Chapter 6: States of Consciousness Flashcards

1
Q

What is CONSCIOUSNESS

A

Consciousness: our moment-­‐to-­‐moment awareness of ourselves and our environments.

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2
Q

What are the 4 Characteristics of Consciousness?

A
    1. Subjective and Private
    1. Dynamic (every changing): continuously flowing stream of mental activity rather than disjointed perceptions and thoughts
    1. Self reflective and central to our sense of self: the mind is aware of its own consciousness.
    1. Conscience is intimately linked to selective attention: the process that focuses awareness on some stimuli to the exclusion of others.
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3
Q

What are the 3 Measuring States of Consciousness?

A
  1. Self Report Measures
  2. Behavioural Measures
  3. Physiological Measures
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4
Q

Explain Self Report Measures:

A

• Self Report Measures: directly ask people to describe their inner experiences:

 Not always verifiable or possible to explain (e.g. whilst we are asleep)

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5
Q

Explain Behavioural Measures:

A

• Behavioural Measures: Record, among other things, performance on special tasks

 BM are objective but they require us to infer the persons state of mind.

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6
Q

Explain Physiological Measures:

3 Characteristics

A

• establish the correspondence between bodily processes and mental states. Through electrodes attached to the scalp, the electroencephalograph (EEG) measures brain-­‐wave patterns that reflect ongoing electrical activity of large groups of neurons.

*** Different patterns correspond to different states of consciousness’s.

*** Allows scientists to examine brain activity that underlines various mental states

***They can’t tell us what a person is experiencing subjectively but they have been invaluable for probing the inner workings of the mind.

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7
Q

List the 2 Levels of Consciousness:

A
  1. Freudian Viewpoint

2. Cognitive Viewpoint

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8
Q

Explain Freudian Viewpoint? - Levels of Consciousness

A

The Freudian Viewpoint:
• Generally believed unconscious processes can effect behaviour
• Sigmund Freud proposed the mind consists of three levels of awareness:

  1. The conscious mind: contains thoughts and perceptions of which we are currently aware
  2. Preconscious mental events: our outside current awareness but can not be easily recalled (e.g. memories of a friend)
  3. Unconscious Events: cannot be brought into conscious awareness under ordinary circumstances. Some unconscious content (e.g. an aggressive encounter) is repressed and kept out of conscious awareness because it would arouse anxiety, guilt or other negative emotions.
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9
Q

Explain Cognitive Viewpoint

- Levels of Consciousness

A
  • View conscious and unconscious mental life as complementary forms of information processing that work in harmony.
  • Divides behaviour into:
  • CONTROLLED (CONCIOUS) PROCESSING
  • AUTOMATIC (UNCONSCIOUS) PROCESSING

• With practice, performance becomes more automatic and certain brain areas involved in conscious thought become less active.

• Automatic processing also facilitates divided attention: the capacity to attend to and perform more than one activity at the same time.
—-Divided attention has limits and is more difficult when two tasks require similar mental sources

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10
Q

Cognitive Viewpoint - divides behaviour into

AUTOMATIC VS CONTROLLED PROCESSING — explain

A

—–Controlled (conscious) processing): the conscious use of attention and effort (E.g. planning a party).
Is more flexible and open to change.

—-Automatic (unconscious) processing): performed without conscious awareness and effort (E.g. writing). Flawed in that it reduces our chances of finding new ways to approach problems. Allows for tasks to be performed faster and better.

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11
Q

Unconscious Perception and Influence - STIMULI

A

Unconscious Perception and Influence:

Stimuli can be perceived without conscious awareness and influence how we behave or feel

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12
Q

Unconscious Perception and Influence = List the 4 Ways

A
  1. Visual Agnosia
  2. Blindsight
  3. Priming
  4. The Emotional Unconscious
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13
Q

Explain Visual Agnosia:

A
  • When the brain accurately processes information about the various stimuli and responds accordingly without any conscious thought.
  • Prosopagnosia: can visually recognize objects but not faces. Their brains are recognizing a difference between familiar and unfamiliar stimuli, but this recognition doesn’t reach conscious awareness’s.
  • DF woman with Visual Agnosia could not consciously perceive the shape, size and orientation of objects, yet had little difficulty performing a card-­‐insertion tasks and avoiding obstacles as she walked across the room.
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14
Q

