CHAPTER 12: HAND - INFECTION, TRAUMA, TRIGGER FINGER, CRPS Flashcards

1
Q

Hand infections - organisms

A

Usually Staphylococcus aureus
Human Bites → mixed aerobe and anaerobes
Cat and Dog bites → Pasteurella multocida
Leeches → Aeromonas hydrophilia - Ciprofloxacin

Aerobic

  • Staph aureus
  • Staph epidermidis
  • Strep

Anaerobic

  • Peptostreptococcus
  • Peptococcus
  • Bacteroides
  • Eikenella corrodens
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2
Q

What are the different types of infections of the fingertip?

A

Acute paronychia

  • infection of tissue proximal to nail fold, b/t germinal matrix and skin
  • drain by separating nail plate from eponychial fold or incising over collection

Chronic paronychia

  • Chronic inflammation → thickening of the cuticle and grooving of the nail
  • Candida albicans

Felon

  • infection of pulp, abscess
  • drain over maximum fluctuance
  • If untreated → osteomyelitis, septic arthritis, flexor sheath infection, tender pulp scar

Herpetic whitlow

  • HSV → superficial fingertip infection
  • small clear vesicles at early stages
  • DO NOT I&D → bacterial superinfection
  • acyclovir
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3
Q

What are the signs of flexor sheath infection?

A

Kanavel’s signs

  1. fusiform swelling
  2. flexed posture
  3. sheath tenderness
  4. pain on passive extension
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4
Q

Treatment of flexor tenosynovitis?

A

emergency

drain and irrigation of sheath

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5
Q

Where can flexor sheath infections of the thumb and little finger drain through?

A
  • can drain through the radial and ulnar bursae → space of Parona = horseshoe abscess.

Lies immediately superficial to PQ at the wrist.

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6
Q

What are the 3 potential spaces in palmar infections?

A
  1. Thenar space
    → radial to oblique septum (from palmar fascia to 3rd MC). Index infections spread here
  2. Mid-palmar space
    → ulnar to oblique septum. Middle and Ring infections spread here
  3. Hypothenar space → rare

Distinguish from radial and ulnar bursae
→ synovial sheaths which enclose flexor tendons of thumb and little finger
Communicates proximally with space of Parona

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7
Q

Where else can infections in the hand occur?

A

Web space infections = collar stud abscess
divided by palmar fascia

Treatment - I&D via volar zig-zag and dorsal longitudinal incisions

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7
Q

What are the 2 potential spaces of the dorsum?

A
  1. Dorsal subcutaneous space
    → large potential space overlying entire dorsum of hand.
    - Communicates in the finger webs with potential space immediately beneath palmar fascia.
  2. Dorsal subaponeurotic space
    → just below extensor retinaculum
    - Drainage by incisions over 2nd MC and b/t 4th and 5th MCs
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8
Q

Hand trauma history.

A
  • Hand dominance, occupation, hand-critical hobbies, smoking.
  • Mechanism of injury and forces involved.
  • Time of injury, ischaemia time.
  • Machinery: type etc.
  • Direction of laceration.
  • Position of the hand at time of injury.
  • Contamination.
  • Immunisation (tetanus).
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9
Q

Hand trauma examination.

A
  1. Is injured hand or finger viable?
  2. Vascular injury or compartment syndrome?
  3. Tendon, nerve or bony injury?
  4. Skin loss?
  • X-rays.
  • Fractures and dislocations
  • Tendons (posture of hand, tenodesis test, pain on resisted flexion / extension suggests partial damage)
• Composite motion produces the following grip types:
1. Power grip
2. Pinch grip
– Pure (tip) pinch
– Tripod pinch
– Key (lateral) pinch.

• Nerve injuries
1. Sensory:
– Sweating - tactile adherence test.
– Static and dynamic 2PD.

  1. Motor
    – APB = median.
    – FDM = ulnar.
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10
Q

What are the extensor compartments of the hand?

A
  1. EPB & APL
  2. ECRB & ECRL
  3. EPL
  4. EDC & EIP
  5. EDM
  6. ECU
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11
Q

What is De Quervains tenosynovitis?

What is Intersection syndrome?

A

DQ

  • Middle aged women
  • ‘Normal’ anatomy in fewer than 20%
  • EPB and APL entrapment – sit in 1st dorsal compartment of wrist
  • Management – Splints, steroids (but can get skin / subcut / fat atrophy, use dilute preps)
  • In pregnancy/lactation - resolves

IS

  • Entrapment of 2nd dorsal comp
  • Management - Non-op - Steroid/Splint, unusual to need op
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12
Q

What are the extensor tendon zones?

A
Kleinert and Verdan 1983
Zone I → DIPJ
Zone III → PIPJ
Zone V→ MCPJ
Zone VI → dorsum
Zone VII → Extensor Retinaculum
Zone VIII → Distal forearm tendons
Added later Zone IX → Muscle bellies
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13
Q

What are the actions of extrinsic and intrinsic extensors?

