DISORDERS OF LANGUAGE Flashcards

1
Q

____________ refers to the production and comprehension of words whereas
_____________ refers to the articulatory and phonetic aspects of verbal expression

A

Language

speech

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2
Q

the
silent processes of thought and the formulation in our
minds of unuttered words on which thought depends

A

inner speech, or endophasia

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3
Q

by which is meant the expression of

thought by spoken or written words and the comprehension of the spoken or written words of others

A

External

speech, or exophasia,

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4
Q

Loss or impairment of the production or comprehension
of spoken or written language because of an
acquired lesion of the brain. This is the condition
called ________

A

aphasia or dysphasia.

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5
Q

A defect in articulation with intact mental functions,
and comprehension of spoken and written language
and normal syntax (grammatical construction of sentences). The terms ___________ are applied to this category of speech disorder.

A

dysarthria and anarthria

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6
Q

An alteration or loss of voice because of a disorder

of the larynx or its innervatioN

A

aphonia or dysphonia

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7
Q

The main executive, or output, region, situated at
the posterior end of the inferior frontal convolution
(Brodmann areas 44 and 45), is referred to as ______________

A

Broca area

and is concerned with motor aspects of speech

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8
Q

visually perceived words are given
expression in writing through a fourth language area, the
so-called ____________ in the posterior part of the
second frontal convolution

A

Exner writing area

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9
Q

The perisylvian language areas are also connected
with the striatum and thalamus and with corresponding
areas in the nondominant cerebral hemisphere through
the _______ and____________

A

corpus callosum and anterior commissure

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10
Q

In general, as a restatement of the Wernicke-Broca scheme, phonologic speech output difficulties are derived from _____________
semantic-comprehension difficulties are the result of ____________

and alexia and agraphia are associated with
_____________

A

left frontal lesions;

left temporal lesions;

inferior parietal lesions.

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11
Q

__________________a procedure that produces
mutism for a minute or two, followed by misnaming,
including perseveration and substitution; misreading;
and paraphasic speech);

A

the Wada test-

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12
Q

_______________ in which different words or phonemes are presented simultaneously to the two ears (yielding a right ear-left hemisphere
advantage)

A

dichotic listening,

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13
Q

the region on the superior surface of the temporal lobe
posterior to Heschl gyri and extending to the posterior
end of the sylvian fissure, is slightly larger on the left
in 65 percent of brains and larger on the right in only
11 percent

A

planum temporale

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14
Q

Left-handedness may result from disease of the left
cerebral hemisphere in early life; this probably accounts
for its higher incidence among the ________ and _______

A

mentally retarded and

brain injured.

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15
Q

T or F

findings suggest a bilateral albeit unequal-representation of language functions in non-right-handed patients.

A

T

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16
Q

the
melody of speech, its intonation, inflection, and pauses,
all of which have emotional overtones

A

prosody

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17
Q

aprosodia localization

A

inferior division of the right middle

cerebral artery.

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18
Q

In the investigation of aphasia, it is first necessary to

inquire into the patient’s __________

A

native language, handedness,

and previous level of literacy and education.

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19
Q

More often, the language most used before the onset

of the aphasia will recover first. What law of aphasia?

A

Prite Law

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20
Q
The bedside analysis of aphasic disorders that we
find most useful entails the systematic testing of six
aspects of language function: 
1
2
3
4
5
6
A

conversational speech, comprehension,

repetition, reading, writing, and naming

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21
Q

Reading aloud single letters, words, and text

may disclose the __________

A

dissociative syndrome of pure word

blindness.