Explain Blindsight:

A
  • People who are blind in part of their visual field yet in special tests respond to stimuli in that field despite reporting that they can’t see those stimuli.
  • E.g. damage to the left hemisphere means a person may be blind in the right half of the visual field yet the stimulus is still perceived unconsciously (guessing accuracy may be 80-­‐100% despite patients saying they can’t see in testing)
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15
Q

Explain Priming:

A
  • When exposure to a stimulus influences how you subsequently respond to that same or another stimulus.
  • People who are subliminally exposed are more likely to complete the word stems with particular words (e.g. you are subliminally exposed to the word hose yet are more likely to complete the stem ho with hose rather than home)
  • E.g. if people are exposed to violent images prior to an image of a person then are more likely to interpret the person from a negative perspective.
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16
Q

Explain Emotional Unconscious

A
  • The belief that emotional and motivational processes operate unconsciously and influence behaviour.
  • E.g. being in a bad mood do to being influenced by events in your environment of which you were not consciously aware
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17
Q

Why do we have consciousness?

A
  • Koch (2004): Conscious awareness provides a summary-­‐ a single mental representation-­‐ of what is going on in your world at each moment and it makes this summary available to brain regions involved in planning and decision making
  • Consciousness helps us override potentially dangerous behaviours governed by impulses or autonomic processing.
  • Consciousness allows us to deal flexibly with novel situations and helps us plan responses to them.
  • Self-­‐awareness and communication enables us to express out needs to other people and coordinate actions with them.
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18
Q

The Neural Basis of Consciousness: WINDOWS OF THE BRAIN.

What are the 2 pathways for processing visual information?

A
  1. Vision for action pathway (neural pathway): extends from the primary visual cortex to the parietal lobe, carries information to support the unconscious guidance of movements.
  2. Vision for perception pathway: extends from the primary visual cortex to the temporal lobe, carries information to support the conscious recognition of objects.
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19
Q

How to study the neural basis of consciousness?

A

• One way of studying the neural basis of consciousness is through using masking to control whether people perceive a stimulus consciously or unconsciously

  • – In experiments participants undergo brain imaging while exposed to masked and unmasked stimuli.
  • – Allow scientists to assess how brain activity differs depending on whether the same stimuli (e.g. photos of angry faces) are consciously or unconsciously perceived.

*** E.g. if an angry face is shown for 30 milliseconds people are aware of seeing the angry face. If following the angry face a neutral face follows for 45 milliseconds people are aware of seeing the neutral face and do not consciously perceive the angry face.

• Emotionally threatening stimuli are processed consciously and unconsciously through different neural pathways.
*** Conscious recognition: prefrontal cortex

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20
Q

Consciousness as a global workspace - explain :

A
  • Views the mind as a collection of largely separate but interacting information-­‐processing modules that perform tasks related to sensation, perception, memory, movement, planning, problem-­‐solving, emotion and so on.
  • The modules process information simultaneously and independently with cross-­‐talk between the (e.g. a answering a math question requires a formula from memory and problem solving skills)
  • Consciousness is a global workspace that represents the unified activity of multiple modules in different areas of the brain.
  • A particular subset becomes joined in unified activity that is strong enough to become a conscious perception or thought.
  • Subjectively we experience consciousness as unitary rather than as a collection of modules and circuits.
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21
Q

What is the Circadian Rhythm?

A

Circadian rhythms: our daily biological clocks

Circadian rhythms: daily biological cycles

 Changes in our core body temperature, certain hormonal secretions, degree alertness, sleeping patterns.

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22
Q

Explain the Process of the Circadian rhythm?

A
  1. Most circadian rhythms are regulated by the brains superchiasmatic nuclei (SCN) in the hypothalamus
  2. SCN neurons have a genetically programmed cycle of activity and inactivity functioning like a biological clock
  3. They link to the pineal gland, which secretes melatonin, a hormone with a relaxing effect on the body.

