Diagnostic imaging - the equine foot Flashcards

1
Q

How much force does the TL of a horse experience at walk, trot and gallopp?

A
  • walk: 1/2 times
  • trot: 1 times
  • gallopp: 2.5 times BWt
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2
Q

What proportion of TL lameness is in the foot?

A

80%

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3
Q

T/F: it is difficult to relate pain to specific structures by direct localisation, palpation or manipulation

A

True

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4
Q

What is another name for radiograph?

A

summation images (they are a 2D image of a 3D structure)

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5
Q

What changes can be seen radiographically?

A
  • tissue density
  • size
  • shape
  • outline
  • position
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6
Q

What degree of mineralisation is required until it is seen on radiographs?

A

30-50% change in mineralisation

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7
Q

What are the 7 main orthopaedic diseases?

A
  • OA
  • OCD
  • fractures
  • tendonitis
  • navicular disease
  • wounds
  • laminitis
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8
Q

Other names for OA

A
  • arthritis
  • arthrosis
  • osteoarthrosis
  • degenerative joint disease
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9
Q

What happens to the joint in OA?

A
  • inflamed synovium
  • reduced viscosity of synovial fluid
  • fibrillated/ destroyed cartilage
  • osteophytes
  • bone sclerosis
  • frayed, cracked meniscus
  • thickened meniscus
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10
Q

CS - arthritis in horses -

A
  • severe joint effusion of tibiotarsal joint (aka bog spavin)
  • high ringbone (OA of the pastern joint, this is because there is a small synovial compartment, you don’t get swelling but instead new bone formation)
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11
Q

What is laminitis?

A

systemic disease that manifests in the foot, mostly an endocrine disease

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12
Q

What is navicular syndrome?

A
  • aka palmar foot syndrome

- where one structure in palmar aspect of foot is affected and causes pain

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13
Q

What are radiographic signs of OA?

A
  • SYNOVIAL MEMBRANE: joint effusion, osteophytes
  • ARTICULAR CARTILAGE: narrowing joint space (can also have widening)
  • SUBCHONDRAL BONE: sclerosis/ lucency
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14
Q

Dx - OA

A
  • affected strucutres (high/low motion joint)
  • type of disease (primary, secondary, traumatic, developmental)
  • how advanced is disease (early versus late)
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15
Q

What affects prognosis of OA?

A
  • structures affected
  • type of disease
  • how advanced the disease is
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16
Q

Tx options - OA

A
  • strategic analgesia
  • joint supplements
  • novel tx concepts
  • salvage procedures
  • exercise modification/ physio
17
Q

How do corticosteroids help tx OA?

A

= anti-inflammatory

- stops cell membrane phospholipid conversion into AA

18
Q

How do NSAIDs help tx OA?

A

= anti-inflammatory

- stops AA conversion into prostaglandins and thromboxanes

19
Q

What are the main inflammatory mediators in OA?

A
  • leukotrienes
  • prostaglandins
  • thromboxanes
20
Q

Outline nuclear scintigraphy

A
  • select a molecule of target tissue (phosphonates for bone)
  • combine with radioactive isotope (technetium 99m)
  • inject into patient
  • detect radioactivity
21
Q

Indications -nuclear scintigraphy - horses

A
  • inflammation
  • pulmonary
  • GIT
  • vascular
  • renal
  • reproduction
    EXAMPLES:
  • acute severe lameness
  • proximal limb/ back/ neck/ head (unblockable areas)
  • multiple site problems
  • unruly horse
  • the puzzling case
  • others
22
Q

What is a core lesion?

A

haemorrhage and GT causing a tendinopathy or desmopathy outside of the foot

23
Q

Define desmopathy

A

a pathology of ligament

24
Q

Non-imaging techniques of examination of horse hoof

A
  • inspect hoof wall and sole
  • palpate at coronary band
  • hoof testers and percussion
  • diagnostic analgesia