Dani - ch 10,11,13 Flashcards
normal impulse conduction
SA –> AV –> bundle of His –> bundle branches –> purkinje fibers
SA –> AV time
.03
AV delay time
.13
AV bundle –> purkinje time
.17
transmission to ventricular muscle
.21 on right
.22 on left
___ ___ = prominent pacemaker
SA node
intrinsic rate of __-__ beats/min for the SA
70-80
__ ___ = back up pacemaker
AV node
intrinsic rate of __-__ beats/min for the AV
40-60
___ ___: back up pacemaker to the AV
purkinje fibers
intrinsic rate of __-__ beats/min for the purkinje fibers
15-40
___ ___ = located somewhere other than SA node and causes an abnormal sequence of events as noted on an ECG
ectopic pacemaker
___ ___: block between SA and AV node
heart block
PS stimulation
- ___ nerve
- ____ (Neurotransmitter)
- innervate ___ and __
vagus
ACh
SA;AV
Sym stimulation
- ____ (Neurotransmitter)
- ___ rate of conduction
- ___ force of contraction
NE
increase
increase
SA –> AV initiates the ___ ___
p wave
R and L bundle branches –> purkinje fibers initiates the ___ ___
QRS complex
p wave = ___ ___
atrial depolarization
QRS complex = ___ ___
ventricular depolarization
T wave = ___ ___
ventricular repolarization
S-T segment is before the ___ of ventricles
repolarization
electrical impulses arrives in the ___
septum
strong electronegativity on the insides of ___
ventricles
strong electropositivity on ____ ___
outer walls
current flows in an ___ pattern from base to apex
elliptical
___ ___ triggers cardiac impulse
sinus node
____ = abnormal rhythm of the pacemaker
arrhythmias
____ = slow heart
bradycardia
bradycardia is present in athletes who have ___ SV
increase
___ ___ syndrome = baroreceptors in carotid sinus are super sensitive and mild pressure on neck has barareceptor reflex which slows HR
carotid sinus syndrome
___ = fast heart
tachycardia
bradycardia = less than ___ bpm
60
tachycardia = more than ___bpm
100bpm
conditions that increase HR
exercise fever stress shock blood loss weakened myocardum
tachycardia cause a ___ reflex to increase HR to pump more blood out
sympathetic
___ ___: the sinus rhythm is influenced b parallel vagus nerve activity during breathing.
sinus arrhythmia
sinus arrhythmia = irregular difference between __ and __ wave
T
P
Sinoatrial block: the impulse is blocked before it enters the R atria so no ___ ___ present
P wave
sinoatrial block = ___ ___ becomes pacemaker
AV node
sinoatrial block = QRS and T are there but are ___
slowed
1st degree heart block = P-R interval more than ___; referred to as “incomplete” heart block because the signal is ____
.20
delayed
acute rheumatic heart disease - causes inflammation of ___ node
AV
second degree heart block - when P-R interval is ___-___
.25-.45
second degree heart block - some impulses pass through the AV bundle but others do not causing ___ ___
drop beats
second degree heart block - p wave is present but no ___ or ___
QRS
T
second degree heart block - failure of conduction system from ___ to ___
atria
ventricles
third degree heart block - complete lack of ___ between atria and ventricles
synchronization
third degree heart block - ventricles spontaneously establish own ___
signal
third degree heart block - P-P interval is consistent but there is a dissociation with ___ and ___
QRS
T
stokes adams syndrome - complete __ block comes and goes
AV
stokes adams syndrome - ventricles stop contracting for 5-30 seconds because of ___-___ ___
overdrive suppression
stokes adams syndrome - person faints due to lack of ___ ___
blood flow
stokes adams syndrome - ____ implanted in RV
pacemaker
premature contraction result from ___ ___
ectopic foci
causes of ectopic foci -
local areas of ____
ischemia
causes of ectopic foci -
____ of cardiac muscle
irritation
causes of ectopic foci -
- toxic irritation from ___, ____ and ___
drugs
caffeine
nicotine
causes of ectopic foci -
- mechanic initiation of PVC during catheterization of ___ ___
RV
premature atrial contractions -
caused by
- lack of ___
- too much ___, ____, ___ and various types of ____
sleep coffee smoking alcoholism drugs
___ ___ can occur with PACs because ventricles do not fill so SV decreases
pulse defecit
___ ___ - the affected atria contracts at a rate of 200-350 bpm because of re-entry wave
atrial flutter
atrial flutter - signals reach ____ too quickly for all impulses to be passed on to the ventricles
AV
atrial flutter - both the av node and av bundles are in a ___ period during many of the impulses
refractory
atrial flutter - p waves are strong but ___ and ___ follow ever 2-3
QRS
T
___ ___: activation of atria is irregular and chaotic so atria becomes useless as a primer pump for the ventricles - efficiency drops by 2-30%
atrial fibrillation
atrial fibrillation causes include ___ resulting from valve dysfunctions that prevent emptying or from ventricular failure because of damming of blood
hypertrophy
premature ventricular contractions - QRS has an ___ form because 1 side depolarizes ahead of the other
abnormal
premature ventricular contractions - t wave is ____
inverted
ventricular tachycardia - contract over a rate over ___bpm
120
ventricular tachycardia - caused by ___ or __ ___
ischemia
myocardial infarction
ventricular tachycardia - can quickly lead to ___ ___
ventricular fibrillation
___ ___ - ventricular depolarization occurs chaotically so some parts contract while other parts are relax so little blood pumped out of heart
ventricular fibrillation
ventricular fibrillation: has multiple re-entry loops from a __ heart muscle or if ___
Dilated
diseased