Equine Flashcards
Strangles agent
Streptococcus equi; gram-positive bacteria that is not normal flora
Age strangles tends to involve
Tend to be young (2-5 years old)
Clinical signs of strangles
Lymphadenopathy, fever, inappetence, lethargy, serous to mucopurulent nasal discharge
What lymph nodes does strangles affect?
Intermandibular lymph nodes and retropharyngeal lymph nodes
Bastard strangles - internal lymph nodes
(T/F): You should treat a strangles case with current abscesses with penicillin
False - should only treat with penicillin before abscesses occurs or if exposed
Complications of strangles (4)
- Internal abscessation –> bastard strangles
- Purpura hemorrhagica –> vasculitis that results in pitting edema of limbs and head, petechiation
- Guttural pouch EMPYEMA –> serves as chronic carriers
- Septicemia/encephalitis
West Nile Virus Clinical Signs
Depressed mental state, ataxia, weakness, muscle fasciculations (especially muzzle), fever, recumbency
Togavirus Encephalitis (EEE, WEE, VEE) Clinical Signs
Fever, anorexia, depression, somnolence (sleeping sickness) to hyperesthesia, cp deficits, recumbency, cerebral/cranial nerve signs (head pressing, propulsive walking, circling, head tilt)
Structures the palmar digital block desensitizes
Caudal region of foot, coffin joint except proximodorsal region
Structures the abaxial sesamoid block desensitizes
Proximal P1 and below (pastern and coffin joint, partial fetlock)
Structures Low 4-Point block desensitizes
The fetlock joint (metacarpophalangeal) joint and below
Structures High 4-Point block desensitizes
Proximal metacarpal region and below
3 things that need to happen for neonatal isoerythrolysis to occur
- Neonatal RBC antigen inherited from sire that mare doesn’t express
- Mare doesn’t express neonatal RBC antigen but has developed maternal antibodies to the specific neonatal RBC antigen
- Neonatal ingestion of colostrum
Type of immune reaction of neonatal isoerythrolysis
Type 2 hypersensitivity reaction
Why do you never give a horse Vitamin K?
Nephrotoxic