Chapter 19 Flashcards

0
Q

45% of blood. Made up of platelets, WBCs, and RBCs

A

Formed elements

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1
Q

Made of water and proteins such as albumins, globulins, and fibrinogen. 55% of blood

A

Blood plasma

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2
Q

Platelets have a _______ life span of ___ to ____ days.

A

Short, 8 to 10

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3
Q

Hormone produced by the liver that stimulates the formation of platelets from megakaryocytes.

A

Thrombopoietin

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4
Q

Process by which the formed elements of blood develop.

Before birth, this occurs in sac of embryo. But red bone marrow becomes the primary site.

A

Hematopoiesis

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5
Q

Increases the number of red blood cell precursors. Produced primarily by cells in kidneys. If kidneys fail, RBC production is inadequate. Bad hematocrit-> no O2 to body tissues.

A

Erythropoietin

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6
Q

Store, accumulate, and pass along o2 very well. Floppiness allows them to flow through capillaries.

Don’t have nucleus, can’t produce proteins, not really living cells, die in abt 120 days. 4-6 mil per microliter

A

Red blood cells

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7
Q

Immature RBCs. Low count indicates low erythropoietin rate. Elevated rate vise versa. As the cell matures, the nucleus disappears, and amt of hemoglobin increases.

A

Reticulocytes

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8
Q

This is packed inside RBC. Carries o2 and co2. Each RBC contains 280 mil molecules of hgb

A

Hemoglobin

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9
Q

Excess RBCs

A

Polycythemia

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10
Q

Weird shaped cells by beta globins

Super bad…

A

Sickle cell anemia

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11
Q

Loss of lots of blood can lead to this

A

Hemorrhagic anemia

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12
Q

Insufficient RBCs, or hemoglobin

A

Anemia

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13
Q

WBC that fights bacterial infection. Is active in phagocytosis.

A

Neutrophil

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14
Q

WBCs that increase during parasitic infection.

A

Eosinophils

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15
Q

WBCs present in histamine reactions. Big role in inflammatory response.

A

Basophils

16
Q

Not granulocytes. They are phagocytes.

A

Monocytes

17
Q

WBCs that aren’t granulocytes. Increase in acute viral infections. Responders to specific foreign antigens. Nucleus takes up whole cell.

A

Lymphocytes

18
Q

Process that stops bleeding

A

Hemostasis

19
Q

Occurs as damaged blood vessels constrict.first stage of hemostasis

A

Vascular spasm

20
Q

Platelets adhere to endotherm that’s damaged. Second stage of hemostasis.

A

Form platelet plug

21
Q

Blood clot. 3rd stage of hemostasis

A

Coagulation

22
Q

2 pathways to coagulation cascade

A

Intrinsic and extrinsic

23
Q

Clotting in an unbroken blood vessel

A

Thrombosis

24
Q

The clot itself

A

Thrombus

25
Q

Drugs that stop/ reverse thrombosis

A

Anticoagulants (rat poison)

platelet inhibiting drugs. Aspirin like

26
Q

Thrombus breaking loose ( embolus), can lead to….

A

Stroke

27
Q

Baby can’t clot. Hereditary genetic disorder.

A

Hemophilia

28
Q

Blood coagulates almost instantly. Found in shrimp shells. Parasitic infection.

A

Chitosan patches (hemostatic bandage)

29
Q

Used for diagnostic testing bc it won’t coagulate in machine

A

Serum