A

if change is less extreme, less stress is placed upon the system (able to make changes in the system)

20
Q

fever

ectotherms and poikilotherms

A

-cold response (deliberately increasing temp.-posterior)
-increasing set-point by pyrogens (body feels cold until reaching set-point-get shivers and put layers on)
when fever breaks-sweaty

21
Q

why fevers?

A
  • invaded cells likely to die
  • reduces Fe (required by bacteria)
  • increasing enzyme activity (somewhat)
22
Q

poikilothermic response to high temp.

A

avoidance: shade, dens, circadian rhythm

23
Q

(poikilothermic response to freezing temp.)

production of antifreeze compounds

A
  • colligative (concentration higher can survive lower temp.) (invertebrates)
  • noncolligative: glycoproteins (prevent crystals)
24
Q

(poikilothermic response to freezing temp.)

supercooling

A
  • cooling of a solution below its freeing point without ice formation
  • ice-nucleating agents: particles that can act as a foci for freezing initiation (ice needs a substance to crystallize on)-(used by shallow fish)
25
Q

(poikilothermic response to freezing temp.)

tolerance of freezing

A

ice (pure H2O) forms in tissues around cells increasing conc. of solutes around ice (hypertonic); then solutes move into cell—decreases freezing point

26
Q

core body temperature

A

-has much tighter regulation than peripheral body temp.
37oC
-circadian rhythm: increase of 1.5-2oC during active phase
-in TNZ (changes won’t affect internal)

27
Q

responding to temp. above TNZ

A

-active evaporation cooling: increased heat loss via accelerated water evaporation (sweating, pantin, gular fluttering)

28
Q

(responding to temp. above TNZ)

sweating

A
  • increase cutaneous evaporation by 50 fold
  • Na, Cl loss
  • humans, horses, camels, kangaroos
29
Q

(responding to temp. above TNZ)

panting

A
  • increased breathing rate in response to heat stress
  • blood CO2 (less in blood when taking in more new air than old air-messes with pH and breathing desire)
  • breathing patterns
30
Q

(responding to temp. above TNZ)

gular fluttering

A

-rapid up/down oscillation of mouth, cavity floor to enhance rate of evaporation cooling by increasing air flow over moist membranes in mouth

31
Q

responding to temp. below TNZ

A
  • thermogenic mechanism: used to deliberately generate heat (shivering and nonshivering)
  • heat conservation (regional heterothermy and countercurrent heat exchange)
32
Q

(responding to temp. below TNZ)

nonshivering

A

-brown fat: in young, metabolically active, uncoupling of oxidative phosphorylation (instead of using energy to make ATP its used to generate heat)

33
Q

(responding to temp. below TNZ)

shivering

A
  • high frequency, unsynchronized (constant forming/breaking bonds) contractions and relaxations of skeletal-muscle motor units
  • increases heat production 4-5x resting levels
34
Q

(responding to temp. below TNZ)

regional heterothermy

A
  • certain regions of the body exhibit different thermal relations than other regions
  • wider temp. range (usually appendages)
35
Q

(responding to temp. below TNZ)

concurrent heat exchange

A

normally: loosing heat as leaves core
concurrent: blood next to each other, as heat given off its absorbed by next door blood vessel (requires flow of fluids in opposite directions to help maintain a heat gradient)