ch 16 frontal lobe Flashcards

1
Q

anatomy

A

tissue anterior to central sulcus-motor, premotor, prefrontal

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2
Q

subdivisions

A

premotor and supplementary motor cortex, frontal and supplementary eye field

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3
Q

prefrontal cortex

A

area of the frontal lobe that receives input from the dorsomedial nucleus of thalamus
divisions: dorsolateral inferior, medial

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4
Q

motor area connections

A

projects to spinal motor neurons, cranial nerves that control the face, basal ganglia and red neuclues

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5
Q

premotor area connections

A

direct movement through coticospinal projections or motor projections, gets input from posterior parietal regions and dorsolateral prefrontal area

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6
Q

eye fields

A

receive from PG and superior colliculus

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7
Q

all premotor areas get projections from

A

dorsolateral prefrontal cortex–controls eye and limb movement

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8
Q

prefrontal area connections

A

end points of ventral (object recognition) and dorsal (spatial info) visual streams
dorsolateral prefrontal area-reciprocal connections with post pari and STS, extensive connections with cingulate cortex, basal ganglia, superior colliculus, input from dopamingeric cells in tegmentum

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9
Q

orbital frontal cortex connections

A

input from temporal lobe, amygdala, gustatory cortex, somatosensory cortex, olfactory cortex, dopamingeric cells in tegmentum, projects to hypothalamus and amygdala

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10
Q

theory of function for frontal lobe

A

planning and selecting, persistence and ignoring other stimuli, keeping track of whats been done–all executive functions, responds to internal external and context clues

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11
Q

theory of function for preomotor cortex

A
  • selects movements to be executed, choose behavior in response to external cues, increase in premotor activity when cues are associated with movement
  • supplementary motor role in selecting and directing internal motor sequences
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12
Q

prefrontal cortex functions

A
  • controls cognitive processes so that appropriate movements are selected at correct time
  • internal cues-temporal memory (what has just happened, can be related to things or movements), prefrontal cortex, dorsolateral
  • external cues-feedback about rewarding properties of stimuli, orbital frontal cortex learning by association, with memory damage use external cues to know how to act
  • context clues-orbital frontal, social interactions, detailed sensory info to inferior from temp lobe/affective cortex(amyg)
  • autonoetic awareness-self knowledge, continuous
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13
Q

motor___movements
premotor___movements
prefrontal____movements___

A

makes
picks
makes sure, done at right time and place

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14
Q

left asymmetry

A

language, encoding memories

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15
Q

right asymmetry

A

nonverbal movements, facial expressions, retrieving memories

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16
Q

differences in frontal lobe function

A

performs variety of functions, damage is unlikely to produce impairment to all functions-diff funct in diff sites

17
Q

disturbances of motor function

A
  • loss of fine movements, speed, and strength-after primary motor damage, loss of corticospinal projections to motor neurons, lose speed and strength contralaterally
  • loss of movement programming-damage to premotor or dorsolateral cortex, serially ordering behavior, supplementary motor cortex-improves rapidly expect in fingers, copying movements
  • changes in voluntary gaze-damage to frontal eye fields, hard to focus on specific things
  • corollary discharge or reafference-internal neural signal that movement will occur, damage disrupts discharge, voluntary movements require command and signal
  • speech-brocas(agrammatism-inability to use verbs, words based on cues), supplementary motor cortex(mute, returns if have unilateral lesion)
18
Q

convergent thinking

A

there is only 1 answer to the question

19
Q

divergent thinking

A

there are multiple answers to the questions, frontal lobe damage-cant do it

20
Q

behavioral spontaneity

A

spontaneous speech (can’t come up with many random words), decreased verbal fluency design fluency general behaviors, L orbitofrontal region, some have difficulty with doodles

21
Q

symptoms of frontal lobe lesions

A
increased perseveration, inability to make strategy(worse when doing new thing)-, 
loss of response inhibition(wisconsin card sorting task, stroop test)-cant change responses, flexibility in behavior broken
risk taking(iowa gambling task)-appears after damage to orbitofrontal cortex, cant follow instructions
self regulation decifits-loss of autonoetic awareness
loss of associative learning-inability to select from competing responses/learn from experience 
impaired social and sexual behaviors-phineas gage
22
Q

poor temporal memory

A

animal experiments show that frontal lobe plays role in temporal memory
area 46-provide internal representation of spatial info, delayed response test
medial regions-object recognition
dorsolateral cortex plays role in monitoring self generated responses
in humans-recency memory(order in which things have occured), prefrontal cortex

23
Q

changes in sexual and social behavior

A

pseudodepression/psychopathy, orbitofrontal lesions-abnormal sex behavior loss of cells that code facial expressions, dorsolateral reduce interest in sex,

24
Q

pseudodepression

A

change from involved and outgoing to quiet and remote after frontal lobe damage, L frontal lobe, loss of initiative, interest in sex, quiet

25
pseudopsychopathy
quiet polite strict to outspoken brash disrespectful, lesion to R frontal lobe, immature, no social graces, promiscuity
26
spatial deficit in frontal lobe?
to understand dorsolateral function must look at relation to posterior parietal, which has role in guiding visuomotor movements in space
27
frontal facial area damage symptoms
sensory and motor functions of face preserved after damage, pre and postcentral gyrus contralateral to lesion, in L-loss of verbal fluency, in R-loss of design fluency
28
schizophrenia
abnormality in mesocortical dopaminergic projection, decrease in blood flow to frontal lobes and atrophy
29
parkinson's disease
loss of dopamine cells in the substantia nigra that project to prefrontal cortex, lack facial expressions, spatial memory, delayed response
30
korsakoff's
alcohol induced damage to the dorsomedial thalamus and deficiency in frontal lobe catecholamines, metabolic disorder of CNS
31
drug addiction
impulsive behaviors, change in structurs of neurosn in orbitofrontal and medial prefrontal regions