A

Extrinsics → EIP EDC EDM

  • primary MCPJ extensors
  • extend IPJs when MPJ is prevented from hyperextension by intrinsics

Intrinsics → Interossei, Lumbricals, thenar and hypothenar muscles

  • flex MCPJ and extend PIPJ and DIPJ
  • contribute to IPJ extension by 3 mechanisms

Wrist extensors → ECRL, ECRB, ECU

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14
Q

Describe and draw the extensor mechanism

A

EDC
Sagittal band
Central slip
Lateral bands
Conjoined tendon
Triangular ligament
Transverse retinacular ligament - edge of flexor tendon sheath at PIP to lat band, prevents dorsal shift of lat bands, attenuation of TRL causes Swan neck, tightening of TRL and attenuation of triangular lig causes Boutonnière’s
Oblique retinacular ligament (Landsmeer) - volar lat crest of PIPJ to lateral terminal extensor tendon
Lumbricals insert into radial lateral band
Interosseii insert into both lateral bands and central slip

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15
Q

How is mallet injury classified?

A

Doyle

I - Closed ± chip fracture of P3
II - Laceration
III - Abrasion with loss of tendon substance (and skin)
IVa - Trans-epiphyseal in children
IVb - Closed + articular surface involving 20-50%
IVc - Closed + articular surface involving >50%

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16
Q

How do you treat a closed mallet injury?

A

Ishiguro Technique (doorstop technique)
o Flex DIPJ to 90deg – pulls mallet fragment distally
o Thin wire dorsally into head of P2 to skewer extensor and crowbar fragment into reduction, drill K wire in at 45deg to DP
o Straighten DIPJ then axial wire across DIPJ (pic on desktop)

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17
Q

What happens if a mallet finger is left untreated?

A

Swan neck deformity (immediate / delayed) due to division of zone 1 EDC and increased pull of central slip. Lateral bands displaced dorsally, PIPJ VP becomes lax & hyperextend
Rx - PIPJ volar plate tightening / Fowler procedure (central slip tenotomy)

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18
Q

How do you treat central slip injury?

A
Open: Repair & Kwire
Closed: gutter splint 3/52, then Capener
or SAM (Short Arc of Motion)
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19
Q

What is a Boutonnieres deformity?

A

Nalebuff Classification
o 1 – Mild – 10-15deg lag
o 2 – Mod – 15-30 deg
o 3 – Severe – 40 deg+

Pathology
o Attenuation/split in central slip
o Transverse Retinacular Ligament pulls PIPJ in to flexion

Stages
o 1 – Dynamic imbalance, passively supple
o 2 – Established – joint not involved, but not passively correctable
o 3 – Joint changes

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20
Q

How do you repair extensor tendons?

A

∘ Zone 1: running / figure of 8 suture that may incorporate the skin (dermotenodesis).

∘ Zone 2: running / figure of 8 suture reinforced with Silfverskiöld cross-stitch.

∘ Zones 3–8: MK core reinforced with Silfverskiöld.

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21
Q

What is the protocol for extensor tendon repair?

A

Zone 1+2: mallet injury.
• Splint in slight hyperextension of DIPJ for 6–8 wks.

Zone 3:
• Static PIPJ immobilisation in extension for 6 wks (gutter splint).
• DIPJ free to flex to ensure lateral bands do not shorten, leading to boutonnière.

Zones 4–7: ‘Norwich’ regime.
• Volar splint – wrist 45∘ extension, MCPJs flexed >50∘, IPJs extended.
• CAM on day 1.
∘ Wk 1-4:
Exercises: 4 per rep, 4 times/day.
(a) Combined IPJ and MCPJ extension.
(b) MCPJ extension with IPJ flexion (hook grip).
∘ Wk 5: splint at night only; MCPJ flexion commenced.
∘ Wk 6: Full power grip.

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22
Q

What is SAM?

A

SAM – Short Arc of Motion

  • For central slip injury
  • Alternative to Capener
  • Volar block: initially limiting flexion to 30deg
  • Gradually cranked out to allow more flexion
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23
Q

What is the classification for FDP ruptures?

A
  1. FDP in palm
  2. FDP at PIPJ - long (more proximal) vinculum remains intact
  3. FDP at A4 pulley - large avulsion fragment, both vinculae intact
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24
Q

What are the principles of flexor tendon repair?

A
  • Number and size of incisions in sheath kept to minimum. Step incision.
  • A2 and A4 preserved if possible.
  • Minimal handling of tendon ends.
  • Epitendinous suture of back-wall first to align tendon.
  • Core suture → McLarney’s Adelaide cruciate 4 strand (locking) repair (or Modified Kessler).
  • Reconstruct sheath if possible but don’t compromise glide.
  • In grafts and transfers use Pulvertaft weave.
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25
Q

What skin incisions do you use?

A

Bruner’s

Midaxial

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26
Q

What types of tendon repairs are there?

A
  1. Modified Kessler
  2. Strickland – 4-strand - Mod Kessler + Horizontal mattress with epitendinous
  3. Tsuge
  4. David Evans – 2 Mod Kesslers at 90° to each other
  5. Bob Savage – 6-strand
  6. Bunnell

Callan + Morrison’s method of tendon delivery – Pickford’s favourite

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27
Q

Name some different hand therapy regimes

A
Early Active Mobilisation
Accelerated Active Motion – Solomons
Allen
Belfast and Sheffield
Strickland/Cannon
Silfverskiold and May
Evans and Thompson

Early Passive Mobilisation
Kleinert
Duran and Houser
Variations on early passive mobilization

Immobilisation
Cifaldi Collins, and Schwarze

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28
Q

Summarise the different types of flexor tendon post-op protocols

A

Belfast regime - controlled active mobilisation
Kleinert - early active extension and passive flexion
Duran - early passive mobilisation

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29
Q

What is the Duran protocol (modified)?

