Common Shoulder Conditions Flashcards

1
Q

Who is likely to get a dislocated shoulder?

A

• Approximately 1.7% of the population will experience a dislocated shoulder.
• Bimodal distribution:
– 20-30 years (Male:Female 9:1)
– 61-80 years (Male:Female 1:3)
• Less common in children as their epiphyseal plate is weaker and
tends to fracture before dislocating
• More common in elderly: reduced collagen crosslinking → weaker capsule / tendons / ligaments

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2
Q

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A

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3
Q

What contributes to the stability of the glenohumeral joint?

A

• Congruency of humeral head in glenoid
fossa and glenoid labrum
• Negative intra-articular pressure (‘suction cup’ effect)
• Static stabilisers e.g. labrum, joint capsule, extra-capsular
ligaments
• Dynamic stabilisers e.g. rotator cuff muscles, long head of biceps brachii

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4
Q

Why is the glenohumeral joint weak inferiorly?

A

Not much support inferiorly - no muscles there

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5
Q

Describe anterior dislocation

A

• 90% of dislocations are anterior; two subtypes:
– Subcoracoid (60%): Head of humerus lies anterior to glenoid fossa, inferior to coracoid
– Subglenoid (30%): Head of humerus lies antero-inferior to glenoid
– Humeral head dislocates antero-inferiorly
– Disruption of glenohumeral ligaments- they are torn through
– Head pulled anteriorly by muscles (subscapularis, pectoralis major)

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6
Q

What is the anterior dislocation mechanism?

A
• “Hand behind head” =most common
– Abduction
– External rotation 
• Trauma pushes arm posteriorly 
• Humeral head dislocates antero-inferiorly OR 
• Direct blow to posterior shoulder
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7
Q

What is the clinical presentation of anterior dislocation?

A
  • prominent acromium = squaring of the shoulder
  • fullness due to numerous head popping out anterior-inferiorly
  • patient holds the weight of their ar with the other hand when elbow flexed
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8
Q

What is a bankart lesion ?

A

• Tear of glenoid labrum +/- glenoid fracture

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9
Q

What is a Hill-Sachs lesion?

A

• Anterior dislocation
• Posterior aspect of humeral head jammed against anterior lip of glenoid
– Held tightly by muscles e.g. infraspinatus - pulled back forcefully against glenoid lip
• Indentation fracture = ‘dent’
• 50% of patients aged <40 yr with anterior dislocation
• 80% of patients with recurrent dislocation

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10
Q

What are the mechanisms of posterior dislocation

A
• 2-4% of cases Mechanisms:
• Violent muscle contraction
– Epileptic seizure 
– Electrocution 
– Lightning strike
• Blow to anterior shoulder
• Arm flexed across body and pushed posteriorly
Have a high index of suspicion for seizure, electrocution etc.
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11
Q

What are the clinical signs of posterior dislocation

A
  • Squaring of shoulder (loss of normal contour)
  • Arm adducted and internally rotated
  • Prominent coracoid process anteriorly
  • Humeral head may be prominent posteriorly
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12
Q

Describe the radiology of posterior dislocation

A
  • Can be missed on AP X-ray as looks ‘in joint’
  • ‘Light bulb’ sign
  • Increased glenohumeral distance
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13
Q

What is inferior dislocation?

A
• Rare ≈ 0.5% of cases
• Hyperabduction injury
• High rate of associated
injuries:
– Nerves 60% 
– Rotator cuff 80% 
– Vascular 3%
Violent injury - high rate of complication
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14
Q

What are the complications of shoulder dislocation?

A

• Recurrent dislocation
– 60% overall risk
– Risk decreases with age (↓ elasticity of capsule, ligaments etc.)
• Axillary artery damage
– More common in elderly
• Nerve injuries
– Axillary nerve most common
– Cords of brachial plexus
– Other branches e.g. musculocutaneous
• Fractures
– Most common during first-time dislocation
– Humeral head, greater tubercle, clavicle, acromion
• Rotator cuff tears
– More common in elderly and after inferior dislocation
Axillary n.

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15
Q

Describe the anatomy of the clavicle

A

• S-shaped bone
• Acts as a strut between sternum and glenohumeral joint
• Lateral 1/3rd is flat
– Insertion of trapezius
– Origin of deltoid
• Middle 1/3rd is tubular
– Weak to axial load
• Medial 1/3rd is quadrangular
– Insertion of sternocleidomastoid
– Origin of pectoralis major (clavicular head)
• Protects apex of lung, subclavian vessels, and trunks & divisions of brachial plexus

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16
Q

How is clavicle fracture treated?