— During the day: SCN neurons become active and reduce the glans secretion of melatonin, raising body temp and heightening alertness

— Nighttime: SCN neurons are inactive, allowing melatonin levels to increase and promoting relaxation and sleepiness

  1. The circadian clock is biological, but environmental cues such as the day-­‐night cycle help keep SCN neurons on a 24 hour schedule (e.g. the light of day in the morning increases SCN activity and helps rest your 24 hour biological clock)
  2. If you were in a cave with no clock or light source you would drift into a natural wake-­‐sleep cycle called a free-­‐running circadian rhythm that is around 24.2 hours long.
  3. “Early birds” go to bed and rise earlier and their body temperature, blood pressure and alertness peak earlier in the day
     Students from Colombia, India and Spain regions with warmer annual climates exhibited greater morningness than students from the US and Netherlands
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23
Q

What are some Environmental Disruptions Of Circadian Rhythms?

TREATMENT?

A
  1. Jet lag: often causes insomnia and decreased alertness. The body naturally adjusts about one hour or less per day to time zone changes. People typically adjust faster when flying west.
  2. Night-­‐shift work: people work in times when your biological clock is prompting sleepiness. Daytime becomes bedtime and you sleep less overtime leading to fatigue, stress and becoming more accident-­‐prone.
  3. Seasonal Affective Disorder (SAD): is a cyclic tendency to become psychologically depressed during certain seasons of the year. The circadian rhythms of DAF suffers may be particularly sensitive to light, so as sunrises occur later in winter, the daily onset time of their circadian clocks may be pushed back to an unusual degree.
     In tropical areas SAD often occurs in summer due to excessive heat and humidity. In cooler climates it tends to occur during winter and autumn weather.

• Treatments include controlling ones exposure to light, taking oral melatonin and regulating one’s daily activity schedule.

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24
Q

Understand Sleep?

A
  • Roughly every 90 minutes while asleep we cycle through different stages in which brain activity and other psychological responses change in a generally predictable way.
  • EEG recordings of your brain’s electrical activity would show a pattern of BETA waves when you are awake and alert
  • As you close your eyes and feel relaxed and drowsy, brain ways slow down and ALPHA waves occur.
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25
Q

What are the 4 stages of Sleep?

A
  1. Stage 1: As sleep begins your brain-­‐wave pattern comes more irregular and slower Theta waves increase. This stage is a form of light sleep in which you can be awakened easily.
  2. Stage 2: Sleep becomes deeper and sleep spindles (periodic bursts of rapid brain wave activity) begin to appear. Muscles are more relaxed, breathing and heart rate decrease, dreams may occur and you are harder to awake.
  3. Stage 3: the regular appearance of very slow and large delta waves.-­‐ slow wave sleep
  4. Stage 4: the delta waves begin to dominate-­‐ slow wave sleep: you body has relaxed, activity in various parts of your brain has decreased, you are hard to awaken and you may have dreams
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26
Q

What are the 4 stages of Sleep?

A
  1. Stage 1: As sleep begins your brain-­‐wave pattern comes more irregular and slower Theta waves increase. This stage is a form of light sleep in which you can be awakened easily.
  2. Stage 2: Sleep becomes deeper and sleep spindles (periodic bursts of rapid brain wave activity) begin to appear. Muscles are more relaxed, breathing and heart rate decrease, dreams may occur and you are harder to awake.
  3. Stage 3: the regular appearance of very slow and large delta waves.-­‐ slow wave sleep
  4. Stage 4: the delta waves begin to dominate-­‐ slow wave sleep: you body has relaxed, activity in various parts of your brain has decreased, you are hard to awaken and you may have dreams
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27
Q

What happens after 4 stages of Sleep?

A
  • After 20 to 30 minutes of stage-­‐4 sleep your EEG pattern changes as you go back through stages 3 and 2, spending a little time in each.
  • Over 90 minutes the cycle is 1-­‐2-­‐3-­‐4-­‐3-­‐2 (Refer to figure 6.10)
  • As the hours pass stage 4 and then stage 3 drop our and REM periods become longer
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28
Q

Explain REM SLEEP:

A
  • High physiological arousal, frequent dreaming and rapid eye movement characterize stage-­‐5 REM sleep.
  • Bursts of muscular activity causes the sleepers eyeballs to vigorously move back and forth between their closed eyelids.
  • Sleepers awoken in this stage reported having dreams
  • Physiological arousal may increase to daytime levels (e.g. increased heart rate)

• Men have penile erections and women experience vaginal lubrication.