A

Modified Duran

  • dorsal protective splint (40 to 50 degrees at the MP joints and from 20 degrees of extension to 20 degrees of flexion at the wrist, with the IP joints allowed to extend to neutral in the splint)
  • omit rubber band traction and strap IP joints in extension between exercises or at night
  • passive individual and composite flexion and extension, active composite extension exercises (manually blocking the MP in greater flexion for more complete active IP extension), and the passive flexion and extension exercises
  • In therapy only, splint is removed for careful protected tenodesis exercises (passive or assisted simultaneous wrist flexion and finger extension, alternating with simultaneous wrist extension and finger flexion)
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30
Q

What is the Kleinert regime?

A

Early active extension with passive flexion.

  • Dorsal protective splint. Finger flexion with rubber band traction. Bands attached to fingernails and the volar aspect of the splint i.e. active extension can occur against the elastic recoil of the bands.
  • Passive flexion provided by the recoil of the bands.
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31
Q

What flexor tendon protocol do you use?

A

Original articles
Small J, Brennan M, Colville J: Early active mobilization following flexor tendon repair in zone 2, J Hand Surg 1989; 14B:383-91 and

Cullen K, Tolhurst P, Lang D, Page R: Flexor tendon repair in zone 2 followed by controlled active mobilization, J Hand Surg 1989

Modified 1+2
Gratton P: Early active mobilization after flexor tendon repairs, J Hand Ther 6:285, 1993).

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32
Q

What is the Belfast regime?

A

Early Active Mobilisation (Belfast)

Early Stage (wk 1-4)

  • splint wrist neutral / 20deg flexion, MCPJ 80-90 deg flexion, IPJs extended
  • volar fingers free
  • 48hrs postop
  • full passive flexion, active flexion, and active extension
  • 1st wk - 30 degrees at PIPJ and 5 - 10 degrees at DIPJ
  • 4th wk - 80-90 degrees at PIPJ and 50-60 degrees at DIPJ
Intermediate Stage (wk 4-6)
Splint discontinued at
4wks - if tendon glide poor
5wks - most patients
3 weeks after splinting is discontinued, residual flexion contractures are treated with Capener (finger based dynamic extension) splints, and passive IPJ extension with MCPJ flexed

8wks - progressive resistive exercise and heavier hand use

12wks - full function
Scar management throughout

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33
Q

What does EAM achieve?

A
  1. Passive flexion → mobilises joints and prevents contraction
  2. Passive flexion and hold → produces isometric force on prox muscle belly. Maintains their function.
  3. Active flexion → results in tendon glide within the sheath, limits the formation of fibrous attachments, ↑ rate of intrinsic healing. ↑ Strength of repair.
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34
Q

What are the specific complications of flexor tendon repair?

A

Rupture: 5%
Adhesions
Joint contractures 17%

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35
Q

What is tenolysis?

A
  • Release of adhesions to restore Active ROM
  • Waste of time in under 12s
  • Neuroleptanalgesia - active pt participation intraop
  • Not less than 3/12 post-op
  • Best at 6-12 months
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36
Q

Name some tendon rupture sites

A
  1. Distal Ulna - Caput ulnae - EDM, EDC Little (Vaughn Jackson)
  2. Scaphotrapezial spur - Mannerfelts critical corner - FPL, FDP Index
  3. Hook of Hamate - FCR
  4. Lister’s tubercle - EPL
  5. Carpal Tunnel - most common site for flexor rupture
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37
Q

What are the indications for 2 stage flexor tendon repair?

A

Indications

  • Primary repair not possible (delayed presentation, attrition rupture, crush injury / segmental loss, infections).
  • Failed primary repair
  • ?Loss of all pulleys

Wound factors

  • Good soft tissue cover, soft and supple scars.
  • Joints should be mobile, PROM>AROM.
  • Stable joints.
  • Sensate.

Patient factors

  • realistic expectations (including recurrence)
  • patient compliance
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38
Q

What are the requirements of a secondary tendon recon?

A
  1. Full passive ROM
  2. Sensate
  3. Access to good Hand Therapy
  4. Motivation
    Law of diminishing returns with each subsequent operation
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39
Q

How do you do a 2 stage tendon graft reconstruction?

A

1st stage
∘ Passive silicone tendon rod (Hunter rod) is placed in flexor sheath.
∘ Pulleys and skin/joint contractures are reconstructed.

Second stage at 3–6 months
∘ Tendon graft is placed into pseudosheath formed around rod (PL, plantaris, long toe extensors/flexors, EIP, EDM or FDS from injured finger).
∘ Distally: Mitek / Sood Elliot technique.
∘ Proximally: Pulvertaft weave: tensioned to recreate natural cascade of fingers (tenodesis test).

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40
Q

How do you do a 2 stage recon?

A
  • Need 4mm of A4, 8mm of A2
  • Reconstruct with extensor retinaculum – go around extensor for A4, deep to it for A2 – otherwise lateral bands get tightened
  • Nagor silicone rods
  • 2nd stage only once soft, supple, with full passive ROM
  • At least 3 passes with Pulvertaft weave – each gives about 25% strength
  • Palmaris longus
  • Elliott/Sood 3-needle technique for distal attachment
  • To get tension just right: stretch out to full opposed range of movement and put graft/transfer in as tight as possible
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41
Q

What are the different ways of insetting the tendon graft?

