Chapter 9 Gastrointestinal, Renal, Endocrine, Immunological System disorders Flashcards

1
Q

Dysphagia and swallowing disorders

Etiologies/types

  1. Facial paralysis
  2. Praxis/motor planning deficits
  3. Sensory impairment of the oral cavity
  4. Weakness of the tongue/base of tongue structures
  5. weakness of the elevation of thepharynx during swallow
  6. vocal cord paralysis
  7. penetration of the bronchioles/bronchi by the bolus when aspiration occurs
  8. Clinical aspiration
A
  1. Facial paralysis - incomplete closure fo the mouth, loss of bolus out of th efron of the oral cavity
  2. Praxis/motor planning deficits - inability to effectvely chew and coordinate tondue movements to propel the bolus toward the base of the tongue
  3. Sensory impairment of the oral cavity - lack of awareness of residual food on the side of the mouth that has decreased sensation, Pocketing food
  4. Weakness of the tongue/base of tongue structures - Inefficient propulsion of bolus at an efficient rate of speed past the base of the tonuge inot the pharyngeal cavitity
  5. weakness of the elevation of the pharynx during swallow - incomplete triggering of the pharyngeal phase of swallowing
  6. vocal cord paralysis - inefficient closure of the vocal folds during the pharyngeal phase of swallow
  7. penetration of the pronchioles/bronchi by the bolus when aspiration occurs - food enters the lun, true aspiration,. can result in introduciton of bacteria, causing pneumonia
  8. clinical aspiration - food enters the airway, can be cleared by airway coughing or it can silently aspirate when the person does not react ot the bolus entering the lung
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2
Q

Dysphagia and swallowing disorders

Bedside swallowing evaluation

A
  1. Assess level of alterness
  2. assess sensory and motor components of swallowing
  3. assess ability to manage own secretions
  4. assess swallowing function using trial boluses - suggest diet modification as indicated
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3
Q

Dysphagia and swallowing disorders

Relationship of swallowing dysfunction to occuaption

A
  • Disruption of the person’s role relative to his/her family unit/decreased ability to comfortably eat at the dinner table (modified diet can be infatilizing
  • Disruption of ability/decreased comfort level for eating out in public
  • Alteration of self-concept concerning life roles and appearance
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4
Q

Dysphagia and swallowing disorders

Interventions

A
  • Provide/family centered intervention to determine an acceptable dinner tabl ealternaitve to interaction
  • work to develop new roles and occupations
  • Provide ongoing education and information to family regarding person’s feeding/nutrition
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5
Q

Gastric Esophageal Reflux Disease (GERD)

  • Symptoms and affects on occupations
  • Intervention
A
  • Symptoms and affects on occupations -
    • Lower esophageal sphincter inefficiently closes, stomach contraction propels acid/acid bolus back into esophagus
    • Symptoms of heart burn, indigestion, or dull chest pain
    • swallowing problems, sensation of feeling that something is getting stuck in their throat.
  • Interventions-
    • sleeping with more than one pillow (elevating the head to discourage regurgitation associated with body posture
    • diet modification (less spice, small meals on more frequent basis)
    • stress management
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6
Q

Small bowel obstruction

  • Etiology
  • Rehab issues
A
  • Etiology - secondary to scar tissue, secondary to radiation of the abdomen, result of tumor obstruction
  • Rehab issues - self-care aspects of stoma care (colostomy) must be addressed for persons with decreased fine motor skills. Decrease mobility of gros movements that cause traction on the healing scar
    • avoid bending, stooping, foot/ lower leg related self-care, dressing, bathinig, nail and foot care.
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7
Q

Neruogenic Bowel

  • Etiology
  • Significance to OT
A
  • Etiology - Sympathetic nerve impairment, generally occuring in persons who have spinal cord injury above the T6 level.
    • Results in loss of analy sphincter control
    • Sensory loss and lack of awareness of feces in bowel
    • Flaccidity of muscles results in incontinence
  • Significance to OT -
    • Autonomic dysreflexia results in an extreme rise in blood pressure.
    • Medical emergency if not reversed
    • complication of a spinal cord injury
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8
Q