A

• Most treated conservatively (in a sling)
• Some indications for surgical fixation:
– Complete displacement
– Severe displacement with tenting of skin (risk of conversion to open fracture if bone end pokes through
– Open fracture /
– Neurovascular compromise
– Interposed muscle
– Floating shoulder (clavicle # + glenoid neck #)

17
Q

What are the complications of clavicle fracture?

A
• Non-union 
• Malunion - unite in a bad place 
• Infection (if an open #) 
• Damage to nerves
– Suprascapular nerve - 1
– Supraclavicular nerves -3 (error in TMA)
– Trunks and divisions of brachial plexus
• Vascular
– Subclavian vein and artery 
• Apex of lung
– Pneumothorax
18
Q

What are rotator cuff tears?

A
• Rotator cuff:
– Supraspinatus
– Infrapspinatus
– Teres minor
– Subscapularis
Most common site for tear: 
• Supraspinatus under coraco-acromial arch
19
Q

Describe the pathology of rotator cuff tears

A

• Acute (following trauma) • Chronic
– Degenerative microtrauma model - any problem with the acromium can damage the supraspinatus tendons it comes underneath
Leads to inflammation etc
See slide for dagnra

20
Q

What are the risk factors for rotator cuff tears

A
• Age 
• Recurrent overhead activity
– Painters
– Carpenters
– Swimming
– Volleyball
– Tennis
– Weightlifting 
• Recurrent heavy lifting 
• OA shoulder with osteophytes
• Abnormally-shaped acromion
21
Q

What are supraspinatus tears?

A

• Can be asymptomatic
• Most commonly anterolateral shoulder pain, radiating down arm
• Precipitated by activity (can be present at rest)
– Above shoulder activity (abduction and external rotation)
– Leaning down on elbow (displaces humeral head superiorly)
– Pushing self out of a chair (displaces humeral head superiorly)
– Reaching forward (flexing shoulder) and lifting e.g. milk from fridge
• May also complain of weakness of shoulder abduction but often only found on examination

22
Q

What is impingement syndrome?

A

• Impinge = to rub or catch
• Rotator cuff tendons impinge on coraco-acromial arch
• Caused by anything that narrows subacromial space
– Thickening of coraco-acromial ligament
– Inflammation of supraspinatus tendon
– Subacromial osteophytes in OA
• Pain, weakness and reduced range of motion
• Pain exacerbated by overhead movement

23
Q

What is the painful arc?

A

When shoulder abducted
No pain up to 60o
Pain from 60-120o - due to phase when greater tuberosity negotiates the subacromial space - Due to narrowing of space - impingement
No pain superiorly to that

24
Q

What is calcification supraspinous tendonitis?

A
• Hydroxyapatite crystals in supraspinatus
tendon 
• Pain on abduction or flexion of shoulder
– Reduced coraco-acromial space 
• Mechanical ‘block’ to movement
– Stiffness
– Snapping sensation
– Catching
– Reduced range of movement
25
Q

Describe the pathology of calcification supraspinatus tendonitis?

A
• Multifactorial 
Theory 1: 
• Regional hypoxia 
• Tenocytes → chondrocytes 
• Endochondral ossification 

Theory 2:
• Metaplasia of mesenchymal stem cells into osteogenic cells
• Ectopic bone formation

Crystalline in resting phase
Reabsorbed by phagocytes (“toothpaste”) Reabsorption phase is most painful

26
Q

What is adhesive capsulitis?

A

“Frozen shoulder”
• Glenohumeral joint is inflamed and stiff
• Pain is constant, worse at night
• Exacerbated by movement and cold weather
• Possible autoimmune component, often triggered by trauma to shoulder

27
Q

What are the risk factors of adhesive capsulitis?

A
• Risk factors:
– Female gender
– Trauma to shoulder
• Acute 
• Calcific tendonitis 
• Rotator cuff tear
– Epilepsy with tonic seizures
– Diabetes mellitus (glucose binds to collagen) 
– Connective tissue disease 
– Thyroid disease (↑ and ↓) 
– Inactivity
• Stroke 
• Trauma 
• Illness
28
Q

Describe the treatment o frozen shoulder?

A
  • Physiotherapy
  • Analgesia
  • Anti-inflammatory medication
  • Sometimes manipulation under anaesthesia
  • Usually resolves with time
  • 90% of shoulder motion regained
  • 6-17% risk in other shoulder within 5 years
29
Q

What is osteoarthritis of the shoulder?

A

-

30
Q

Give a treatment overview for

A

(Mild)
Non-pharmacological management - Education, activity modification
2. Non pharmacological management - physiotherapy, simple analgesia e.g. paracetamol
3. Pharmacological management - NSAIDS, opioids, glucosamine + chondroitin sulfate, steroid injection, visosupplementation
4. Surgery - arthroscopy, hemiarthroplasty - semi replacement, total shoulder replacement
(Severe)