A

Distally 1st
FDP stump (split in 1/2 n bury graft in middle)
Bone (Bunnell)
Nailbed (Pulvertaft)

Proximal
Pulvertaft weave
Bunnell cross-cross
Kessler

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42
Q

What are the potential donor sites for tendon grafts?

A
PL 13cm
Plantaris 31cm (anterior and medial to Achilles)
Toe extensors 30cm
FDS, EI, EDM
Tendon allografts
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43
Q

How many % of patients do not have PL or FDS little?

A

Absent Palmaris – 15%

Absent FDS to little – 15%

44
Q

What are the complications specific to 2nd stage recon?

A

Intraop
NV injury

Early
Synovitis, infection
Buckling of implant
Implant extrusion, migration
Pulley breakdown
Skin flap necrosis

Late
Chronic flexion deformity
CRPS type 1

Tendon adhesion
Rupture of graft
Quadriga - tendon too short / tethered - the other FDPs cannot shorten sufficiently to achieve full flexion
Lumbrical plus - tendon too long

45
Q

What are the alternatives to FDP repair?

In zone 2 + 3

A
  1. DO NOTHING!!
  2. Tenolysis
  3. Tendon transfer
  4. Primary tendon graft
    ∘ Scar within sheath, or sheath incompetence, requires two-stage reconstruction.
  5. Two-stage tendon graft

Zone 2
- DIPJ arthrodesis

Zone 3

  • Interpositional graft
  • Tenodese to adjacent FDP
  • FDS transfer
46
Q

What are the pre-op requirements and intra-op pearls for tendon transfers?

A

Preop

  • all wounds healed
  • full passive ROM
  • sensate

Intraop

  • operate in reverse order - prepare recipient site, tunnel before raising muscle
  • avoid interference with other structures e.g. compress nerve
  • apply correct tension (EIP→EPL alway loosen, Opponensplasties always tighten so start loose)
  • tendon transfers must not pass under areas of scar tissue or skin grafts
47
Q

How do you reconstruct the flexor tendon pulleys?

A
  • Indicated when pulley mechanism is insufficient to prevent bowstringing that limits active joint flexion.
  • A2 and A4 pulleys minimum.
• Materials:
∘ Free tendon graft.
∘ Segment of wrist extensor retinaculum.
∘ Fascia lata.
∘ Slip of FDS.
∘ Dacron, silicone, PTFE.

• Method
∘ Wrap around PP + MP in a double loop.
∘ Weaving through remnants of pulleys in a zigzag fashion.
∘ Suturing into holes drilled into phalanx.
∘ Bone anchors.

48
Q

What are the fundamental principles of fracture treatment?

A
  1. Reduction
  2. Immobilisation (+/- Fixation)
  3. Rehabilitation
49
Q

In hand fractures, what other factors must be considered?

A
  1. Life-threatening injuries.
  2. Open wound?
  3. Associated NV injury?
  4. Associated tendon / ligament injury?
50
Q

How is fracture reduction achieved?

A

• By closed manipulation
• By mechanical traction
• By open surgery.
– Minimal displacement or displacement of no functional consequence do not need reduction.
– Translation is generally better tolerated than angulation or rotation.

51
Q

What is the purpose of immobilisation and how can it be achieved?

A
  • To prevent displacement of fragments.
  • To prevent movement that might interfere with bone union.
  • To relieve pain.
– Immobilisation can be achieved by:
• External splint, e.g. POP or Zimmer splint
• Continuous traction
• External fixation
• Internal fixation.
52
Q

What are the indications for fixation and methods of internal fixation?

A
  • Irreducible fractures, particularly if malrotated
  • Intra-articular fractures
  • Subcapital fractures of phalanges
  • Open fractures
  • Segmental bone loss
  • Polytrauma with hand fractures
  • Multiple hand or wrist fractures
  • Fractures with soft tissue injury (vessel, tendon, nerve, skin).
Methods of internal fixation:
• K wires
• Interosseous wires
• Intramedullary devices
• Screws
• Plates.
53
Q

What are the aims of hand therapy?

A

Aims of rehabilitation:
1. Preserve hand function during fracture union.
2. Return hand function to normal once fracture has united.
– Immobilise usually no > 3–4 weeks.

54
Q

What are the complications of hand fractures?

A

Early
• NV and other soft tissue injury.
• Compartment syndrome.

Late
• Delayed union
• Non-union
• Malunion
• Joint stiffness and contractures
• AVN
• CRPS
• Osteomyelitis
• Growth disturbance or deformity
• OA
55
Q

When planning the incision for access to an ORIF, what should be considered?

A

Aims

  • provide adequate exposure
  • preserve skin’s blood and nerve supply
  • extendible if necessary
  • avoid scar contracture.

Dorsal, lateral or volar approaches.

56
Q

What are the different open surgical approaches to hand fractures?

A

Dorsal

  • skin: H,Y,L over DIPJ, longitudinal over PIPJ + MCPJ
  • zone III EDC: midline split or Chamay approach: V shaped incision through extensor based distally.

Lateral

  • Midaxial incision (dorsal extremes of IPJ flexion creases)
  • Midlateral incision (more volar)

Volar
- very rarely used, e.g. VP avulsion fracture, MCPJ collateral ligament avulsion (lig travels from MC to volar base of PP).

57
Q

How are MC fractures managed?

A
  • Check for rotation: make a fist, DIPJ extended (fingers should point to scaphoid tubercle).
  • 5th MC neck fractures: well tolerated due to mobility of CMCJ, treatment not required unless rotated / angulated >70∘

• Stable fracture: splint for 3–4 weeks.
∘ Buddy strapping
∘ Bedford gaiter
∘ POP, aiming for 3-point fixation, e.g. ulnar gutter, Barton short hand cast, clam-digger cast.