Kidney disease

  • general symptoms/impact on performance
    • Motor dysfunction
    • Sensory system function
    • cognitive dysfunction
    • Perceptual/neuro behavioral dysfunciton
A
  • General symptoms/ impact on performance skills and client factors -
    • Motor dysfunciton - fatigue, muscle pain, edema limiting mobility, weakness
    • Sensory system function -
      • Neuropathy (diabetes related, toxicity related )
      • vision loss (diabetes related)
    • cognitive dysfunction - alteration of body image due to dialysis or post transplant, delusions due to sepsis or toxicity
    • Perceptual/neuro behavioral dysfunction - dementia or stroke related
      *
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9
Q

Kidney disease

Intervention/impact on areas of occupation

  • self care
  • instrumental ADL -
  • Social context
A
  • self care - alteration in urination, need for sanitary technique with self dialysis, adherence to restrictive diet, alteration in secuality (impotence, alteration of self image, need for use of adaptived equipment, (tub bench, build-ups, reaching assistive devices,
  • instrumental ADL - housekeeping, need for ligher work load, altered role in family, community mobility (adapted vehicles) meal prep (training to change habits for dietary limiations), problem solve with client on how they can participate in chose ov acitivity with minimal rsk an dawareness of precautions for participation.
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10
Q

Stages of cancer

  • Stage 1
  • Stage 2
  • Stage 3
  • Stage 4
A
  • Stage 1 -
    • tumor present, no percieved spread of disease, leasion operalble
    • Prognosis: good
  • stage 2 -
    • localized spread of the tumor, lesion is operable and can be removed with margins. responds well to treatment (chemo, immunotherapy)
    • Prognosis : 50% survival in 5 years
  • Stage 3 -
    • Extensive evidence of a primary tumor that has spread to other organs in the body, tomor can be surgically debulked but some cells may remain behind, deeper spread of tumor cells in teh lymphatics
    • Prognosis: 20% survival
  • Stage 4 -
    • inoperable primary lesion. Multiple metastases
    • Prognosis: <5%
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11
Q

Cancer

Rehabilitation Pre-operative Intervention

A
  • pre operative functional assessments
  • client and caregiver educaiton concerning recovery and follow up care/functional expectations and client enagement
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12
Q

Cancer

Rehabilitation post-operative Intervention

A
  • Intervention planning is based on medical status
  • Precautions related to structural change from surgery (dependent on location of the tomor, if joint is replaced, abdominal precautions when the tumor ois in the abdominal cavity, regional precautions when there is an incision near a joint
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13
Q

Cancer

Rehabilitation Convalecence Intervention

A
  • rehabilitation of motor impairements, sensory impairments, cognitive impariemtns, neurobehaviroal impairements
  • provide suppor to enhance coping ability during recover from cancer treatment phase
    • Liminality - self recognition of vulnerability and self sense of mortality
    • Occupational rol and body image adjustment
  • development of health supporting behaviors with follow-up support (diet, exercise, stress management, vocational skill support
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14
Q

Cancer

Rehabilitiation End of life care (hospice)

A
  • support quality of life as desease advances and fucntional status declines
  • privde client with as much control as the can and desire to have
  • listen and counsel as possible concerning progression of disease and sense of liminalrity
  • encourage planning for deat, control over goodbyes, funeral arrangements, advenced directives
  • empower life celebration oand life reflection
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15
Q

Scleroderma

  • 3 main componenets
  • Types
A
  • 3 main components -
    • Vascular - Raynaud’s phenomenon, constant recurrent constriction of small blood vessels leading to pulmonary hypertension.
    • Fibrotic - Scar tissue resulting from excess collagen causing thickness of skin and burning sensation in the skin. Fibrosis of the lungs causing restrictive lung disease
    • Autoimmunity - cell-porduced antibodies
  • Types -
    • Limited - Skin involvement (with good prognosis)Linear scleroderma (Bands of thicker skin with good prognosis
    • Systemic - Sclerosis of internal organs (life threatening), CREST with good prognosis
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16
Q

Sequelae of scleroderma and recommendations

Poor ciruclation, as in Raynaud’s phenomenon

A
  1. Use of dressing layers of clothing for warmth
  2. guided imagery to concentrate on improveing distal circulation
  3. education to encourage skin inspection
  4. activity modifications to prevent trauma to fingers and toes.
17
Q

Sequelae of scleroderma and recommendations

Contractures

A
  1. Splinting at optimal resting length for hands/wrists to attempt to slow progressive development of contractures
  2. use of silicone gell in the pams of the hands
  3. Use of electrical/ mechanical vibration to stimulate rapidly adapting type A nerve fibers and decrease burning sensation in hands
18
Q