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22
Q

The patient may be unable to
repeat what is said to him, despite relatively adequate
comprehension-the hallmark of_______________

A

conduction aphasia

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23
Q

Preserved repetition is also characteristic
of _________ and occurs occasionally with
subcortical lesions

A

anomic aphasia

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24
Q

examples of this include conduction aphasia,
word deafness (auditory verbal agnosia), and word
blindness (visual verbal agnosia or alexia

A

disconnection

language syndromes,

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25
Q

patients may repeat a few stereotyped
utterances over and over again, as if compelled
to do so, a disorder referred to as ____________

A
monophasia (Critchley),
recurring utterance (Hughlings Jackson), verbal stereotypy,
or verbal automatism.
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26
Q

In Broca’s aphasia, speech is sparse with _________as compared with the normal 100 to 115 words
per minute

A

10 to 15 words per

minute

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27
Q

If a patient with nonfluent Broca’s aphasia has no difficulty
in repetition, the condition is termed ________

A

transcortical

motor aphasia

28
Q

________________
causes the patient to be wordless (mute) but leaves inner
speech intact and writing undisturbed

A

pure word mutism (aphemia ),

29
Q

The location of the lesion in cases of Wernicke’s aphasia
is the ___________ near the primary
auditory cortex

A

left superior lateral temporal lobe

30
Q

The substitution of one word for another (“The grass is
blue”) is termed____________
and is even more characteristic of Wernicke’s aphasia.

A

verbal paraphasia or semantic substitution

31
Q

components of the peri-sylvian region resulting wernicke’s aphasia

A

posterosuperior temporal, supramarginal,

angular, and posterior insular gyri

32
Q

What is the arcuate fasciculus

A

This fiber tract streams out of the temporal
lobe, proceeding somewhat posteriorly; around the posterior end of the sylvian fissure;

there it joins the superior longitudinal fasciculus, deep in the anteroinferior parietal region, and proceeds forward, deep to the suprasylvian operculum, to the motor association cortex, including the
Broca and Exner areas

33
Q

In____________ the patient suffers a deficit of auditory and visual word comprehension, making writing and reading impossible, in every way conforming to Wernicke’s aphasia.
Speech remains fluent, with marked paraphasia, anomia, and empty circumlocutions. However, unlike the deficit in Wernicke’s and conduction aphasias, the ability to ________ is preserved

A

transcortical sensory aphasia

repeat the spoken
word

34
Q

lesions of transcortical sensory aphasia

A

posterior parietooccipital

region

35
Q

Two clinical contexts of transcortical motor aphasia

(1) in a mild or partially ____________ in
which repetition remains superior to conversational
speech (repeating and reading aloud are generally easier
than self-generated speech) and

(2) in states of ________with frontal lobe damage

A

recovered Broca’s aphasia

abulia and
akinetic mutism

36
Q

impairment of auditory comprehension

and repetition and an inability to write to dictation.

A

pure word deafness

37
Q

The most striking feature of this syndrome is the retained
capacity to write fluently; after which the patient cannot
read what has been written

A

alexia without agraphia

38
Q

the individual loses the ability to understand written
script, and, often, to name colors, i.e., to match a seen
color to its spoken name

A

visual verbal color anomia

39
Q

In alexia, In some cases, the patient manages to read single letters but not to join them together

A

(asyllabia).

40
Q

lesions associated with alexia

A

posterior part (splenium) of
the corpus callosum, wherein lie the connections between
the visual association areas of the two hemispheres

41
Q

the patient loses all capacity to speak while retaining perfectly the ability to write, to understand spoken
words, to read silently with comprehension, and to repeat
spoken words

A

Pure Word Mutism (Aph e m i a )

42
Q

An anomie type of aphasia is often an early sign of
Alzheimer and Pick disease (minor degrees of it are common in old age) and is a principal feature of one type of degenerative lobar cerebral atrophy in the category of
the __________

A

primary progressive aphasias

43
Q

lesions for foreign accent syndrome

A

The syndrome arises
after a left-sided lesion, most often a stroke with a mild
associated Broca’s aphasia

44
Q

Mechanisms of agraphia

A

there is a specific difficulty in transforming phonologic
information, acquired through the auditory sense, into
orthographic forms;

others see it as a block between the visual form of phonemes, and the cursive movements of
the hand

45
Q

lesions of agraphia

A

imaging sometimes discloses a lesion of the posterior

perisylvian area

46
Q

letters and words are formed clearly

enough but are wrongly arranged on the page

A

constructional agraphia .

