CHAPTER 13: FLAPS Flashcards

1
Q

Medial plantar flap - anatomy & usage

A

Cutaneous flap of glaborous skin
Artery - medial plantar perforator (landmark = medial cuneiform plantar surface) from medial plantar artery (from posterior tibial artery)
Veins - venae comitantes & superficial veins
Sensory - medial planatar or saphenous branch (if present)

Locate PT artery at behind medial malleolus
Tom, Dick ANd Harry - from ant to post
1. Tib post
2. FDL
3. PT artery and tibial nerve
4. FHL

Flap usage

  • small defects around medial malleolus, distal medial foot or medial first metatarsal head.
  • Calcaneal defects if pedicle is traced back to PT
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2
Q

Medial plantar flap - raising

A

Flap 7 x 2cm
Doppler perforator
- flap can be designed eccentrically (if used for local rotation) or centrally (if free)
Axis of flap = plantar edge of the first metatarsal, first cuneiform, and navicular

  • First incision made at plantar aspect of flap
  • Enter fascia over abductor hallucis is entered
  • Raise flap from plantar to dorsal
  • Perforators between abductor hallucis and flexor hallucis tendons are identified and preserved
  • abductor hallucis muscle is retracted and the perforators are followed to their origin on the medial plantar system
  • superficial vein is traced from dorsal incision
  • donor site closed directly / SSG
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3
Q

Flaps to look up

A

lateral arm flap

dcia

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4
Q

Complications of free fibula flap

A
  • leave at least 6cm of proximal fibula to avoid destabilising knee and damage to common peroneal nerve
  • CT angiography is recommended, as peroneal artery may be dominant in 8% patients (peroneal arteria magna) / the only perfusing artery in PVD or trauma pts
  • identify and preserve lesser saphenous vein and sural nerve for skin paddle
    can increase bone - bone contact with step / wedge osteotomy
  • Include cuff of soleus muscle or flexor hallucis longus muscle to improve reliability of skin paddle if skin perforators are small
  • 6 to 8 cm of distal fibula must remain intact to stabilise ankle. In skeletally immature patients, perform a syndesmosis at the lateral malleolus following fibula harvest
    Once fibula is visualised, dissection continues in a subperiosteal plane at superior margin of fibula until peroneal nerve is identified; then dissection may be continued superficial to periosteum to prevent iatrogenic deep peroneal nerve injury.
  • tibial nerve and posterior tibial artery must be identified before ligating vessels and harvesting flap
  • keep operating field dry during dissection of FHL off the fibula and peroneal vessels, visualise and ligate all branching vessels from the peroneal bundle.
  • ensure pedicle is not compressed, twisted or kinked. Use wide exposure of recipient site and inset under clear visualisation. Cover pedicle with SG if necessary
  • After preparing fibula for inset, perform 1-2cm subperiosteal dissection to separate pedicle of flap and prevent kinking
  • unicortical screws prevent pedicle injury.
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5
Q

What are the risks of free fibula flap?

A
  • bleeding -> compartment syndrome
  • damage to common? deep / superficial peroneal nerve
  • sural nerve sacrificed - parasthesia lat foot and leg
  • swelling, lymphoedema
  • stiffness
  • unstable ankle - problems with running
  • flap failure
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6
Q

What is a flap?

A

A unit of tissue that maintains its own blood supply while being transferred from a donor site to a recipient site.

Most flaps can be classified according to:

  1. method of mvmt
  2. vascularity
  3. tissue composition
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7
Q

What kinds of local flaps are there?

A

Advancement - slides directly forward into defect
Rotation - semicircular flap rotated about a pivot point into a defect to be closed
Transposition - rotated laterally about a pivot point into an immediately adjacent defect
e.g. Z plasty, rhomboid (Limberg), Dufourmentel

Interpolation - rotates on a pivot point into a defect, with pedicle passing over or under intervening tissue e.g. deltopectoral, Littler NV digital pulp flap

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8
Q

What is a Z plasty?

What are the theoretical gains in length?

A

2 opposing triangular transposition flaps

Degrees            % gain in length
30                             25
45                             50
60                             75 
75                             100
90                             120
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9
Q

What are distant flaps?

A

where donor and recipient sites are not close to one another
e.g. thenar, cross-leg, groin
When 2 sites cannot be approximated, flap can be ‘waltzed’

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10
Q

What is a free flap?

A

a unit of tissue with an artery and vein is divided and reanastomosed at the recipient site with blood flow re-established

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11
Q

what is an angiosome? Who described this?

A

Taylor and Palmer

- a composite unit of skin with its underlying deep tissue supplied by a source artery

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12
Q

What is the fasciocutaneous plexus?

Who described the the different vessel types that perforate the deep fascia to supply the fasciocutaneous plexus?

A

A communicating network of subfascial, intrafascial, suprafascial, subcutaneous and sub dermal vascular plexuses

Nakajima

  1. direct cutaneous branch of a muscular vessel
  2. septocutaneous perforator
  3. direct cutaneous
  4. musculocutaneous
  5. direct septocutaneous
  6. perforating cutaneous branch of a muscular vessel
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13
Q

Cutaneous flaps: how are they classified?

A

McGregor and Morgan

  1. Random pattern: based off the subdermal plexus, limited to 3:1 length:width ratio
  2. Axial pattern: contains a specific direct cutaneous artery within the longitudinal axis of the flap

(3. Reverse axial pattern flaps (reverse flow flap)
4. Island flap)

Nakajima: classified cutaneous flaps based on vascularisation

  1. cutaneous (equivalent to axial flap)
  2. fasciocutaneous
  3. adipofascial
  4. septocutaneous
  5. musculocutaneous
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14
Q

How are perforator flaps classified?

