Ch.35 PAIN Flashcards

1
Q

Awareness and information of the body’s deep and superficial parts.
Relays information regarding touch, temperature, pain, and body position.
The cell body of the dorsal root ganglion neuron, its peripheral branch, and its central axon form a sensory unit.

A

Somatosensory system

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

3 types of fibers

A

Type A, B, and C

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Type A fiber

A

myelinated, fastest rate of conduction, convey pressure, touch, cold sensation, and heat information. ACUTE PAIN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Type B fiber

A

myelinated, transmit info from cutaneous & subcutaneous receptors

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Type C fiber

A

unmyelinated, slowest rate of conduction, convey warm and hot sensation, mechanical and chemical as well as heat and cold induced pain. CHRONIC PAIN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

3 levels of Neurons:

A

FIRST ORDER- detects sensation ( periphery to CNS)
SECOND ORDER- communicates with various reflex network ( sensory pathway- brain)
THIRD ORDER -Relay info from brain to cerebral cortex/ feeling of pain (process info)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

travel up spinal nerves to the spinal cord

A

sensory impulses

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

a multidimensional experience. unpleasant sensory/ emotional experience associated with actual or potential tissue damage. ALWAYS SUBJECTIVE, UNPLEASANT, AND EMOTIONAL EXPERIENCE

A

PAIN

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Both are parallel pathways that carry info from spinal cord to the thalamic level of sensation

A

Discriminaticce and Anterolateral pathway

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

used for the rapid transmission of sensory information such as discriminative touch. composed of rapid, large, myelinated fibers

A

discriminative pathway

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

provides transmission of sensory information such as pain, thermal sensations, crude touch and pressure that does not require discrete localization of signal source

A

Anterolateral pathway

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Affects arousal, mood, attention
Activates the reticular activating system (controls sleep/wake cycles
slower conducting, mostly made- up of type C fibers
associated with chronic pain as well as visceral pain
concerned with diffuse and dull aching pain

A

Paleospinothalmic tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Allows localization, identification of pain
rapid transmission from sensory to thalamus, made up of mostly type A fibers
associated with sharp fast reacting

A

Neospinothalmic tract

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Theory that regards pain as a separate sensory modality. Evoke the activity by specific receptors that transmit info by special nerve endings. Pain centers are regions in the forebrain

A

Specificity theory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Theory where pain signals are sent to the brain only when stimuli come together only when there is a specific pattern being created

A

Pattern Theory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Theory of the modification of the specificity theory, presence of neurogating mechanism in spinal cord account for pain or any author sensory modalities. Excess of impulses which is then sent to the brain

A

Gate control theory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Theory that proposes the brain contains a widely distributed neural network multiple sensory, limbic, thalamic components. Consist of two loops between the thalamic and cortex. Create Pain Sensation when nothing is present

A

neuromatrix theory

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

PAIN MECHANISMS:

initiated by pain receptors (nociceptors) that are activated by harm to peripheral tissue, “ pain sense”

A

nociceptive pain

19
Q

PAIN MECHANISMS:

arises from distinct injury or dysfunction of the sensory axons or peripheral or sensory central nerves

A

neuropathic pain

20
Q

PAIN MECHANISMS:

point at which the person perceives that it is painful

A

pain threshold

21
Q

PAIN MECHANISMS:

total pain experience, maximum intensity that a person is willing to endure the pain.

A

pain tolerance (everyone has different tolerances)

22
Q

Cutaneous Pain

A

superficial, localized, sharp, burning

23
Q

Deep Somatic Pain

A

muscles, tendons, joints, radiate, swelling

24
Q

Visceral Pain

A

organs, common pain produced by disease

25
Q

Referred Pain

A

pain that originates in the abdomen, diffuse, poorly localized

26
Q

Acute Pain

A

Short duration
Remits when underlying process is resolved
Elicited by surgery or trauma to body tissue
Should be aggressively managed
Give preemptive and multimodal therapy
Give pain medication before the pain becomes severe
Management helps with mobility and respiratory movements

27
Q

Chronic Pain

A

Pain that last longer than might be reasonably expected
Highly variable
Nociceptors are persistently stimulated
Serves no useful purpose
Causes psychological, physiological, familial, and economical stress
Management is complex and depends on the cause of the pain, the underlying health problem, and life expectancy of the individual
It is best managed by a multidisciplinary team

28
Q

Altered pain sensitivity:

continued stimulation causes pain (hypersensitivity)

A

hyperpathia

29
Q

Altered pain sensitivity:

spontaneous, unpleasant sensations (foot asleep)

A

paresthesias

30
Q

Altered pain sensitivity:

distortions of somesthetic sensation (diabetic neuropathy)

A

dysesthesia

31
Q

Altered pain sensitivity:

reduced pain sensation

A

hypalgesia

32
Q

Altered pain sensitivity:

absence of pain

A

analgesia

33
Q

Altered pain sensitivity:

pain after no noxious stimulus (fibromyalgia)

A

allodynia

34
Q

neurogenic stimulation

A
Tissue damage
inflammatory mediators
stimulate nociceptors
impulses run up C fibers
dorsal nerve root reflex
inflammatory mediators move back down and are released into tissues
35
Q

Causes of neuropathic pain

A
Pressure on nerve
Physical injury to neuron
Chemical injury to neuron
Infection of neuron
Ischemia
Inflammation
36
Q

Neuropathic pain syndromes include

A

Trigeminal neuralgia (tic douloureux)
Postherpetic neuralgia
Phantom limb pain

37
Q

Pain in children:

A

Pain begins in the neonatal period
Able to accurately and reliably report pain at 3 years of age
Children remember pain
Careful assessment is key
Use numeric scales as well as picture scales
Pharmacologic and nonpharmacologic methods

38
Q

Pain in older adults:

A

Pain increases in older adults
Unrelieved pain can led to immobility and falls
Impaired appetite, sleep disturbances, and cognitive dysfunction
Decreases quality of life
If patient is lucid , pain is easier to assess
Drug metabolism is different in older adults and should be taken into account when prescribing pharmacologic methods
Nonpharmacologic

39
Q

Pain Management:

ASSESSMENT

A
COLDSPA
pain is subjective cannot be measured
elimination of the cause
careful history
numeric pain scale
40
Q

Pain Management:

A

include nonpharmacalogic and pharmacologic

41
Q

Nonpharmacologic include:

A

Cognitive behavioral interventions
Physical agents
Electroanalgesia
Acupuncture

42
Q

Pharmacalogic include:

A

use of drugs
combo of narco and non-narco and adjuvant
analgesics (tolerable pain)
long term use= increase in dosage

43
Q

Nonnarcotic analgesics include

A

aspirin and NSAIDS ( Antipyretic and anti-inflammatory effect
Inhibition of COX-enzymes)
Acetaminophen : no an anti-inflammatory ( low temp/relieve pain)

44
Q

Opioid Analgesics

A

Group of medications with morphine-like actions
Morphine and codeine
Acute and chronic pain
Helps to give routinely before pain is severe