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Periodontics II > Diagnosis and Classification of Perio Diseases > Flashcards

Diagnosis and Classification of Perio Diseases Flashcards

1
Q

Factors of Diesease

A
  • Microbial plaque
  • genetics/host factors
  • Aquired/environmental factors
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2
Q

Things we have control over changing

A

microbial and host factors

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3
Q

reason for loss of… is due to

A

response due to biofilm

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4
Q

history and physical examination includes

A 
chief complaint
risk history, medical and dental history
EO/IO
Clinical examination
Radiographic examination
supplemental diagnostic test
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5
Q

order of diagnosis

A

History/exam -> Diagnosis -> Prognosis -> Treatment -> Non/surgical -> asses outcome -> history/exam or prognosis

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6
Q

Making a diagnosis

A
  • describing the absence or presence of inflammation

- measuring the level of attachment loss

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7
Q

examle of logical inferences

A
  • Raidographs and attachment levels indicate what happened in the past
  • signs of inflammation such as erythema, swelling, bleeding etc .. indicate what is happening now
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8
Q

Periodontal probe

A
  • in mm

- used evaluate the health of periodontal tissues

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9
Q

pocket depth

A

histological depth

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10
Q

probe depth

A

clinical depth

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11
Q

function of probe is used to determine the

A
  • health of the perio tissues
  • extent of damage to the perio tissues
  • miniature rulers for making intraoral measurements
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12
Q

bleeding on probing

A

for 4 consecutive visits 30% chance of clinical attachment loss

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13
Q

Probe position with attachment loss

A

tip touches the root below CEJ
Usually greater than 3mm
Disease present (bleeding)
Difficult to measure

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14
Q

finding that important for diagnosis perio disease

A 
probing depths
recession
bleeding on probing
furcation involvement
mobility
Spacing
rotation
caries
anatomy (crowns)
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15
Q

1999 World Workshop Class I

A

gingival diseases

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16
Q

1999 World Workshop Class II

A

Chronic Periodontitis

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17
Q

1999 World Workshop Class III

A

Aggressive Periodontitis

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18
Q

1999 World Workshop Class IV

A

Periodontitis as a manifestation of Systemic Diseases

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19
Q

1999 World Workshop Class V

A

Necrotizing Periodontal Diseases

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20
Q

1999 World Workshop Class VI

A

Abscesses of the Periodontium

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21
Q

1999 World Workshop Class VII

A

Periodontitis associated with endodontic lesions

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22
Q

1999 World Workshop Class VII

A

developmental or acquired deformities and conditions

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23
Q

Dental plaque-induced gingival diseases

A
  • associated with dental plaque only
  • modified by systemic factors
  • modified by medications
  • modified by malnutrition
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24
Q

Non-plaque induced gingivallesions

A
  • bacterial orgin
  • viral orgin
  • fungal origin
  • gentic origin
  • manifestation of systemic conditions
  • traumatic lesions
  • foregin body reactions
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25
Q

two main categories of gingivitis are

A

plaque induced

non-plaque induced

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26
Q

plaque induced gingivities results from

A

interplay between plaque bacteria and host denfense system

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27
Q

plaque induced inflammation the results is primarily attributable to the

A

body’s response in the presence of bacteria and or their products

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28
Q

1989 world workshop Class I

A

adult perio

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29
Q

1989 world workshop Class II

A

-
-

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30
Q

1989 world workshop Class III

A

Perio associated with systemic disease

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31
Q

1989 world workshop Class IV

A

necrotizing Ulcerative Perio

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32
Q

1989 world workshop Class V

A

Refracting Perio

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33
Q

you can treat gingivitis with a

A

prophylaxis

34
Q

slight gingivitis can be

A

slight areothemia

35
Q

plaque induced gingivitis can occur in

A

area where there had been attachment loss previously, but is stable and not progressing

36
Q

Gingivititis: _____ _____ must be available for proper diagnosis, including ___ ____ ___

A

longitudinal records, clinical attachment levels

37
Q

can you make diagnosis of gingivitis from a radiograph?

A

NO

38
Q

amlodipine induces

A

gingival enlargemnet

39
Q

gingivitits associated with systemic medications

A
  • phenytoin (dilantin)
  • Cyclosporin
  • Calcium channel blockers (ex. Nifedipine)
40
Q

Guidelines of extent of disease are

A

> 30% = Generalized

41
Q

non-plaque induced gingivitis represents a small percentage of cases and can result from

A
  • virus
  • fungi
  • allergic reactions
  • autoimmune reactions
  • Trauma
42
Q

desquamative means

A

shloffing off tissue

43
Q

auto immune reactions such as desquamative gingivitis, caused by?

A

lichen planus
mucous membrane
pemphyigold and pemphigus vulgaris

44
Q

lichen planus can lead to

A

squamous carcinoma

45
Q

Clinical attachment loss depths for slight, moderate, severe for periodontitis

A

slight: 1-2 mm CAL
moderate: 3-4 mm CAL
severe: > 5 mm CAL

46
Q

chronic periodontitis is characterized as a

A

slowly progressing disease

47
Q

can patients exhibit short periods of rapid progression?