• Open fracture: operative.

• Fixation can be achieved by:
∘ K wires: transverse, longitudinal, ‘bouquet’ wiring.
∘ Lag screws
∘ Plates – low profile 
∘ Interosseous wires
∘ External fixation.
58
Q

How are phalangeal fractures managed?

A

DP: splint / Kwire/ Ishiguro ‘doorstop’ technique for bony mallets.

MP/PP: cross Kwire, lateral plate.

59
Q

How are intra-articular fractures managed?

A

Aim: to restore joint congruity and:

  1. Prevent deformity
  2. Prevent OA
  3. Restore early movement.

Unicondylar fractures
- percutaneous K wire, ORIF + lag screws.

Pilon fractures (comminuted)

  • e.g. axial loading of PIPJ → MP base fracture.
  • Suzuki frame (K wires and rubber bands dynamic external fixator), Hynes–Giddins device (K wires only).

Some fractures may require ORIF and bone graft.

60
Q

How are DIPJ and PIPJ dislocations managed?

A

DIPJ
• Dislocations and subluxations rare
• May be associated with tendon avulsions

PIPJ
• Volar plate injuries (Eaton):
∘ Type I (hyperextension)
– Avulsion of VP without fracture; collateral ligaments split.
∘ Type II (dorsal dislocation)
– Complete dorsal dislocation; VP avulsion without fracture.
∘ Type III (fracture subluxation)
– Fracture subluxation with a palmar fragment.

Treatment

  • reduction, EAM with dorsal blocking splint or
  • dynamic ex-fix, ORIF of palmar fragment
61
Q

Does PIPJ usually dislocate dorsally or volarly?

A
usu dorsal (VP avulsion)
volar rare (central slip avulsion)
62
Q

How are MCPJ and CMCJ subluxations / dislocations managed?

A

VP may rupture and flip into joint
Rx: open reduction and repair.

CMCJ

  • thumb → ring fingers: rare (high energy trauma).
  • 5th CMCJ subluxation: may be associated with MC base fracture (reversed Bennett’s) or dorsal avulsion from hamate.

Treatment:
∘ Closed reduction and splint
∘ Closed reduction and K wire into adjacent MC or carpus
∘ ORIF.

Inadequate reduction → poor grip.

63
Q

What is a Bennett’s fracture and how is it managed?

A
  • Fracture–subluxation of 1st CMCJ.
  • Mechanism of injury = axial loading of flexed CMCJ →
  • palmar oblique (‘beak’) ligament holds fragment in anatomical position
  • Remainder of MC is adducted and supinated by adductor pollicis, and pulled proximally by APL.

• Reduced by:

  1. Longitudinal traction
  2. Pronation of metacarpal
  3. Pressure at base of MC.

• Immobilisation by
1. Thumb spica or

  1. Percutaneous K wires:
    a) Transfixion of MC base to trapezium.
    b) Transfixion of 1st MC base to 2nd MC.
    c) a+b.
  2. ORIF
64
Q

What is a Rolando fracture and how is it managed?

A
  • 3-part intra-articular fracture of base of 1st MC.
  • Non-operative: usually inadequate
  • ORIF with T-plate (dorsal or radiopalmar approach).
  • External fixation
65
Q

What are

  • Bennett’s fracture
  • Rolando fracture
  • Reverse Bennett’s fracture
A

Bennett’s Fracture

  • 2 part # dislocation of 1st MC on trapezium
  • Forces – APL and Adductor Pollicis
  • Ulnar-Volar Fragment
  • MC base subluxed dorsally, radially and proximally due to pull of APL

Rolando

  • ≥3 part # dislocation of 1st MC on trapezium
  • Comminuted T or Y-shaped fracture at base of 1st MC

Reverse Bennett’s

  • Base of 5th Metacarpal
  • Forces – ECU and Hypothenar muscles
66
Q

How is an MCPJ UCL avulsion managed?

A
  • Skier’s thumb: acute
  • Gamekeeper’s thumb: chronic attenuation of UCL.

• Stener lesion: avulsed ligament displaced dorsal to adductor aponeurosis. Spontaneous bony or ligamentous healing not possible due to interposing adductor aponeurosis.
∘ Radial stress test with MCPJ in 30∘ of flexion.
• Treatment: Mitek bone anchor, immobilisation for 4wks.

67
Q

How do you treat thumb MCPJ UCL ruptures?

A

Acute rupture = skier’s thumb
Chronic repeated hyperabduction = gamekeeper’s thumb

Stener lesion (80% of all ruptures)
- torn ligament / bony fragment is avulsed from its distal insertion and sits superficial to adductor aponeurosis

Radial stress test
- accessory collateral ligament - test with thumb extended
proper collateral ligament - test with thumb flexed

> 40 deg instability or >15deg difference to contralateral normal side = tear

Partial tear - thumb spica 4wks
Complete tear - surgery

68
Q

Describe the procedure for Stener lesion repair

A

Lazy-S incision - ulnar midlateral to dorsum of MP joint.
Longitudinal incision in add aponeurosis, 3mm volar to EPL tendon.
Remove loose bone if found.
- Middle 2/3 tears - suture
- Distal (common) or proximal (rare) - pull through or Mini-Mitek.
- Large Artic Fragments - ORIF.
Repair add aponeurosis

Green says consider following 4 steps before closure:

  1. Suture distal volar repaired lig to volar plate for added stability.
  2. Repair dorsal ulnar capsule.
  3. Test repair with radial stress.
  4. Consider over ulnar deviation of joint in mild flexion with K-wire.
69
Q

What is unique about paediatric hand fractures?

