Ch.25&27: Renal and Urinary Part 2 Flashcards

1
Q

the tendency of biological systems to maintain relatively constant conditions in the internal environment while continuously interacting with and adjusting to changes originating within or outside the system… homeostasis actually involves continuous motion, adaptation, and change in response to environmental factors.

A

homeostasis

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2
Q

Acute kidney injury:

A

inability to efficiently/effectively remove waste from blood
develops rapidly
oliguria, azotemia, increased serum creatinine, and urea
fluid and electrolyte disorders
high mortality rate

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3
Q

AKI CLASSIFICATION:
sudden and severe drop in blood pressure (shock) or interruption of blood flow to the kidneys from severe injury or illness
Ischemia of nephrons
Decreased glomerular filtration rate (GFR)
Sharp decrease in urine output *

A

pre-renal

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4
Q

AKI CLASSIFICATION:
direct damage to the kidneys by inflammation, toxins, drugs, infection, or reduced blood supply
Glomerulonephritis

A

intra-renal

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5
Q

AKI CLASSIFICATION:
sudden obstruction of urine flow due to enlarged prostate, kidney stones, bladder tumor, or injury
SYMPTOMS: abdominal distention/ suprapubic tendernessRisk

A

post-renal

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6
Q

Disorders associated with pre-renal:

A
Cardiogenic shock
Heart failure
Myocardial infarction
Burns
Trauma
Renal artery obstruction
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7
Q

Risk factors

A

reduced grr, immobility, diabetes, diuretics, hypotension, dehydration, polypharmacy, CVD, infection

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8
Q

Management of acute kidney injury:

Nutritional concern

A

manage the potassium and sodium
adequate calorie intake
carbs/fat prevent ketosis

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9
Q

Nursing management of AKI:

A

monitor( VS, weight, lab, mental)
skin/color (appearance, edema, Jed, oral)
ausculate ( lungs/heart)
urine (output color, glucose, sediment, protein)

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10
Q

Kidney function
Progressive, irreversible loss
Mortality
High; Between 19-24% for those on dialysis
Major causes
#1: Diabetes; #2. Hypertension
Notable facts
*Mortality increases as the disease progresses
*CKD is more common than AKI
*CKD is often asymptomatic, underdiagnosed
*CKD = when GFR < 60 mL/min/1.73 m2 > 3 months
*ESRD is the last stage when GFR is <15.
*Transplant or dialysis are the only options to sustain life when ESRD is diagnosed

A

chronic Kidney Disease

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11
Q

Higher than normal levels of creatinineor urea in the blood
Blood or protein in the urine
Evidence of kidney damage in an MRI, CT scan, ultrasound or contrast X-ray
A family history of polycystic kidney disease

A

Stage 1 cKD

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12
Q

Higher than normal levels of creatinineor urea in the blood
Blood or protein in the urine
Evidence of kidney damage in an MRI, CT scan, ultrasound or contrast X-ray

A

Stage 2 CKD

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13
Q

Fatigue
Fluid retention, swelling of extremities and shortness of breath
Urination changes (foamy; dark orange, brown, tea-colored or red if it contains blood; and urinating more or less than normal)
Kidney pain felt in their back
Sleep problems due to muscle cramps or restless legs (Davita, 2017)

A

Stage 3 CKD

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14
Q

Fatigue
Fluid retention, swelling, shortness of breath
Urination changes (foamy; dark orange, brown, tea-colored or red if it contains blood; and urinating more or less than normal)
Kidney pain felt in their back
Sleep problems due to muscle cramps or restless legs
Nausea and/or vomiting
Taste changes such as a metallic taste in the mouth
Bad breath due to urea buildup in the blood
Loss of appetite
Difficulty in concentrating
Numbness or tingling in the toes or fingers (Davita, 2017)

A

Stage 4 cKD

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15
Q
Loss of appetite
Nausea or vomiting
Headaches
Being tired
Being unable to concentrate
Itching
Making little or no urine
Swelling, especially around the eyes and ankles
Muscle cramps
Tingling in hands or feet
Changes in skin color
Increased skin pigmentation
A

Stage 5 CKD

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16
Q

End stage renal disease treatment options

A

transplantation and dialysis

17
Q

May result with diminished urine output (oliguria, anuria), CKD
May be result of high potassium diet, drugs, blood transfusion, bleeding
Weakness, fatigue, nausea, tingling sensations, slow heartrate, weak pulse, cardiac arrest

A

hyperkalemia

18
Q

May result with excessive urine output (diuresis), sweating, vomiting, diarrhea, laxative overuse, certain medications, CKD
May be result of low potassium diet
Weakness, fatigue, abnormal heart rhythm, leg cramps, constipation, muscle weakness/spasms, tingling/numbness

A

hypokalemia

19
Q

low potassium foods include:

A

green beans, cabbage, eggplant, apples, cherries, celery, almond milk, rice , popcorm

20
Q

high potassium foods include:

A

avocados, potatoes, broccoli, brussel sprouts, bananas, cantaloupes, milk, oranges, milk, tomatoes

21
Q

results due to inability of GI tract to absorb calcium in absence of active vitamin D
If serum calcium level is low, parathyroid tells the bones to release calcium, which causes weakened bones

A

hypocalcemia

22
Q

may occur w/calcium and Vitamin D supplementation (hold Vit.D and replace binders w/non-calcium binders)
Hyperparathyroidism may require parathyroidectomy

A

hypercalcemia

23
Q

what food types are high in phosphorous

A

dairy products and dark colas

24
Q

Treatment for ESRD ANEMIA:

A

exogenous erythropoietin- titrated base on hemoglobin lab values-individualized dose (lowest)
IV/ subcut.
iron supplementation