complete venous circulation Flashcards
بسم الله وكفي به وكيلا
Where is the refence point ?
In midpoint of the Right atrium and is located at the level of the costosternal junction
which pressure equals Zero
What happens in case of increased vr to the refence point ?
the pressue increases above the 0 so heart increase its contraction and pumping to eject the added amount to retain the 0 pressure at mid point of the right atrium
what happens in case of decreased VRto the Refernce point ?
the pressure is below the 0 and this decrased the blood to the left ventricle decreasing its activity and contractility and pumping and this leads to retain the 0 pressure at mid point of right atrium
Mechanism of action
• Diuresis →↓ plasma volume → ↓ venous return (VR) → ↓ preload → ↓pulmonary congestion & peripheral edema
• ↓ signs & symptoms of venous congestion such as orthopnea & nocturnal dyspnea
لما يرمي الحجم الزائد في الشارع ايه هيحصل؟
هيقلل اعراض الاحتقان كمان هيقلل الحمل علي القلب فهيقلل الحمل علي الرئة
لان القلب الان شايف شغله علي الكمية الباقية
Hydrostatic indifferent point explain /
it is a subdiapharagamtic point lies 2-3 inches below the opening of the inderior vena cava opeining in the diapharagm and the pressue at this point is not changed according to the position either supine or erect
all the point below it show increase in pressure in erect than supine
all the points above it show decrease in pressure #####
all humans have antigravity mechanisms to reduce effect of gravity on our circuation>
this point repersnts balance between gravit and anti gravity mechanisms
if anti gravit mechanisms are strong they cause its upward shift
if anti gravity are weaker it causes it downward shift
Mention antigravity mechanisms and theri role .
They are trying to decrease capcatiance to increasr VR 1- venomotor tone 2- incrasing blood vloume 3-skeletal muscle contraction 4-water immersion VISWA they cause upward shift of HIP
The Factors causing downward shift of the HIP .
AlHOr
1-alpa blockers DERESING VR
2-hypovlemia due to hemorrhage DECRASING VR
3-orthostatic hypotension postural hypotension DECRASING VR
Decrasing the activity of anitgravity m mechanisms
ليا دعوة باللي ظاهر ! ! ! ! ! !
اعمل اسكيمات براحتك طالما هتراجعها كتير
What is VR ?
It is the amount of venous blood entering the right atrium per minute and it is always equal to cardiac output and it is
VR=Change of pressure / resistance of veins
resistance of veins is ngelected
so (VR= Change of pressure )
VR = PVP-CVP
Factors affecting venous return mention .
Forces from the front visa foronto and forces fron the behind visa tergo
1- forces from the front
affect by decreasing the CVP to increase the gardient
a-thoracic pump
b-cardiac suction
2-forces from behind
affect by increaing the pvp
PMA VB
pressure gradient muscles pump arterial pulsaiton venomotor tone blood volume V MAP
Describe cardiac suction as vis A fornto .
During rapid ejection phase the ventricle contract casuing dilation of the right atria by the pulled cusps rings and the connecting CVENIS causing their VD and decrasing the CVP so the gradient increase and the VR increase !
in case of heart failure what happen to cardiac suction
the ventricle cannot contract cannot dilate the ra and the veins and the vr decreasses
لا حل مع الآلام والعقبات إلا بمواصلة العمل التشكي والتأوه لا يصنعان شيئا بل يزيدان الآلام والعقبات
ومن للأمة الغرقي إن كنا نحن الغريقينا ؟!
ان تدرس تعمل تدرس تتعلم تدرس ترضي ربك تدرس تزاد علما وفقها تدرس ترتقي منازل الصالحين تدرس تعلم الناس تدرس تنشر العلم
the effect of inspiration on the VR?
it increases negativity of the intrathoracic pressure thus decreases compression on the veins and casuing its dilatitation and decrasing pressure the veins central so the CVP decreases increaing the pressure graident and the Venous Return >.
expiration and VR ?
This incrasing the intrathoracic pressure and the central veins so the Central venous pressure increases decresing the pressure gradient so the VR is decreased
Factor incrasing the pressure graident by increasing the peripheral pressure :?
1-Arteriolar dilatation !
2-precapillay sphincters constrictions !