ch #10 mood disorders lecture notes Flashcards

1
Q

describe depression in infants

A

anaclitic depression (spitz)—infants raised in a clean but emotionally cold institutional environment or in severely dysfunctional families show depression like reactions

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2
Q

describe depression in preschoolers

A

may appear extremely somber and tearful, lacking exuberances; may display excessive clinging and whiny behaviour around mothers.

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3
Q

describe depression in school-age children

A

similar as preschool children, plus increasing irritability, disruptive behaviour and tantrums

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4
Q

describe depression in preteens

A

irritable, self blame, low self-esteem, persistent sadness, and social inhibition

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5
Q

discuss the prevalence of mood disorders and depression

A

-between 2% and 8% of children 4-18 experience MDD
-depression is rare among preschool children (1-2%)
-the increase during school age years due to social pressures, increase in cognitive abilities and self-awareness
-the sharp increase in adolescence may result from biological maturations at puberty interacting with developmental changes

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6
Q

describe onset, course and outcome of MDD/depression

A

-onset my be gradual or sudden
-age of onset: 13-15 yrs
-average duration of an episode: 8 months
-reoccurrence of other episodes is common: up to 70% chances of reoccurrence within 5 years
-30% will later develop bipolar disorder

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7
Q

describe the comorbidity associated with MDD

A

-90% of young people with depression have one or more other disorders
-50% have two or more other disorders
-depression+anxiety=73.8%–85% of cases anxiety comes first
-depression+behaviour problems=47.2%
-60% of adolescents with MDD have comorbid personality disorders, especially borderline personality disorder.
-dysthymia and substance use disorder

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8
Q

what are the differences between MDD and PDD?

A
  • milder symptoms (at least 2 instead of 5)
    -longer duration (instead of 2 weeks)
    -associated with poorer response to treatment
    -if PDD is presented with MDD it is called Double Depression
    -PDD develops earlier (11yrs)
  • average episode 2-5 years
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9
Q

what are some associated characteristics with mood disorders in regards to intellectual and academic functioning

A

-difficulty concentrating, loss of interest and slowness of thought and movement may affect academic functioning–lower scores on tests, poor teacher rating, and lower levels of grade attainment

-interference with academic performance, but not necessarily related to intellectual deficits–may have problems on tasks requiring attention, coordination and speed

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10
Q

what are some associated characteristics in regards to cognitive biases and distortions

A

-selective attentional biases to negative information (negative emotional expressions)

  • feelings of worthlessness, negative beliefs, attributions of failure, self-critical and automatic thoughts

-depressive ruminative style, pessimistic outlook and negative self-esteem

-negative thinking and faulty conclusions generalized across situations, hopelessness and suicidal ideation

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11
Q

what are some associated characteristics in regards to social, peer and family problems

A

social and peer problems:
-few close friendships, feelings of loneliness, and isolation
-social withdrawal and ineffective styles of coping in social situations

Family Problems:
-have more conflicted relationships with parents and siblings
-feel socially isolated from families and prefers to be alone (often to avoid conflict)
-be negative towards family members who may in turn be negative/harsh or dismissive towards them

-most youngsters with depression think about suicide and as many as one-third who think about it, attempt it–most common methods for those who complete suicide are firearms, hanging, suffocation, poisoning and overdose.

-worldwide the strongest risk factors are having a mood disorder and being a young female

-ages 13-14 are peak periods for a first suicide attempt by those with depression

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12
Q

what are some associated characteristics in regards to suicide

A

In Canada in 2016:
-5 children/youth out of 100000 die of suicide
-200 children/youth out of 100000 were hospitalized because of self injury

Depression is one of the strongest risk factor and warning signs for suicidality

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13
Q

what are some causes of depression?

A

-Biological factors (genes, brain)
-Environmental factors (family, stess)
-Psychological factors (cognitive errors)

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14
Q

describe the biological causes of mood disorders

A

-twin and other genetic studies suggest moderate genetic influence, with heritability estimates ranging from 30-45%

-children of parents with depression have about 3x risk of having depression

-what is inherited is likely a vulnerability to depression and anxiety, not one specific gene for depressive symptoms (with certain environmental stressors needed for this disorders to be expressed –> gene environment interaction

-abnormalities in the structure and function of several brain regions that regulate emotional functions

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15
Q

describe the environmental causes of mood disorders

A

the families of children with depression tend to be:
-more critical and punitive toward the depressed child than toward other children
-more likely to experience high levels of stress
-more likely to experience marital conflict
-more likely to experience lack of social support
-more likely to be depressed (especially postpardum depression) which negatively affects their parenting skills
-more likely to be characterized by insecure attachment styles

triggers for depression may involve:
-interpersonal stress and actual or perceived personal losses
-life changes
-violent family environment
-everyday life events/challenges

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16
Q

what are the psychological causes of mood disorder

A

-theory that explains how youth and adults with depression tend to have altered cognitions about themselves, the world around them and future (learned helplessness?)