A

• Most can be managed non-operatively.
• Physis: deformity can be remodelled with growth.
• Greatest remodelling potential is seen:
∘ When angulation occurs in plane of adjacent joint motion.
∘ When fracture is in close proximity to physis.
∘ When fracture is in a young child with many years of remaining growth.
• Rotational and angular deformity has little remodelling potential.
• Physis should be respected during surgery.

70
Q

Please classify Salter Harris Fractures

A

Salter Harris Classification of Paediatric Epiphyseal Fractures (1963)

I – S = SLIP (separated or straight across). Fracture through the physis
II – A = ABOVE. The fracture lies above the physis, or Away from the joint.
III – L = LOWER. The fracture is below the physis in the epiphysis.
IV – T = THROUGH. The fracture is through the metaphysis, physis, and epiphysis.
V – R = RAMMED (crushed). The physis has been crushed

71
Q

What is Salter Harris Classification?

physis = growth plate

A

Type I – 6%
A transverse fracture through the physis

Type II – 75%
- through the physis and the metaphysis, sparing the epiphysis

Type III – 8%
- through physis and epiphysis, sparing metaphysis

Type IV – 10%
- fracture through all three elements: growth plate, metaphysis, and epiphysis

Type V – 1%
- compression fracture of the growth plate

72
Q

What is a Seymour fracture?

A
  • Looks like a mallet deformity, but is transepiphyseal rather than transarticular.
  • Extensor tendon pulls on proximal fragment and FDP on distal fragment → distraction and volar displacement of fracture.
  • Usually associated with nail bed lac (open fracture).
  • Requires debridement, reduction, nail bed repair and replacement of nailplate as a splint +/- axial transarticular K wire if unstable.
73
Q

How are phalangeal neck fractures managed in children?

A

• Typically occur following trapping a finger in a door.
• Distal fragment is angulated dorsally into extension
∘ Results in incomplete flexion at PIPJ.
• Minimally displaced fractures: splint + weekly X-rays.
• Displaced fractures: closed reduction and K wire.

74
Q

What is the definition of trigger finger?

What is the pathogenesis?

A

Stenosing tenosynovitis

Acquired condition
Flexor tendon sheath thickens and narrows canal so flexor tendon cannot glide freely through it

Pathology
Fibrocartilagenous metaplasia and hypertrophy of A1 pulley
Reactive swelling of tendon

75
Q

What symptoms do patients present with?

A

pain in palm, intermittent snapping/triggering, locking (in flexion or extension)
Typically worse in the morning

76
Q

What are the differential diagnoses of trigger finger?

A

Dupuytren’s contracture, post-traumatic joint contracture or locking of the metacarpophalangeal joint (rare).

77
Q

What is the epidemiology of trigger finger?

A

Lifetime risk 2%
Can occur in children (thumb > fingers) and adults
Risk factors - diabetes, thyroid disease, RA< renal disease

78
Q

How can you classify trigger finger?

A

Wolfe 2005
Mild (“pre-triggering”) - ANALGESIA
- History of pain, catching or “click”
- Tender A1 pulley; but fully mobile finger

o Moderate - STEROID INJECTION
- Triggering with:
A - Difficulty actively extending finger
B - Need for passive finger extension. Loss of complete active flexion

o Severe - SURGICAL RELEASE
- Fixed contracture

79
Q

How do you perform steroid injection for trigger finger and how effective is it?

A
  • triamcinolone or dexamethasone (water soluble - decreased risk of tendon rupture)
    +/- LA, insulin needle, 1ml, inject into palm at level of A1 pulley
  • 1st injection 49-78% effective
  • 2nd injection 50%
80
Q

When is surgical release indicated?

A
  1. after failed conservative treatment
  2. recurrent triggering after 1-2 injections of steroid
  3. severe symptoms at presentation
  4. in people who are unlikely to benefit from steroid injections (e.g. diabetic with multiple digits affected and severe symptoms)
81
Q

How do you manage paediatric trigger thumb?

A

Acquired
presents ~6/12 - 2yrs
IPJ locked in flexion
palpable ‘Notta’s node’ at MCPJ level

Conservative - passive stretching (splinting)
steroid inj not advocated
36-78% of cases will resolve spontaneously
Different authors advocate surgery if failed conservative management age 3-5

82
Q

How do you perform a trigger finger release?

What is important in releasing the thumb?

A

Management
Steroid lnjection - in sheath/subcut/both

Littler Ratio - Double the distance of central P3 whorl to PIPJ crease = A1 pulley location

Thumb - release A1 on radial side to avoid oblique pulley (between A1 and A2) which runs ulnar to radial

83
Q

When is this op contraindicated?

A

Rheum Arthritis - check for FDP entrapment by inflammation at level of FDS decussation - A1 pulley is one preventer of ulnar drift so nice to preserve if possible - tenosynovectomy and nodule excision instead.

84
Q

How do you perform a trigger finger release?

What is important in releasing the thumb?