How are they named?

A

Direct / indirect

  1. Musculocutaneous (indirect)
  2. Septocutaneous (indirect)
  3. Direct cutaneous (direct)

Named after nutrient vessel (e.g. DIEP) unless multiple flap can be raised from the nutrient vessel e.g. ALT flap from descending branch of lat circ femoral

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15
Q

What are the advantages and disadvantages of perforator flaps?

A

Adv
numerous donor sites, versatile size and thickness
can incorporate muscle, fat bone into flap design
preserve muscle function
reduce donor site morbidity

Disadv
tedious pedicle dissection
perforator anatomy variable
increased risk of fat necrosis cf musculocutaneous flaps

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16
Q

What other flaps do you know of?

A

Neurocutaneous & venocutaneous flaps - based on perforating arteries accompanying cutaneous nerves and veins (e.g. sural, saphenous)

Venous flaps - supplied through a venous pedicle
Type I - single-pedicled: in & outflow through single vein
Type II - bipedicled ‘flow-through’ flap
Type III - arterialised venous flap: artery anastomosed to vein proximally and vein outflow distally

17
Q

How are flaps classified by tissue composition?

A
Fascial and fasciocutaneous flaps
Muscle and myocutaneous flaps
Vascularised bone flaps
Visceral flaps
Innervated flaps
Compound and composite flaps 
Prefabridcated flaps
18
Q

Who classified fasciocutaneous flaps?

A

Mathes
Direct cutaneous pedicle
Septocutaneous
Musculocutaneous

19
Q

Who classified muscle flaps?

A
Mathes and Nahai
I One dominant pedicle
II Dominant pedicle & minor pedicle (s)
III Two dominant pedicles
IV Segmental pedicles
V One dominant & secondary segmental pedicles
I TFL
II Gracilis
III Rectus abdominis, gluteus maximus
IV Sartorius
V Latissimus dorsi
20
Q

Tell me about MC vs FC flaps

A

all demonstrate marked increase in blood flow
decrease in bacterial concentration MC>FC (count = 100 vs 10000)
collagen deposition MC>FC

21
Q

Bone flaps

A

supplied by their nutrient vessels
can reconstruct large defects, withstand DXT and implantation
e.g. fibula (peroneal artery), radius (radial), iliac crest (DCIA)

22
Q

Visceral flaps

A

e.g. jejunum, colon, omentum

23
Q

Innervated flaps: functional muscle and sensory flaps

A

functional muscle: motor nerve coated after free flap transfer
e.g. gracilis, LD, serratus, pec minor

24
Q

Compound and composite flaps

A
Contains diverse tissue components
Solitary composite
Siamese
Conjoint
Sequential
25
Q

Prefabricated flaps

A

2 stage technique where flap is surgically altered by partial elevation, structural manipulation and incorporation of other tissue layers in the 1st stage to create a specialised composite flap
e.g. nasal recon

26
Q

How is blood flow to the skin regulated?

A

Systemic
1. Neural: sympathetic alpha adrenergic (vasoconstriction), beta adrenergic (vasodilatation). Cholinergic (bradykinin release, vasodilatation)
2. Humoral
Vasoconstriction: adr, noradr, serotonin, thromboxane A2, PGF2alpha
Vasodilatation: bradykinin, histamine, PGE1

Local (autoregulation)
Metabolic
Vasodilatation: hypercapnia, hypoxia, acidosis, hyperkalaemia

Physical
Vasoconstriction: myogenic reflex in response to distension of cutaneous vessels; local hypothermia and increased blood viscosity

27
Q

What happens to blood flow when a flap is transferred?

A

Homeostasis is disrupted, including

  • loss of sympathetic innervation
  • ischaemia (anaerobic metabolism, lactate, superoxide radical, changes in blood viscosity and clotting)

Ischaemia induced reperfusion injury is direct cytotoxic injury from free radicals during flap ischaemia. Fat and bone can tolerate ~3hrs, muscle and GI mucosa much less.

28
Q

What is the theory of flap delay?

A

Flap delay: surgical interruption of a portion of the blood supply to a flap at a preliminary stage (10d - 3wks) before transfer

  1. Conditions flap to ischaemia
  2. Opens choke vessels

Tissue expansion is also form of delay

29
Q

What are the 5 mechanisms of delay?

A
  1. Sympathectomy
  2. Vascular reorganisation
  3. Reactive hyperaemia
  4. Acclimatisation to hypoxia
  5. Non-specific inflammatory reaction
30
Q

What factors contribute to increase flap survival?

A

Physical
warm, well filled, pain free

Pharmacological

  1. Anticoagulants: heparin, thrombolysis (streptokinase, t-PA), leeches
  2. Vasodilators: Ca channel blockers, topical GTN, topical lignocaine
  3. Antiinflammatory agents: aspirin
31
Q

How are flaps monitored?

A
  1. Clinical - subjective: colour, cap refill, warmth, turgor, scratch test
    Temp difference >3 degrees = significant
  2. Vital dye
    Fluorescein
  3. Photoelectric
    US doppler
    Doppler venous coupler
    Laser doppler
  4. Metabolic
    transcutaneous O2 tension (measures O2 partial pressures)
    Photoplethysmography - pulse ox, infrared light measures fluid vol by different light absorption by skin, disappointing results clinically