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29
Q

What is REM SLEEP PARALYSIS?

A

• The brain sends signals making it more difficult for voluntary muscles to contact-­‐ e.g. arm and leg muscles become relaxed or ‘paralyzed’ thus this state is termed REM sleep paralysis.

30
Q

EXPLAIN Getting a night’s sleep: from brain to culture

A
  • There is no single area in the brain that regulates sleep – no ‘sleep centre’
  • Areas include: base of the forebrain (basal forebrain), within the brain stem, the pontine reticular formation and REM sleep.

• REM sleep can involve:
 Decreased activation in certain prefrontal lobe regions (involved in high level mental processes and increased activity in parts of the amygdala (regulates emotions) and hypothalamus, thalamus, primary motor cortex (but its signals don’t reach our limbs) and association areas in the occipital lobe (processing of dream images).

  • Several brain regions regulate sleep, and genetic, psychological and environmental and social factors affect sleep duration and quality.
  • Environmental factors such as changes in season effect sleep.
  • Several aspects of sleep such as its timing and length vary across cultures.
  • Cultural norms influence several behaviours related to sleep (e.g. sleeping in a bed vs. a hammock or co-­‐ sleeping where children sleep with their parents.)
31
Q

How much do we sleep?

A

As we age three important changes occur:

  • We sleep less: elderly sleep ~ 6 hours and 19-­‐30 year olds ~ 7.5 hours
  • REM sleep decreases dramatically during infancy and early childhood but remains relatively stable thereafter.

• Time spent in stages 3 and 4 decline. By old age we get relatively little slow-­‐wave sleep
Studies indicate that sleep length and sleep patterns are related to genetic factors and non-­‐genetic factors: differences in marital status, employment, work hours and lifestyle pressures.

32
Q

Explain Sleep Deprivation?

A
  • Sleep deprivation negatively affects mood, followed by cognitive then physical performance, although sleep loss significantly impaired all three behaviours.
  • It takes several nights to recover from extended sleep deprivation and we so not make up all the sleep time that we have lost.
33
Q

List the ways in which WHY DO WE SLEEP?

A
  1. Sleep and Bodily Restoration
  2. Sleep as an evolved adaptation
  3. Sleep and Memory Consolidation
34
Q

Explain Sleep and Bodily Restoration:

A
  • The restoration model proposes that we sleep to recover from physical and mental fatigue.
  • Activities that increase daily wear on the body should increase sleep (e.g. tend to sleep longer on days that we have exercised)
  • Researchers believe this is due to cellular waste product adenosine which as it accumulates inhibits brain circuits responsible for keeping up awake, thereby signaling the body to slow down because too much cellular fuel has been burned. During sleep our adenosine levels decrease.
35
Q

Explain Sleep as an Evolved Adaptation;

A
  • Evolutionary/Circadian models state that each species developed a sleep-­‐wake cycle that maximized its chance of survival in relation to its environmental demands.
  • E.g. ancestors hunted during the daylight and slept at night for safety purposes and ease.
  • Over the course of evolution each species developed a circadian sleep-­‐wake pattern that was adaptive in terms of is status as predator or prey, its food requirements and its method of defense from attack.
  • Sleep may also have evolved as a mechanism for conserving energy-­‐ bodies overall metabolic rate during sleep is about 10 to 20% lower than during waking rest.
36
Q

Explain Sleep and Memory Consolidaton:

A
  • The body needs REM sleep and slow wave sleep and will compensate by increasing the length of these periods if they are disrupted (REM rebound effect)
  • These sleep stages are thought help us remember important information by enhancing memory consolidation: a gradual process by which the brain transfers information into long term memory.
  • Some researchers argue against these theory as drug impaired patients which eliminate or suppress REM sleep do not show impaired abilities to remember new information, experiences or skill.
  • Some researchers argue the function of REM sleep is to keep the brain healthy by offsetting the periods of low brain arousal during restful slow-­‐wave sleep.
37
Q

List some Sleeping disorders?