Sequelae of scleroderma and recommendations

Facial disfigurement and lateration in body image and self-identitity

A
  1. Lood good/ feel better programs
  2. Guide choosing of adaptation and new accessories to eas adjustment to changing appearance
  3. Support groups
19
Q

Secondary conditions/Sequelae of HIV infection

A
  1. Generalized lymphadenopathy/enlarged lymph nodes - fatigue, weight loss, malabsorption of nutrients, general malaise.
  2. Fever, diarrhea which contribut to the dereased tolerance for activity participation and lack of energy.
  3. Neurologicla impairment
    1. Cognitive impairment - safety issues, communication and expression impairements, alterations of personality, decreased ability to engage as before in interpersonal relationships.
    2. Affective changes -
    3. Sensory changes - associated with dementia
    4. Basic ADL- impaired abiliyt to hold an maipulate objects (money, combs, etc
    5. Myelopathy (spinal cord pathology)
    6. Peripheral neruopathy
    7. Visual impairment
20
Q

Rehabilitation for Immunological system Disorders

Interventions for impairment level problems

A
  • Set personal goals to invest behaviorally in one’s helth
  • Provide support to those dealing with immunologicla system disorders
  • provide supportive counseling and suppor tfo psychological disorders
  • Counsel people to be compliant with screening and teatment regimens
  • refer physician for drug therapy
21
Q

Rehabilitation for Immunological system Disorders

Interventions for activity level problems

  • Self care
  • Work
  • Leisure/sports
  • Rest
A
  • self care -
    • adaptations and training to do self care tasks. (ie for scleroderma, alter grasp and pinch patterns and levle of demand and upper extremitity demand, alter size of feeding utensils and tooth brushes to accommodate decreased ability to open mouth)
  • work -
    • work capacity evaluations,
    • modification to work site to alllow participationi in component tasks and activities
    • Counsel for transition to disability status when work is no longer possible
  • Leisure/sports -
    • modify specific tasks and activities
    • Evaluate interests and skills to introduce new leisure activities of interest
  • Rest -
    • Monitor sleep positioing and habits. Interviene when strategies are needed to relax
22
Q

Rehabilitation for Immunological system Disorders

Acute hospitalizaion phase

A
  • Early mobilization
  • Preservation of function
  • positioning
  • psychological/emotional support
  • Prevention of long term disability
23
Q

Rehabilitation for Immunological system Disorders

Inpatient rehabilitation

A
  • Eval and restoration of functional abilitiies (BADL, IADL)
  • Energy conservation and work simplification
  • restoration of activity/exercise tolerance
  • Achievement and maintenance of quality of life
  • Role readjustment intervention
    *
24
Q

Diabetes

general symptoms/ complications

A
  • Symptoms - frequent urination, excessive thirst, unexplained weightloss, extreme hunger, visual changes, sensory changes (tingling/numbness) in hands or feet, fatigue, very dry skin, slow healing wounds
  • Complications -
    • fatigue, dereased actiivty tolerance, urinary distrubances
    • visual loss, low vision, blindness
    • Peripheral nuropathy (amputations)
    • development of wounds
    • increased rate of infections disruptiong live roles and activity participation
25
Q

Diabetes

Hypolycemia

  • Symptoms -
  • Immediate action
A
  • Symptoms - vagueness, dizziness, tachycardia, pallor, weakness, diaphoresis, seizures/coma
  • Immediate action -
    • if person is conscious, provide carbohydrates in teh form of hard candy, fruit juice or honey
    • If person is unconscious, immediately pcall for emergency medical care
26
Q

Diabetes

Hyperglycemic crisis

  • Symptoms
  • immediate action
A
  • Symptoms - Ketoacidosis(dehydration krapid and weak pulse, and acetone breath) Hyperosmolar coma (stupor, thirst, polyuria, and neruologic abnormalities
  • Immediate action - Call for emergency medical services as IV fluids and insuline are required.
27
Q

Diabetes

Rehab focus

A
  • preventative exercise
  • education on medical managment of condiditon
  • life style readjustment to address complications
    • low vision
    • safety, physical adaptation
    • Protective issues regarding peripheral neruropathy, eductation on risk associated with sensory los, skin care,
    • Early attention to wound management - teach skin care and inspection techniques