ch 13 occipital lobe Flashcards

1
Q

anatomy

A

no clear subdivisions on lateral surface
medial surface-parietooccipital surface, calcarine sulcus (has primary visual cortex, separates upper and lower visual fields)
ventral surface-lingual gyrus (V2&VP), fusiform gyrus (V4)

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2
Q

color vision

A

V4, distributed throughout

detects movement, depth, and position (shadows)

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3
Q

primary visual cortex (V1) connections

A

input of lateral geniculate nucleus of thalamus, output to all other levels

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4
Q

secondary visual cortex (V2) connections

A

output to all other levels

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5
Q

connections after V2

A

output to parietal lobe-dorsal (where) stream
output to inferior temporal lobe-ventral (what) stream
output to superior temporal sulcus (STS)

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6
Q

visual pathways-dorsal stream

A

visual guidance of movements

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7
Q

visual pathways-ventral stream

A

object perception

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8
Q

visual pathways-STS

A

visuospatial functions and perception of certain movements

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9
Q

theory of function

A

vision begins in V1 and travels to more specialized zones, selective lesions up hierarchy produce specific deficits, lesions to V1 not aware of seeing

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10
Q

vision for action

A

see in order to act, parietal visual areas in dorsal stream (reach, duck, catch)

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11
Q

action for vision

A

move to see it all, visual scanning-move eyes and selective attention, saccades and pursuit problems

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12
Q

visual recognition

A

temporal lobes, object recognition

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13
Q

visual space

A

knowing where you are in space, parietal and temporal lobes, spatial location-egocentric (relation to self) and allocentric (relation to other objects)

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14
Q

visual attention

A

selective attention for specific visual input, parietal lobes guide movements and temporal lobes help in object recognition

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15
Q

dorsal stream is set of systems for online visual control of action

A

know because visual neurons in parietal corext only active when brain acts on visual info

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16
Q

STS stream characterized by

A

polysensory neurons, neurons responsive to both auditory and visual input or both visual and somatosensory input
originates from structures in parietal and temporal cortex

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17
Q

monocular blindness

A

destroy retina, optic nerve at bundles, loss of sight in 1 eye

18
Q

bitemporal hemianopia

A

loss of vision from both temporal fields, lesion to optic chiasm, peripheral vision on both sides lost

19
Q

nasal hemianopia

A

loss of vision to 1 nasal field, lesion of lateral chiasm

20
Q

homonymous hemianopia

A

blindness of entire visual field, results from complete cut of optic tract, lateral geniculate body, or area V1

21
Q

macular sparing

A

sparing of central or macular region of visual field, lesion to occipital lobe

22
Q

hemianopia, quadrantoanopia

A

complete loss of vision in 1/2 or 1/4 of fovea, lesion to occipital lobe

23
Q

scotoma

A

field defects, small blind spots, small occipital lobe lesions

24
Q

conclusions to be made from case studies

A

distinct syndromes of visual disturbance, some provide evidence for fundamental dissociation between dorsal and ventral streams, visual experience not unified, asymmetry in functions

25
Q

apperceptive agnosia

A

type of object agnosia, inability to perceive structure of objects
simultagnosia-inability to see multiple objects at a time
bilateral damage to lateral occipital lobe

26
Q

associative agnosia

A

object agnosia, perceive objects but not identify them, anterior temporal lobe lesions

27
Q

prosopagnosia

A

can’t recognize faces, facial features or expressions, or tell human from nonhuman face

28
Q

alexia, dyslexia

A

cant read, form of object agnosia-can’t construct wholes from parts, form of associative agnosia-word memory damaged/inaccessible, damage to left fusiform and lingual areas

29
Q

visual imagery

A

neural structures mediate perception and visualization not completely independent, R hemisphere usually does mental rotation, L hemisphere for image generation

30
Q

V1 features

A

complex laminar organization, appears anatomically homogeneous but can be heterogeneous when stained with cytochrome oxidase-blobs
lesions in V1 can still get to V2, but must function so brain can make sense of whats happening

31
Q

V2 features

A

can also be hererogeneous when stained with cytochrome oxidase, reveals stripes

32
Q

3 pathways from V1&V2

A

v1->v4-color aread
v1->v2->v5-middle temporal, motion
v1&v2->v3-shape of objects in motion, form perception

33
Q

V1 to temp lobe in ventral stream

A

knowing what an object is

34
Q

V1 to pari lobe in dorsal stream

A

controlling visual guidance of movements

35
Q

visual field organization

A

left half of each retina sends projections to R brain, R half of each retina sends projections to L brain

36
Q

infarct

A

dead tissue

37
Q

angioma

A

collection of abnormal blood vessels

38
Q

optic ataxia

A

deficit in visually guided hand movements

39
Q

prosopagnosia

A

facial recognition deficits

40
Q

visuospatial agnosia

A

can’t find way around, spatial perception/orientation