A
  1. Insomnia
  2. Nacrolepsy
  3. REM Sleep behaviour disorder
  4. Sleep Walking
  5. Nightmares and Night Terrors
  6. Sleep apneoa
38
Q

Explain Insomnia:

A
  • Insomnia: chronic difficulty in falling asleep, staying asleep or experiencing restful sleep/
  • Most common sleep disorder.
  • Some people are genetically predisposed toward insomnia
  • It can be caused by sleep disruption through drug use, shift work, stress and lifestyle factors and medical conditions such as anxiety and depression.

• Treatments include:

  • Stimulus control: conditioning your body to associate stimuli in your sleep environment (such as your bed) with sleep
  • Regular sleep-­‐wake pattern
  • Regular amount of sleep hours throughout the week
  • Avoid napping during the day
  • Avoid stimulants
  • Avoid alcohol and sleeping pills
  • Try to go to bed in a relaxed state
  • Avoid physical exercise before bedtime
  • Avoid performing non-­‐sleep activities in your bedroom.
39
Q

Explain Nacrolepsy

A
  • Narcolepsy: involves extreme daytime sleepiness and sudden uncontrollable sleep attack that may last from less than a minute to an hour.
  • People with it may also experience attacks of cataplexy: a sudden loss of muscle tone often triggered by excitement and other strong emotions.
  • More prone to accidents, impaired quality of life, may be misdiagnosed as having a mental disorder
  • Some people may be genetically predisposition toward developing narcolepsy
  • No cure-­‐ stimulant drugs and daytime naps reduce daytime sleepiness and antidepressants can decrease attacks of cataplexy
40
Q

Explain REM Sleep Behaviour Disorder

A
  • Define: the loss of muscle one that causes normal REM sleep paralysis is absent.
  • If awakened RBD patients often report a dream content that matches their behaviour (e.g. that they were flailing their arms-­‐ which they do in their sleep)
  • They may kick violently, throw punches, get out of bed and move about wildly.
  • Research suggests that brain abnormalities may interfere with signals from the brain stem that normally inhibit movement during REM sleep, but in many cases the cases of RBD are unknown.
41
Q

Explain Sleep Walking

A
  • Usually occurs during stage3 or stage 4 period of slow-­‐wave sleep.
  • Sleepwalkers often stare blankly and are unresponsive to other people.
  • Many seem vaguely conscious of their environment.
  • People often awake with no memory of the event.
  • A tendency to sleepwalk by be inherited and daytime stress, alcohol and certain illnesses and medications increase sleepwalking.
  • Treatments: psychotherapy, awakening children before the time they typically sleepwalk or creating a safe sleeping environment for a child to outgrow it in.
42
Q

Explain Nightmares and Terrors

A
  • Night mares: Usually occur during REM sleep.
  • Night Terrors: are frightened dreams that arouse the sleeper to a near-­‐panic state. Are most common during slow-­‐wave sleep (stages 3 and 4) and are more intense and involve greatly elevated physiological arousal – heart rate increases
43
Q

Explain Sleep APNOEA

A
  • People with sleep apneoa repeatedly stop and restart breathing during sleep.
  • It is most commonly caused by an obstruction in the upper airways-­‐ the chest and abdomen keep moving but no airs get to the lungs. Finally reflexes kick in and the person gasps and breathes and is awoken.
  • The person falls asleep again without recollection of being awake.
  • Resolved through surgery to remove the obstruction or wearing a mask that pumps air into the passages.
44
Q

The nature of dreams: Why do we dream and what do we dream about?

A
  • Hypnagogic State: the transitional state from wakefulness through early stage-­‐2 sleep-­‐ continued mental activity became less ‘thought’ like and more ‘dream like’
  • Dreams occur throughout sleep but are most common during REM periods when activity in many brain areas is highest
  • Across most cultures most of our dreams contain negative content.
  • Our cultural background, gender difference, current concerns and recent events influence what we dream about.
45
Q

List Theories on why we dream:

A
  1. Freud’s Psychoanalytic Theory
  2. Cognitive Theories
  3. Activation-Synthesis Theory
  4. Neurocognitive Theories
46
Q

DREAMS - FREUD’S PSYCHOANALYTIC THEORY

A
  • Freud proposed that dreams are the gratification of our unconscious desires and moods:
  • Freud distinguished between:
  1. A dreams manifest content, the surface story that the dreamer reports
  2. It’s latent content, which is its disguised psychological meaning.
  • Dream Work: the process by which a dream’s latent content is transformed into the manifest content. It occurs through symbols and by creating individual dream characters who combine features of several people in real life.
  • Critics of dream analysis say that is its highly subjective; the same dream can be interpreted differently to fit the particular analyst’s point of view
47
Q