A

Yes

48
Q

what is the most commonform of periodontitis?

A

chronic perio.

49
Q

what induces chronic perio.?

A

plaque

50
Q

chronic periodontitis leads to inflammation of the periodontal tissue that results in:

A
  • inflammation of the periodontal tissues
  • destruction of connective tissue of PDL
  • destruction of supporting alveolar bone
51
Q

diagnosis of perio. must have both what at the same time and location?

A
  1. loss of periodontal attachment from the cemento-enamel junction
  2. Presence of gingival inflammation that extends beyond the marginal gingiva into the connective tissue
52
Q

what did aggressive periodontitis replaced?

A

early onset periodontitis

53
Q

Generalized Aggressive

Periodontitis

A

-usually seen in clinically healthy

54
Q

common feautres of localized (LAP)and generalized aggressive periodontitis (GAP)

A 
Clinically healthy subjects
Rapid onset of attachment loss and bone loss
Familial aggregation
Phagocyte abnormalities
Hyperresponsive macrophage phenotype
55
Q

????protein associated with aggressive periodontitis?

PMN trianlge

A

protein GP 120?

56
Q

Amounts of microbial deposits are ______ with the severity of periodontal destruction

A

inconsistent

57
Q

bugs associated with aggressive periontitis

A

A.a. and P.g.

58
Q

most import part of treatment?

A

re evaluation

59
Q

Aggressive Perio facts for localized

A
  • circumpubertal onset
  • First molar or incisor w/ attachment loss atleast 2 permanent teeth, one of which a first molar
  • sig. serum antibody response to infecting agents
60
Q

aggressivePerio facts for generalized

A
  • usually under age 30,but may be older
  • gen attachment loss aff. least 3 teeth other than first molar and incisors
  • poorserum response to infecting agents
  • epdisoic nature of destruction
61
Q

necrotizing periodontal diseases two basic forms:

A
  1. Necrotizing Ulcerative gingivitis (NUG)

2. Necrotizing Ulcerative periodontitis (NUP)

62
Q

Common feautures of NUG and NUP include:

A
  • Rapid onset of pain
  • Ulceration and necrosis of marginal gingiva (papillais the classis location)
  • predisposition to decreased systemic resistance to certain bacterial infections
63
Q

NUG is/has

A
  • interproximal necrosis and ulceration of gingival papilla or margin(black triangle)
  • Rapid onset of pain
  • Fetid breath
  • may have pseudomembrane on the surface
  • may have lymphadenopathy, fever, malais, anorexia
64
Q

smell is coming from

A

valital fatty acid spirochetes and dead tissue

65
Q

NUG factors associated with disease

A
  • emotional stress
  • heavy cigarette use
  • lack of sleep
  • poor diet
  • immunosuppression
66
Q

Nup is/has

A
  • sim. to clinical apprearance of NUG
  • often involves sign loss of connective tissue and alveolar bone
  • sometimes bone exposure and sequestration is seen
  • associated with severe immuno-suppression is seen in small proportion of HIV- infected patients or those protein -energy malnutrition
67
Q

periodontal abscess is

A
  • circumscribed collection of neutrophil-laden pus in the sift tissue wall of the periodontal pocket
  • usually has a swelling
  • can have a fistula
68
Q

fistula is

A

An abnormal connection between organs

69
Q

causes of perio abscess

A
  • deep periodontal pockets
  • incomplete calculus removal
  • occlusion of the pocket by foreign bodies
  • use of antibiotics without scaling and root planing to remove plaque and calculus
70
Q

Periodontal Abscess is classified into what categories:

A

Gingival Ascess
Periodontal Abscess
Pericoronal Abscess

71
Q

gingival abscess

A

localized to the gingiva only

72
Q

periodontal abscess

A

localized to the tissues adjacent to the periodontal pocket that may lead to the periodontal ligament and bone

73
Q

Pericoronal abscess

A

associated with the crown of a partially erupted tooth

74
Q

developmental or acquired deformities / conditions

A
  • cervical enamel projections
  • enamel pearls
  • furcation anatomy
  • root proximity
  • tooth position
  • root surface grooves
75
Q

what are mucogingival deformities

A

lack or absence of keratinized marginal gingival tissue

76
Q

what can cause mucogingival deformities

A
  • trama
  • hist of periodontitis
  • ortho (moving teeth)
77
Q

occlusal tramuma results from

A

loads on a tooth that exceeds the ability of the periodontium to withstand them

78
Q

primary occlusal tramuma is

A

highly moblie

79
Q

second occlusal tramuma is

A

most common , loss attachment and is now normal but has movment

80
Q

the two recognized forms of occlusal trauma

A

primary and secondary occlusal tramuma

81
Q

Periodontitis as a manifestation of systemic diseases is

A

asscoiated with hematologic disorders ( neutropenias, leukemias)

82
Q

associated genetic disorders with Periodontitis as a manifestation of systemic diseases are

A
  • familial and cyclic neutropenia
  • down syndrome
  • leukocyte adhesion deficiency syndrome
  • papillon-leferve syndrome
  • chediak-higashi syndrom

Periodontics II (10 decks)

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