ECG and Cardiac Arrhythmias COPY COPY Flashcards
Ventricular Arrhythmias: PVCs
premature beat arising from ventricle
- no P wave & bizarre/wide QRS followed by long pause
- serious PVCs = >6/min
- unifocal = same configuration
- multifocal = different configurations
- Bigeminy = normal sinus impulse followed by PVC
- Trigeminy = PVC after 2 normal beats
Ventricular Arrhythmias: Ventricular Tachycardia (VT)
- >3 PVCs occuring sequentially
- 150-200 bpm
- no P wave, wide/bizarre QRS
Ventricular Arrhythmias:
Ventricular Fibrillation (VF)
chaotic activity of ventricle originating from multiple focii
- pulseless, emergency situation
- requires CPR, defibrillation, medications
- unable to determine rate
- bizarre activity on ECG w/ no QRS, no effective CO, clinical death within 4-6 minutes
Atrial Arrhythmias (Outside SA Node)
ECG signs of A-fib
Rate for Tachycardia, Atrial Flutter and fibrillation
- Rapid, and repetitive firing outside SA node
- P waves are not identifiable, (a-fib) or abnormal
- Atrial tachycardia 140-250
- Atrial Flutter - 250-350
- fibrillation - >300bpm
May occur with a normal heart from caffiene, stress, smoking, alcohol and any type of heart disease
PACs are generally benign, flutter is bad
Atrioventricular Blocks
abnormal delays or failure to conduct though normal conduction system
- if ventricular rate is slow, CO decreases
ECG Readings
ST depression
ST elevation
inverted T wave
Wide QRS wave
- ST depression = ischemia downslope 2-3 mm
ST elevation = infarct or pericarditis
inverted T wave = ischemia
Wide QRS wave = bundle branch heart blocks
ECG Changes with and Acute MI
Location and change in lead:
- Anterior Wall: V1-V6
- Anteroseptal: V1-V2
- Ateroapical: V3-V4
- Anterolateral: V5-V6, I, aVL
- Lateral wall: I, aVL
- Inferior wall: II, III, aVF
- Posterior wall: not seen typically, V7-9
ECG Changes from Drug influence
Digitalis
Quinidine
Beta Blockers
Nitrates
Anti-arrhythmatic agents
- Digitalis - depresses ST, flattens T, shorter QT
- Quinidine - QT lengthens, T flat, QRS long
- Beta Blockers - decrease HR
- Nitrates - increase HR
- Anti-arrhythmatic agents - prolong QRS and QT
ECG Changes from Metabolic Influence
Potassium Levels
Calcium Levels
Potassium Levels
- Hyperkalemia - widens QRS, flattens P, “peaked” T
- Hypokalemia - flattens T (or invert), produces U
Calcium Levels
- hypercalcemia - widens QRS, shortens QT interval
- Hypocalcemia - prolongs QT interval
Determining Orthostatic Hypertension
Drop in BP that accompanies change from supine to standing
- Initial HR and BP when pt sitting for >5 minutes
- move directly into standing and repeat HR, BP immediately, and repeat after 3 minutes
Patient OH = systolic decreased > 20 mmHg or if diastolic decreased >10 mmHg