DREAMS - COGNITIVE THEORIES

A

Cognitive Theories: Foulkes (1982)

  • According to problem solving dream models, because dreams are not constrained by reality they can help us find creative solutions to our problems and ongoing concerns.
  • Critics argue that because so many of our dreams don’t focus on personal problems, its difficult to see how problem solving can be the broad underlying reason for why we dream.
  • Cognitive process dream theories focus on the process of how we dream and propose that dreaming and waking thought are produced by the same mental systems.
  • Rapid content shifting of attention is a process common to dreaming and waking mental activity.
48
Q

DREAMS - ACTIVATION SYNTHESIS THEORIES

A

Activation-­‐Synthesis Theory: Hobson and McCarley (1977)

  • Activation-­‐synthesis Theory: dreams do not serve any particular function – they are merely a byproduct of REM neural activity.
  • Activation-­‐synthesis theory views dreaming as the brains attempt to fit a story to random neural activity.
  • During REM sleep the brain stem bombards our higher brain centers with random neural activity (the activation component)
  • Because we are asleep, this neural activity does not match any external sensory events, but our cerebral cortex continues to perform its job of interpretation.
  • It does this by providing a dream-­‐ a perception-­‐ that provides the best fit to the particular pattern of neural activity that exists at any moment.
  • Our memories, experiences, desires and needs can influence the stories that our brain develops (have a meaning)
  • Critics claim that the activation-­‐synthesis theory overestimates the bizarreness of dreams and pays too little attention to NREM dreaming.
49
Q

DREAMS - NEUROCOGNITIVE THEORIES

A
  • Bridge the cognitive and biological perspectives by attempting to explain how subjective aspects of dreaming correspond to physiological changes that occur during sleep.
  • Dreaming involves and integration of perceptual, emotional, motivational and cognitive processes performed by various brain modules.
  • These models acknowledge that motivation factors-­‐ our needs and desires-­‐ can influence how the brain attaches meaning and emotion to the neural activity that underlies our dreams.
  • E.g. activation-­‐synthesis.
50
Q

How do factors affect sleep and dreaming? BIOLOGICAL

A
  • Circadian rhythms affect our readiness for sleep
  • Different species have evolved different sleep-­‐wake cycles
  • Certain brain circuits actively promote falling asleep, while others regulate various sleep stages
  • Sleep stages are marked by distinct patterns of physiological activity
  • Heredity partly accounts for differences among people in sleep length and the likelihood of developing sleep disorders
51
Q

How do factors affect sleep and dreaming?

ENVIRONMENTAL AND SOCIAL

A

• The day-­‐night cycle and time cues help regulate our circadian rhythms and sleep readiness.

• Night-­‐shift work and jet travel across time zones can disrupt circadian rhythms
and impair sleep

  • A noisy sleep environment can impair sleep quality.
  • Experiences from waking life can show up in our dream content
  • Cultural norms influence sleep-­‐related behaviours (e.g. co-­‐sleeping) and meaning attached to dreams
52
Q

How do factors affect sleep and dreaming? PSYCHOLOGICAL

A
  • Worries and stress may hinder falling asleep or contribute to other sleep problems
  • Learned sleep habits can facilitate or impair a sound nights sleep
  • Mental activity occurs throughout sleep, ranging from fragmented through and images to story like dreams.
  • Ongoing psychological problems or concerns may show up in our dream content.

• Dreaming has be theorized to serve various psychological functions, including memory consolidation, unconscious wish fulfillment and problem
solving

53
Q

How do drugs pass into the brain?

A

• Drugs pass through the BLOOD–BRAIN BARRIER and alter consciousness by facilitating or inhibiting synaptic transmission.

54
Q

Explain the 3 steps in SYNAPTIC TRANSMISSION

A
  1. Neurotransmitters are synthesised inside presynaptic (sending) neurons and stored in vesicles.
  2. They are released into the synaptic space, where they bind with and stimulate receptor sites on postsynaptic (receiving neurons)
  3. Neurotransmitter molecules are deactivates by enzymes or by reuptake.
55
Q

What are AGONISTS? What do they Do?

