Elimination Flashcards

1
Q

Melena

A

black stools d/t gi bleeding

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2
Q

Hematochezia

A

red blood in stool

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3
Q

Normal Bristol stool

A

3-4

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4
Q

Constipated Bristol stool

A

1-2

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5
Q

Diarrhea Bristol stool

A

5-7

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6
Q

Types of Incontinence

A
Urge
Stress
Overflow
Functional 
Mixed
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7
Q

Location of Micturition Reflex

A

Sacral region

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8
Q

Location of Sympathetic Nerves

A

Thoracolumbar

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9
Q

Location of Parasympathetic Nerves

A

Cranial/Cervical, Sacral

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10
Q

Definition & Causes of Urge Incontinence

A

sudden, intense need to void

neurogenic bladder

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11
Q

Definition & Cause of Stress Incontinence

A

small increase in intraabdominal pressure –> voiding
multiple vaginal births
weak pelvic floor muscles

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12
Q

Definition & Causes of Overflow Incontinence

A

overdistended bladder leads to small leakage throughout the day
benign prostatic hyperplasia

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13
Q

Functional Incontinence

A

caused by a functional impairment that prevents someone getting to the bathroom in time

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14
Q

Bladder anatomy

A

body = stores urine
neck = continuous with urethra
trigone area = triangular area of ureteral openings & urethra

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15
Q

Parasympathetic receptors

A

muscarinic (M)

nicotinic (N)

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16
Q

N receptors

A

located in the synapses of SNS/PNS neurons at the motor end plate of striated muscle fibers @ the external sphincter or pelvic floor

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17
Q

M receptors

A

located in the post-ganglionic parasympathetic endings of detrusor muscle

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18
Q

A-1 receptors

A

sympathetic receptor located in the trigone area

contraction = prevent bladder emptying

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19
Q

B-2 receptor

A

located in the detrusor muscle

increase relaxation = increase storage capacity

20
Q

Vesicoutereal reflux

A

when urine backflows from bladder to kidneys via the ureters

21
Q

Ureter anatomy

A

do not have valves

enter the bladder posteriorly @ an oblique angle. bladder expansion occludes the ureter preventing backflow

22
Q

Etiology of bladder obstruction

A
BPH
Gonorrhea/STI's
bladder tumors
constipation, fecal impaction 
poor pelvic floor support
23
Q

Bladder diverticulae

A

outpouchings that form when inner layers of the bladder herniate through the serosa
lack muscle fibers –> noncontractile
contribute to urinary stasis and infection

24
Q

Excretion definition

A

excretion of waste (urine/stool) from the body

25
Parasympathetic innervation
controls contraction of the detrusor muscle and internal sphincter promotes urination
26
Sympathetic innervation
promotes relaxation of the detrusor muscle and trigone area | promotes bladder retention
27
Somatic innervation
controls relaxation/contraction of the external sphincter
28
Pontine Storage center
located in the brainstem | receives afferent stimuli from bladder
29
Pelvic nerve
innervates detrusor muscle
30
Pudendal nerve
innervates external sphincter
31
Bladder volumes
150 mL = sensation of needing to go (can be delayed) 400-500 mL = full bladder 700 mL = painful. may cause overflow
32
Determinants of urinary elimination
``` intake/output of fluids renal perfusion (determines GFR) GFR nephron function bladder capacity unobstructed flow functional bladder, sphincters, pelvic floor muscles intact neurological function ```
33
Aging & Urinary elimination
``` decreased nerve function muscular atrophy --> weak pelvic floor muscles altered fluid intake polypharmacy co-morbidities BPH (men) decreased kidney function (decreased GFR) decreased mobility/cognition ```
34
Post residual volume
volume remaining in bladder post-void usually <50 mL increases with age
35
DIAPPERS acronym
``` causes of urinary incontinence D I A P P E R S ```
36
Oxybutynin MOA
anticholinergic drug. inhibits action of acetylcholine @ post-ganglionic receptors. used to treat neurogenic/overactive bladder by inhibiting contraction of GU smooth muscle.
37
Oxybutynin therapeutic effects
increased bladder capacity delayed desire to void (decreases sensory input) decreased urge incontinence, urinary urgency, frequency
38
Neurogenic bladder
disruption in the neural pathways can cause either 1) overactive bladder (increased spasticity --> urge incontinence) 2) areflexic bladder (lack of contractions --> overflow incontinence)
39
Loperamide MOA
aka Imodium antidiarrheal. inhibits nervous stimulation of GI wall decreasing peristalsis, prolonging GI transit time & increasing absorption.
40
Loperamide Therapeutic fx
decreased diarrhea decreased fecal volume decreased loss of fluid & electrolytes
41
Normal urinary findings
``` SG: 1.010-1.025 pH: 4.5-6.5 (acidic) straw color output: 30 mL/hr no bacteria, protein, RBCs, glucose ```
42
Urinary Incontinence Interventions
voiding diary limit fluids 2-3 hrs before bed/outings limit caffeine, alcohol, carbonated drinks (diuretics) time/prompted voiding bladder retraining biofeedback --> electrical stimulation of pelvic area kegel exercies/physiotherapy
43
Fecal Incontinence Interventions
``` limit caffeine, alcohol (diuretics) limit fatty foods, dairy, spicy food (if diarrheic) increase fiber intake kegel exercises timed voiding ```
44
Geriatric absorption
increased gastric pH decrease in absorption, GI secretions, motility decreased peripheral perfusion
45
Geriatric distribution
decreased CO, peripheral perfusion --> decreased distribution decrease in muscle mass, increase fatty tissue reduced plasma albumin levels --> enhanced drug fx
46
Geriatric metabolism
decreased hepatic & renal function | reduced first-pass metabolism
47
Geriatric excretion
decreased renal function --> decreased excretion