deck 9 Flashcards

1
Q

What is the rhythm?

A

bradycardia-dependent LBBB

  • pertinent EKG findings:
    • ​LBBB pattern at slower HR
    • instantaneous reversion to normal HPS conduction at slightly faster HR
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2
Q

EKG features of HCM? (4)

A

mnemonic: Ladies Love Hunky Persists

  1. LAD (20% of cases)
  2. LVH (50-60% of cases)
  3. pseudoinfarct pattern (Q waves in I, aVL, V4, V5)HSTs
  4. HSTs (hypertrophy-related ST changes)
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3
Q

Which 2 syndromes can mimic MAT? How can you distinguish MAT from these syndromes?

A

MAT can resemble:

  1. Sinus tachycardia w/ frequent APCs (MAT will have absence of one dominant atrial pacemaker)
  2. Coarse AFib (MAT will have isoelectric baseline between p waves unlike AFib)
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4
Q

What does the term “pseudo-RVH” refer to?

A

large R wave w/ TWI in V1 (seen in HOCM)

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5
Q

Describe 4 EKG changes that are typical of digitalis EFFECT.

A

dig effect changes are similar to those of hyperkalemia:

  1. Salvador dali mustache (sagging ST segment)
  2. decreased QT interval (due to shortened phase 2 of action potential)
  3. prominent U Waves
  4. increased PR interval
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6
Q

What are the characteristics of anti-arrhythmic drug toxicity? (6)

A
  • slowing of ventricular repolarization: marked prolongation of QTctorsades de pointes
  • slowing of conduction in His-Perkinje system: QRS widening and AVB
  • slowing of automaticity of PM cells: sinus bradycardasinus arrest
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7
Q

Miller (Varecki) algorithm for VT diagnosis? (4)

A

mnemonic: Ivana Wants New Dick

ANY of the following present in aVR is diagnostic of VT:

  1. Initial R-wave
  2. Width of r or q wave > 40 ms
  3. Notch on descending limb of neg QRS
  4. dvi/dvt

**velocity ratio = dvi/dvt Where dvi is change in voltage over initial 40 ms and dvt is change in voltage over terminal 40 ms

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8
Q

Which type of VT will respond to verapamil?

A

fascicular VT

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9
Q

Most common cause of ST elevation on resting EKG?

A

normal-variant early repolarization

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10
Q

ER resident calls you for cath consult but says he thinks this is early repol. He tells you there are 3-4 mm concave-down ST elevations in V2 & V3.

What to tell him?

A

this is likely NOT early repolarization because STEs are TOO HIGH AMPLITUDE & are CONCAVE DOWN

  • STE characteristics in early repolarization:
    • up to 3 mm in precordial leads (V2-V4) & up to 1 mm in limb and lateral leads
    • concave up
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11
Q

Differential diagnosis of prolonged JT segment? (2)

A
  1. hypocalcemia
  2. LQTS (type III)
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12
Q

Which electrolyte abnormalities can prolong the QT interval? (3)

A
  1. hypocalcemia
  2. hypomagnesemia
  3. hypokalemia

note that the above electrolyte deficiencies prolong different regions of the QT interval

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13
Q

Criteria for coding accelerated idioventricular rhythm? (4)

A
  1. QRS morphology similar to PVCs
  2. AV dissociation
  3. +/- capture/fusion beats
  4. rate = 60-110 bpm
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14
Q

Name 2 characteristics of AV dissociation.

A
  1. atria & ventricles depolarize independently from one another
  2. ventricular rate is usually greater than atrial rate
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15
Q

distinguishing EKG characteristics of AVNRT? (3)

A
  1. usually starts w/ APC (no warm up period)
  2. short RP tachycardia
    • pseudo R’ in V1 - P wave buried in V1 that was not present in NSR
    • RP interval usually
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16
Q

EKG diagnosis of left atrial abnormality? (3 different criteria)

A

code for LAA if ANY of the following are present:

  1. V1 criterion: terminal P > 1.5 mm deep x 0.04 ms wide
  2. II criterion: p > 0.12 ms wide
  3. p-mitrale: II has BIFID p-wave AND peak-to-peak interval > 0.03 s
17
Q

criteria for diagnosis of acute posterior wall MI (2)

A

ALL of the following must be present in V1 AND V2:

  1. pathologic R waves (R>S AND R > 40 ms wide)
  2. ST depression > 1-2mm
18
Q

How to distinguish blocked APC from 2nd degree AVB?

A
  • blocked APCs: shortened PP interval followed by absent QRS
  • 2nd degree AVB: constant PP followed by absent QRS
19
Q

DDx of prominent upright U waves? (6)

A

mnemonic: CHAD Loves Urine (Urine stands for u wave)

  1. CAD
  2. Hypothermia/Hypokalemia
  3. ARD effect
  4. Digoxin effect
  5. LVH
20
Q

DDx of prominent inverted U waves? (2)

A
  1. LVH
  2. CAD
21
Q

Criteria for interventricular conduction delay? (2)

A

either of the following:

  1. QRS ≥ 0.11 s AND pattern not c/w RBBB/LBBB
  2. abnormal notching of QRS (with or without prolongation)
22
Q

Conditions that can cause IVCD pattern on EKG? (5)

A

Slowed HPS conduction

  • due to electrolyte disturbance → hyperkalemia
  • Slowed conduction due to low body temphypothermia
  • Increased amount of myocardium to depolarize* → LVH/RVH
  • Bypass tract with Abnormal ventricular activation* → WPW
  • abberent intraV conduction* → prior infarct (Fractionated QRS)
23
Q

Criteria for diagnosis of complete LBBB? (3)

A

All of the following must be met:

  1. QS or rS in Rt precordial leads (V1 & V2)
  2. broad R in lateral leads (I, V5, V6)
  3. General Criteria for complete BBB met**

**all complete BBBs must have:

  • QRS > 120 ms
  • Delayed intrinsicoid deflection (> 50 ms)
  • Appropriate Secondary ST changes
24
Q

Criteria for diagnosis of complete RBBB? (3)

A

All of the following must be met:

  1. qR or rSR’ in Rt precordial leads (V1 & V2)
  2. wide slurred S in lateral leads (I, V5, V6)
  3. General criteria for complete BBB met**

**all complete BBBs must have:

  • QRS > 120 ms
  • Delayed intrinsicoid (> 50 ms)
  • Appropriate Secondary ST changes
25
Q

RBBB morphologic criteria for diagnosing VT using Brugada method? (4)

A

RBBB-morphology with ANY of the following features suggests VT:

  1. monophasic R (V1 or V6)
  2. qR (V1 or V6)
  3. notched QS (V1 or V6)
  4. R/S ratio > 1 (V6 only)