-biases in self-referential processing have been interpreted as reflecting the presence of negative self-schemas in individuals with depression and these negative biases predict the onset of a new depressive episode

-attentional bias towards negative stimuli (dot probe task)

-INTERPRETATION: bias in interpretation of ambiguous stimuli (ambiguous words-dye/die or ambiguous faces)

-MEMORY: negative bias in encoding and retrieval, but also deficit in remembering details about positive events

-COGNITIVE CONTROL: all the processes that control the information in our working memory
-individuals with depression tend to struggle ignoring negative informations
-have issues removing negative memories from their working memory (ruminating)

-cognitive emotion regulation strategies , 1. rumination 2. reappraisal 3. distraction

17
Q

what is reappraisal

A

reinterpreting the meaning or interpretation of an emotion eliciting situation in order to modify the emotional experiences

18
Q

what are the most common interventions for depression

A

-psychosocial interventions (CBT, interpersonal therapy)
-medications
-family oriented interventions

19
Q

what is cognitive behavioural therapy

A

-aimed at addressing the behavioural and cognitive component of the disorders

ACTION PROGRAM:
A=always find something to do to feel better (target=behavioural)
C= catch the positive (build positive self-schema)
T= think about it as a problem to be solved (reframing)
I= inspect the situation
O= open yourself to the positive
N= never get stuck (identification of negative thoughts and use of cognitive restructuring)

20
Q

what is interpersonal psychotherapy for adolescent depression

A

-improving interpersonal communication
-addressing romantic relationship, separation from parents and peer relationships
-identification of social triggers and social supports

21
Q

what are some medications for depression?

A

-tricyclic antidepressants consistently fail to demonstrate any advantage over placebo in treating depression in youth (have serious cardiovascular side effects)

-SSRI’s (Prozac, zoloft) are the most commonly prescribed medications for treating childhood depression (side effects include suicidal thoughts and self harm and lack of information about long-term effects on developing brain)

-up to 60% of depressed youth respond to placebo

22
Q

what are some symptoms of bipolar disorder

A

restlessness, agitation, sleeplessness, pressured speech, flight of ideas, racing thoughts, sexual disinhibition, surges of energy, expansive grandiose beliefs

23
Q

what are the three subtypes of bipolar disorder

A
  • bipolar I disorder
    -bipolar II disorder
    -cyclothymic disorder
24
Q

describe how bipolar disorder presents in adolescence

A

with mania may present with atypical symptoms–volatile and erractic changes in mood, psychomotor agitation, and mental excitiation
-irritability, belligerence, mixed manic-depressive features occur more often than euphoria

-classic symptoms for children with mania include pressured speech, racing thoughts and flight of ideas

25
Q

what is the prevalence of BD

A

-lifetime estimates of BD range from 0.5 to 2.5% of youth 7-21 yrs old

-in youngsters milder bipolar II and cyclothymic disorder are more likely than bipolar I

-extremely rare in young children–rate increased after puberty

26
Q

what is the onset, course and outcome of BD

A

-60% of patients with BD have a first episode prior to age 19 (onset before age 10 is extremely rare)

-adolescents with mania typically have: psychotic symptoms, unstable moods, and severe deterioration in behaviour

-early onset and course is chronic and resistant to treatment (long term prognosis is poor)

27
Q

what are the biological causes of BD

A

-genes
-brain-imaging studies suggest mood fluctuations are related to abnormalities in areas of the brain related to: emotion regulation prefrontal and anterior cingulate cortex, hippocampus, amygdala, thalamus and basal ganglia

28
Q

what is the treatment for BD

A

-no cure

multimodal plan includes:
-monitoring symptoms closely
-educating the patient and the family
-matching treatments to individuals
-administering medication(lithium)
- addressing symptoms and related psychosocial impairments with psychotherapeutic interventions