A

• Agonists are drugs that increase a neurotransmitter system’s activity:

—Enhance a neurons ability to synthesize, store or release neurotransmitters

—Bind with and stimulate postsynaptic receptor sites (or make it easier for neurotransmitter to stimulate these site)

56
Q

What are ANTAGONISTS?

What do they do?

A

• Antagonist: is a drug that inhibits or decreases the action of a neurotransmitter

– Reduce a neurons ability to synthesize, store or release neurotransmitters

– Prevent a neurotransmitter from binding with the postsynaptic neuron, such as by fitting into and blocking the receptor sites on the postsynaptic neuron.

57
Q

What is Tolerance? What causes Dependence? Withdrawal?

DRUGS

A
  • TOLERANCE: decreasing responsivity to a drug administered at the same dosage level over time.
  • Person may take increasingly larger does to achieve the desired physical and psychological effects.
  • If a drug changes bodily functioning in a certain way, the brain tries to restore balance (HOMEOSTASIS) by producing compensatory responses which are reactions opposite to that of the drug (e.g. lowering heart rate when a drug stimulates increased heart rate.
  • When drug use is stopped compensatory responses continue and produce withdrawal symptoms.
  • WITHDRAWAL: is the occurrence of compensatory responses after discontinued drug use.
  • Substance dependence is a maladaptive pattern of drug abuse.
58
Q

Learning, drug tolerance and overdose - Classical Conditioning?

A
  • Environmental stimuli associated with repeated drug use begin to elicit compensatory responses through a learning process called classical conditioning – leading to cravings when they enter a setting associated with drug use as the environmental stimuli triggers compensatory responses that without drugs to mask their effect, cause the user to feel withdrawal symptoms.
  • If the user takes their usual high dose in a familiar environment the bodies compensatory responses are at full strength-­‐ a combination of compensatory reactions to the drug itself and also to the familiar, conditioned environmental stimuli.
  • But in unfamiliar environment the conditional compensatory responses are weaker and the drug has a stronger physiological net effect than usual
59
Q

Types of DRUG ADDICTION AND DEPENDENCE (3)

A
    1. Substance dependence: a maladaptive pattern of substance use that causes a person significant distress or substantially impairs that persons life
    1. Physiological dependence: if drug tolerance or withdrawal symptoms have developed.
    1. Psychological dependence: situations in which people strongly crave a drug because of its pleasurable effects, even if they are not physiologically dependent.
60
Q

Some misconceptions about Substance dependence

A
  • People can become dependent on drugs, that produce only mild withdrawal (cocaine). The drug’s pleasurable effects-­‐ often produced by boosting dopamine activity-­‐ play a key role in causing dependence.
  • May drug users who quit and make it through withdrawal eventually start using again, even though they are no longer physiologically dependent.
  • Many factors influence drug dependence, including genetic predispositions, religion beliefs, family and peer influences and cultural norms.
61
Q

Depressants: Physiological effects, Examples, Typical effects, Risk of high and/or chronic uses

A

**Decrease neural activity.

  1. Alcohol =
    Relaxation, lowered inhibition,
    impaired physical and psychological functioning Disorientation,
    unconsciousness, possible death
  2. Barbiturates, tranquilisers =
    Reduced tension, impaired reflexes and motor functioning drowsiness Shallow breathing, clammy skin, weak and rapid pulse, coma,
    possible death
62
Q

Stimulants: Physiological effects, Examples, Typical effects, Risk of high and/or chronic uses

A

**Increase neural firing and arouse the nervous system boost mood

  1. Amphetamines, 2. Cocaine, 3. Ecstasy

Increased alertness, pulse and blood pressure; elevated moos, suppressed appetite, agitation, sleeplessness

Hallucinations, paranoid delusions, convulsions, long-­‐term impairments, brain damage, possible
death

63
Q

Opiates: Physiological effects, Examples, Typical effects, Risk of high and/or chronic uses

A

**Stimulate receptors normally activated by endorphins, thereby producing pain relief, and increase dopamine activity which can induce euphoria and mood change.
Highly addictive