47
Q

language formulation is correct
and the spatial arrangements of words are respected, but
the hand has lost its skill in forming letters and words

A

apraxic agraphias.

48
Q

types of linguistical agraphia

A

phonologic, lexical,and semantic types.

49
Q

or there may
be preserved ability to write nonsense words but not
irregular words, such as island ___________

A

(lexical agraphia);

50
Q

patients
with __________ have difficulty incorporating the
proper meaning into the written word, e.g., “the moon
comes out at knight. “

A

semantic agraphia

51
Q

Aphasia has also been described frequently with
_______________, particularly if they
extend laterally into the subcortical white matter of the
temporal lobe and insula.

A

dominant striatocapsular lesions

52
Q
\_\_\_\_\_\_ or the production of vocal sounds, is a
function of the larynx, more particularly the vocal cords
A

Phonation,

53
Q

the past, bilateral paralysis of the
palate, causing nasality of speech, often occurred with
_______ and ________ but now it occurs most
often with _________ a form of motor
neuron disease

A

diphtheria and poliomyelitis,

progressive bulbar palsy,

54
Q

What state is this?

the jaw jerk and other facial reflexes usually
become exaggerated, the palatal reflexes are retained or
increased, and emotional control is impaired (spasmodic,
crying, and laughing

A

the pseudobulbar affective state

55
Q

_________ is
a condition in which the signs of spastic and atrophic
bulbar palsy are combined.

A

Amyotrophic lateral sclerosis

56
Q

A severe dysarthria that is difficult to classify, but
resembles that of cerebellar disease, may occur with a
left hemiplegia, usually the result of ___________

A

capsular or right

opercular infarction

57
Q

rapid
mumbling and cluttered utterance and slurring of words
and syllables. The voice is low-pitched and monotonous,
lacking both inflection and volume (hypophonia), and
trailing off in volume at the ends of sentences

A

R i g i d ( Extra pyra m i d a l ) Dysa rt h r i a

58
Q

Talking is loud, harsh, improperly stressed or accented, and poorly coordinated with breathing: what type of speech?

Associated with what conditions?

A

(hyperkinetic dysarthria).

chorea and myoclonus

59
Q

The Tourette syndrome
of multiple motor and vocal tics is characterized both by
____________ (barking noises, squeals, shrieks,
grunting, sniffing, snorting) and by ____________,
notably stuttering and the involuntary utterance of
obscenities__________

A

startling vocalizations

speech disturbances

(coprolalia).

60
Q

The
speech is loud, slow, and labored; it is poorly coordinated
with breathing and accompanied by facial contortions
and athetotic excesses of tone in other muscles

A

double athetosis.

61
Q

Scanning
dysarthria, speaking metronomically as if scanning poetry
for meter, is another distinctive cerebellar pattern and is
most often a result of __________

A

mesencephalic lesions involving the

brachium conjunctivum

62
Q

interruptions o f the

normal rhythm of speech by involuntary repetition prolongation or arrest of uttered letters or syllables

A

stuttering

63
Q

Stuttering differs
from ____________ in which there is repetition of a word or phrase with increasing rapidity, and from ________, in
which there is an obligate repetition of words or phrases

A

palilalia,

echolalia

64
Q

A condition in which otherwise healthy, who lose
the ability to speak quietly and fluently. Any attempt to
speak results in simultaneous contraction of the speech
musculature, so that the patient’s voice is strained and
speaking requires an effort.

A

spasmodic dysphonia

65
Q

conditions associated with spasmodic dysphonia

A

with blepharospasm, spasmodic torticollis, writer ‘s

cramp, or some other type of segmental dystonia

66
Q

The most effective tx for spasmodic dysphonia

A

The most effective
treatment, comparable to treatment of other segmental
dystonias, consists of the injection of 5 to 20 U of botulinum
toxin, under laryngoscopic guidance, into each
thyroarytenoid or cricothyroid muscle