A

Management
Steroid lnjection - in sheath/subcut/both

Littler Ratio - Double the distance of central P3 whorl to PIPJ crease = A1 pulley location

Thumb - release A1 on radial side to avoid oblique pulley (between A1 and A2) which runs ulnar to radial

85
Q

When is this op contraindicated?

A

Rheum Arthritis - check for FDP entrapment by inflammation at level of FDS decussation - A1 pulley is one preventer of ulnar drift so nice to preserve if possible - tenosynovectomy and nodule excision instead.

86
Q

What is Stanton-Hicks definition of CRPS?

A

CRPS (1995) = a variety of painful conditions following injury … pain exceeding both in magnitude and duration of the expected clinical course of the inciting event

RSD and causalgia no longer used

87
Q

How is CRPS classified?

Type 1

A
  • Most Common
  • Not involving direct nerve injury
  • 60-70% - Simple trauma
  • Female : Male 4:1, 3rd-4th decade
  • Younger get over it, older don’t
  • Allodynia – Pain with normal stimulus
  • Hyperpathia – additive pain with repeated stimuli
  • Hyperalgesia – exaggerated pain response
  • Pain in uninvolved joints
  • Autonomic instability – increased temperature, sweating, swelling (sweating reduces later)
  • Trophic Changes – Nails, Hair, Skin, Osteoporosis
  • Weakness – small movements, tremor
88
Q

CRPS Type 2

A
  • Identifiable primary nerve insult

- causalgia + vasomotor, sudomotor and trophic changes

89
Q

What is the proposed pathogenesis of CRPS?

A

sensitisation of CNS to a painful stimulus following peripheral nerve injury by cytokine mediated feedback to hypothalamus

90
Q

What patients are susceptible?

A
  • Distal Rad/Uln # - up to 21%
  • Palmar br. median n.
  • Sup br. radial n.
  • Tight dressings/POP
  • Can be isolated to a single digit
91
Q

What are the symptoms?

A

Symptoms

  • pain
  • allodynia: pain due to stimulus which doesnt usually provoke pain
  • hyperalgesia: increased pain to normally painful stimulus
  • hyperaesthesia: increased sensitivity to a stimulus
  • hyperpathia: excessive perception of a painful stimulus
  • dysaesthesia: abnormally perceived unpleasant sensation, spontaneous or provoked
  • causalgia: burning pain, allodynia and hyperpathia after traumatic nerve injury
92
Q

What are the signs?

A

Signs

  • stiffness
  • swelling
  • discolouration (dermographia - skin changes with light object dragged across skin)
  • skin changes: inc hair, shiny skin, hyperhidrosis, osteoporosis
93
Q

What investigations may be useful?

A
  • thermography: temp difference 0.6 degrees
  • Abnormal bone scan in 60%
  • Reduced blood flow at later stage (Increased initially)
  • Phentolamine infusion test - blinded with saline - distinguishes sympathetic dependent from sympathetic independent CRPS.
94
Q

How is it diagnosed?

A
pain out of proportion
abnormal sympathetic function
swelling
mvmt disorder
changes in tissue growth
95
Q

What are the stages of CRPS?

A

Acute - Traumatic - 1-3mths
Subacute - Dystrophic - 3-12mths
Chronic - Atrophic - years (Sudeck’s atrophy)

96
Q

What is the mainstay of treatment?

A

Early diagnosis is key

  • Pain
  • Function – Don’t rush – gentle passive initially, building up slowly
  • Psychiatric Support
97
Q

What treatments are available?

A
  • Treat primary insult
  • Physio
  • TENS
  • Free Radical Scavengers (Vitamin C 500mg tds for 50 days, DMSO (Dimethylsulphoxide), NAC – N-Acetylcysteine)
  • Calcitonin
  • Steroids – for acute inflammatory phase
  • Simple Analgesia
  • Opiates
  • Antineuropathics
  • NMDA Antagonists – e.g. ketamine infusions
  • Sympathectomy
  • Blocks (guanethidine)
  • Epidurals
  • Implantable Spinal Cord Stimulator – good for function as well as pain
  • HBO
98
Q

What is Elson’s test?

A

It is a test for central slip continuity.

Place patients fingers over edge of table and flex fingers at PIPJs
Ask patient to extend at PIPJ against resistance.
Patient will be unable to do this if central slip is ruptured.

99
Q
Explain 
Quadriga effect
Lumbrical plus
Bunnell Littler test
Bouvier test
A
  1. Quadriga phenomenon (a Quadriga = an ancient Greek four horse chariot)
    o Condition in which the flexor tendon excursion in unaffected finger is reduced due to altered FDP excursion in the adjacent injured finger (by stiffness, injury or adhesion) (Verdan 1960).
    o When testing for FDS, the FDP is defunctioned because the FDP tendons are combined, while the FDS muscles are separate in the forearm.
    o Following repair or reconstruction of an FDP tendon the tension must be identical to the other FDPs, since the excursion of the combined tendons is equal to the shortest tendon.
    o Suturing extensor tendon to flexor tendon following amputation will result in Quadriga effect.
  2. Lumbrical plus - opposite to Quadriga
    If FDP is allowed to retract after e.g. DP amputation, during attempted flexion, extension of PIPJ results because retracted FDP causes increase in lumbrical pull. To avoid this, secure FDP stump to A4 pulley or partial / complete excision of lumbrical.
  3. Bunnel-Littler Test For intrinsic tightness.
    1) With the MCPJ in extension the intrinsics are put on a stretch. Try to flex the PIPJ with MCPJ in extension. If it doesn’t flex = tight intrinsics or joint capsule contracture.
    2) With MCPJ in flexion the intrinsics are relaxed. Thus if unable to flex PIPJ= tight capsule. NB- prior to test check that passive motion of PIPJ is possible (i.e. normal PIPJ)
    Tight intrinsics occur in: ‘Intrinsic Plus’ hands due to ischaemia or fibrosis of intrinsics or RA.
  4. Bouvier’s Test (BOUtonniere’s deformity)
    • To determine if PIPJ capsule & ext. mech. are working normally.
    • If PIPJ capsule & ext. mech. are functioning normally then blocking MCPJ hyperextension allows IPJ extension.
    • Positive test occurs as a result of: attenuation of central slip, adherent central slip at PIPJ or volar subluxation of lateral bands.
100
Q

