Chapter 11: Aphasia and Other Disorders of Higher Cortical Function Flashcards

1
Q

Aphasia is…

A

Any acquired abnormality of language.

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2
Q

Don’t confuse aphasia for…

A

Anything that may hinder the ability to speak or write like dysarthria, stuttering, decreased consciousness.

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3
Q

Which feature is present in all aphasias…

A

Anomia and problems with writing

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4
Q

Aphasia and writing

A

No aphasic patient writes normally. Asking a patient to write a paragraph can easily screen for aphasia.

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5
Q

Broca’s Aphasia

A
Nonfluent, hesitant, telegraphic speech output, cannot repeat, but relatively preserved comprehension (cannot understand complex grammar). 
Phonemic substitutions (based on sound)
They are frustrated.
Singing is not impaired
Weakness in face and arm
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6
Q

Telegraphic speech

A

Saying only the key nouns and verbs.

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7
Q

Paraphasias

A

Word Substitution errors

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8
Q

Wernicke’s aphasia

A

Fluent word salad; semantic paraphasias; neologisms; they would sound normal if you didn’t speak their language

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9
Q

Conduction apahasia

A

Arcuate fasciculus; many paraphasic errors; or lesions involving temporal and parietal lobes but sparing Wernicke’s area

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10
Q

Transcortical motor aphasia

A

Similar to broca’s, but less severe (can repeat)

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11
Q

Transcortical sensory aphasia

A

Similar to Wernicke’s, but less severe (can repeat)

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12
Q

Global aphasia

A

Think ICA stroke, everything in language is knocked out

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13
Q

Subcortical aphasias

A

Dominant hemispheric deep structures: basal ganglia and thalamus.
Accompanied by hypophonia of the voice.
Can resemble Broca’s or Wernicke’s.

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14
Q

Pure Alexia (alexia without agraphia) cause

A

Dominant occipital lobe and splenium of the corpus callosum.

Fibers connecting visual cortex on either side to Wernicke’s area in the dominant hemisphere is interrupted, preventing input of langugae through visual means.

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15
Q

Commonly found with pure alexia

A

Contralateral homonymous hemianopia

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16
Q

Apraxia is

A

The inability to perform a learned motor task despite preservation of the necessary basic motor, sensory, and cognitive capacities.
Dominant hemisphere

17
Q

How to test for apraxia

A
  1. Ask patients to pretend they are performing an action
  2. To mimic the examiner performing an action
  3. To use actual objects in performing an action
18
Q

Frontal lesions in apraxia

A

Patient can identify task done correctly but can’t do it themselves

19
Q

Parietal lesions in apraxia

A

Patient cannot recognize the task done correctly

20
Q

Agnosia is

A

The inability to recognize objects despite preservation of the basic sensory modalities being used

21
Q

Visual agnosia

A

Can’t identify things with vision, but feeling it would help identify it.

Lesion of visual association area: Occipitotemporal region anterior to the primary visual cortex.

22
Q

Cause of agnosia

A

Lesions in the sensory association areas of the brain

23
Q

Prosopagnosia

A

Inability to recognize faces.

Lesion of visual association area: Occipitotemporal region anterior to the primary visual cortex.

24
Q

Gerstmann’s syndrome 4 elements

A
  1. Agraphia
  2. Acalculia
  3. Right-left confusion
  4. Finger agnosia
25
Q

Gerstmann’s cause

A

Lesion in the inferior parietal lobule of the dominant hemisphere (ANGULAR GYRUS)

26
Q

Which hemisphere gets neglect

A

The nondominant hemisphere

27
Q

Definition of neglect

A

Directed inattention, or relative lack of attention to one hemispace

28
Q

Most sensitive sign of Neglect

A

Extinction to double simultaneous stimulation. Touching one arm, then the other, then both and see if they miss any side.

29
Q

Neglect cause

A

Lesion in right frontal or parietal lobe, ultimately may get motor or sensory neglect

30
Q

Anosognosia definition

A

Being unaware of your deficit