  1. Opium, 2. morphine, 3. codeine, 4. heroin

Euphoria, pain relief, drowsiness, impaired motor and psychological functioning

Shallow breathing, convulsions, coma, possible death

64
Q

Hallucinogens: Physiological effects, Examples, Typical effects, Risk of high and/or chronic uses

A

***** Powerfully distort sensory experience and can blur the line between
reality and fantasy

1.LSD, 2. mescaline, 3. phencyclidine

Hallucinations and visions, distorted time perception, loss of contact with reality, nausea Psychotic reactions (delusions, paranoia), panic, possible death

65
Q

Marijuana: Physiological effects, Examples, Typical effects, Risk of high and/or chronic uses

A

**THC binds to receptors on neurons throughout the brain – increases GABA activity and slows down neural activity, producing relaxing effects, increases dopamine activity which has
pleasurable effects

Mild euphoria, relaxation, enhanced sensory experiences, increased appetite, impaired memory and reaction time

Fatigue, anxiety, disorientation, sensory distortions, possible psychotic reactions, exposure to carcinogens.

66
Q

What do drugs depend on?

A

A drugs effect depends on its chemical actions, the physical and social setting, cultural norms and learning, the users genetic predispositions, expectations and personality.

67
Q

Drug Induced states involve the interplay of BIOLOGICAL factors:

A
  • Drugs increase or decrease the activity of particular neurotransmitter systems
  • The body produces compensatory responses to counteract a drug’s effect, possibly leading to tolerance
  • Withdrawal symptoms occur when drug use stops, but the body’s compensatory responses continue
  • Genetic factors influence biological reactivity to specific drugs
68
Q

Drug Induced states involve the interplay of PSYCHOLOGICAL factors:

A
  • Drugs can alter numerous aspects of psychological functioning, including mood, memory, attention, decision making, social inhibition’s and pain awareness
  • User’s attitudes and expectations about drugs can influence their psychological reactions to a drug

• A user’s level of personal adjustment can influence the likelihood of a negative drug
reaction

69
Q

Drug Induced states involve the interplay of ENVIRONMENTAL AND SOCIAL factors:

A
  • Cultural norms and experiences can shape users drug attitudes and expectations
  • Repeated drug use in a particular setting can produce conditioned compensatory stimuli
  • The social context and behaviour of other drug users who are present can affect how a person responds to the drug
70
Q

Explain in detail what HYPNOSIS IS

A
  • Hypnosis: is a procedure in which one person in guided by another to respond to suggesting for changes in subjective experience, alterations in perception, sensation, emotion, thought or behaviour.
  • Hypnosis involves an increased receptiveness to suggestions. Hypnotized people experience their actions as involuntary, but hypnosis has no unique power to make people behave against their will or perform amazing feats. Hypnosis increases pain tolerance, as do other psychological techniques
  • Some people can be led to experience hypnotic amnesia and posthypnotic amnesia
71
Q

Explain DISSOCIATION THEORIES view on hypnosis

A

• Ernest Hilgard (1994) proposed that hypnosis creates a division of awareness in which the person simultaneously experiences two streams of consciousness cut off from the other:

*** One stream: responds to the hypnotists suggestion

*** Second stream: the part of the consciousness that monitors behaviour-­‐ remains in the background but it aware of everything that goes on (‘ the hidden observer’)

• This dissociation explained why behaviours that occur under hypnosis seem involuntary or automatic. The mainstream consciousness that responds to the command is blocked by this awareness and perceives that body is involuntarily performing an act.

72
Q

Explain Social Cognitive theories on hypnosis:

A
  • Social cognitive theories propose that hypnotic experiences result from expectations of people who are motivated to take on the role of being hypnotized.
  • People motivated to conform to the role of being in a trance-­‐like state and responsiveness develop a readiness to respond to the hypnotist’s suggestions and to perceive hypnotic experiences as real and involuntary.
  • Our expectations strongly influence how the brain organizes sensory information-­‐ hypnotic responses are still completely real and may indeed represent altered experiences

Brain imaging reveals that hypnotized people display changes in neural activity consistent with their subjectively changes in neural activity consistent with their subjectively reported experiences. This supports the view that hypnosis involves an altered state but doesn’t establish whether it is a dissociated state.