What are the causes of quadriga effect and a lumbrical plus digit?

A

Quadriga:
1. Adhesion / scarring of FDP tendons
2. Over tightening / distal advancement of FDP after rupture and repair
3. After amputation, if FDP is sutured to EDC
Result: weak grasp on remaining fingers, and cannot make full fist because if 1 tendon is shortened the others cannot flex fully.

Lumbrical plus:
e.g. if FDP allowed to retract after amputation of digit
A lax FDP results in tension in the lumbricals.
Result: when patient makes a fist, the affected finger extends at the PIPJ due to lumbrical pull.

101
Q

What is Bouvier’s test?

A

To test intrinsic or extrinsic finger extension?

Intrinsic: extension of PIPJ with MCPJ flexed
Extrinsic: extension ofIPJ with MCPJ extended.

102
Q

What are the patient selection and recipient site factors for tendon transfers?

A

Patient Selection

  • Well motivated
  • Able to understand nature and limitations of surgery
  • Able to co-operate with post op physio

Recipient site factors

  • Good soft tissue cover
  • A stable skeleton
  • Full range of passive movement
  • Normal sensation
103
Q

What are the donor muscle factors in choosing muscles for tendon transfer?

A

APOSLE
1. Amplitude of motion→ similar excursion
o Wrist flex and ext = 3 cm
o Finger extensors = 5cm
o Finger flex = 7cm
Effective amplitude of donors can be ↑ by use of tenodesis, freeing fascial attachments of the donor muscle unit.

  1. Power and control → similar pull
    o Lose at lease 1 grade after transfer
    o BR does not adapt well
  2. One tendon – one function
    o effectiveness if try to do 2 functions
  3. Synergistic Action
    o Muscles which normally act together to produce a composite movement should be used to replace each other whenever possible.
    o Finger flex assoc with synergistic wrist extension
    o Finger ext assox with wrist flex
    o A transfer of a wrist flexor to a finger extensor = synergistic → better function
  4. Line of Pull
    o Best if direct from origin to insertion
    o Deviations around pulleys ↓ effectiveness
  5. Expendability
    o Only transfer expendable tendons
    o Important that one wrist flexor remains intact when producing wrist and finger extension in radial palsy.
104
Q

What is your evidence based approach to flexor tendon repair?

(An evidence based approach to flexor tendon laceration repair. Lalonde (Canada) PRS 2011.

A
  • No need to repair if tendon is >40% intact
  • Core: 4 strand is better than 2 (less gapping)
  • Locking is better than grasping
  • 3/0 stronger than 4/0 by 50%
  • Longer core suture purchase length is superior (1cm vs 0.4cm). Optimum = 0.7-1.0cm.
  • Absorbable comparable to non-absorbable sutures (for 4 strand core).
    (Caulfield et al, 2008 JHS(E))
  • Mitek superior to button technique for FDP avulsions (patient satisfaction and lower morbidity).
  • Flexor sheath repair: results contradictory and mainly animal-based. 1 study on children showed better mvmt with vein graft sheath repair.
105
Q

What evidence is there to guide your post op rehab regime?

A

Cochrane review (2004) showed good results and trend toward EAM protocols vs immobilisation.

Kitis 2009 (Scand) compared Kleinert (controlled active) vs Duran (controlled passive): showed CAM had better TAM and DASH scores.

106
Q

What evidence is there on post-operative outcomes?

(a) Nerve repair mobilisation vs immobilisation.
(b) Rupture rate

A

(a) Digital nerve repairs: mobilise from day 4 vs immobilise for 3wks: no difference in outcome. (2005).

(b) Zone 1+2 EAM (233pts, prospective: Elliott JHS 1994).
- FDP: 5.8%
- FPL: 16.6%

Rupture rate review paper (Tang 2005 Hand Clinics).

  • FDP = 4-10%
  • FPL = 3-17%

Tenolysis rate ~33% (Swiss)

107
Q

A 25-year-old, working, single mother of two
is referred to you 2 weeks after she has lacerated the profundus tendon and digital nerves in zone 2. How would you manage her based on current best evidence?

A

She would likely be best treated with repair of the nerves (Level III Evidence) and the profundus tendon.
She should be treated with an early protected active movement protocol (Level II Evidence).
She should be allowed to return to work at 8 weeks after repair (Level II Evidence).

108
Q

Draw an Adelaide locked 4 strand cruciate core suture.

A
  • McLarney (JHS (Am) 1999) 1st described non-locking version of this core suture.
  • Showed it was superior to MKS 2 strand, Strickland and modified Savage 4 strand repairs.
  • It required twice the force (44N) to produce 2mm gap compared with the others.