Eating Behaviours Flashcards
Neural / hormonal eating behaviour A01
- Hyp = regulates glucose / homeostasis
- dual centre model (LH/VMH/NPY)
- Ghrelin = hunger stimulant stomach
- Leptin = satiety = adipose excess
Neural and Hormonal eating behaviours A02
Hetherington and Ranson: Lesioning rats VMH = hyperphagic – overeat – no satiety.
Lincinio: people cannot produce Leptin study – leptin replacement therapy – 18m 40% loss of weight / 49% decrease in food intake.
neural and hormonal eating behaviours Eval
Good
= supp research - rats = emp.
= Practical. = leptin replacement therapy
Bad
= rats = reductionist
= Oversimplified = CCK (hunger stim)
Psychodynamic - AN A01
- Minuchin - enmeshment - autonomy - conflict avoidance - Rigidity
- Bruch = starvation = control
- Psychomatic family
Psychodynamic - AN A02
Strauss and Ryan: female AN patients = greater disturbed autonomy // poorer communication with families
Robin et al: BFST in 11 AN patients == 16m 6 patients recovered // 3 more 1yr follow-up
Psychodynamic - AN Eval
Good
= research supp.
= BFST
Bad
= Germillion - Gender Bias
= reductionist
= causal issues
Bio - Obesity A01
- Genes = family / twin studies
- NT = LOW S-NT - metabolite 5HIAA = inhibits VMH // dysfunctional D-NT norm = pleasure
Bio - Obesity A02
Chaput et al = CR = 20-25% = moderate but not substantial hereditability.
Locke = 300,000+ genomes = 97 BMI genes no single gene 97 = 2.7% BMI variation
Watson = found 400+ genes
Ohia = serotonin receptor 2c = not in ‘knockout mice’ who overeat and are obese.
Wang = low dopamine D2 receptors in the striatum mean no reward from eating – eating for pleasure.
Bio - Obesity Eval
Good
= plausible research (Stunkard strong corel. bio adopt study)
= Supp. = Spitz = DRD2 - LOW D-NT obese
Bad
= drug treatments although - chronic / side effects
= diathesis
= MA = leptin more indicative
