Case 20- Speech and stroke Flashcards

1
Q

Difficulties faced by patients with language impairement

A
  • Difficulty exchanging information accurately with health professionals
  • Potential exclusion from shared decision making approaches
  • Loss of autonomy- by having to rely on others to communicate
  • Increased risk of adverse events in health care
  • Being treated as if they have a cognitive impairment when they don’t
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2
Q

Patient centred strategies to enhance communication with a patient who has a language impairement

A
  • Aim for a quiet, well-lit environment, avoiding distractions (TV, radio etc).
  • If the person normally wear glasses, hearing aid or dentures, make sure these are available.
  • Make sure the patient’s other needs are met before you begin (e.g. they are not hungry or in pain).
  • Choose the time of day when your patient communicates best.
  • Assume the person can hear and understand well, in spite of any difficulties responding, unless you learn otherwise.
  • Listen carefully as well as speaking – you can gain a lot of additional information from facial expression, gesture and eye contact
  • Allow extra time
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3
Q

FRAME model- highlights five areas of communication adjustment

A
  • F: familiarise yourself with how your patient communicates before starting the medical interaction
  • R: reduce rate
  • A: assist patient with communication
  • M: mix communication methods
  • E: engage patient to respect their autonomy
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4
Q

FRAME model- F

A
  • Ensure the patient has some way to express himself or herself.
  • Ask the patient about preferences for communication or existing strategies.
  • Assess how well the patient can understand what you say
  • Clarify the role of others present (family) and if the patient wishes them to be involved in helping with communication.
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5
Q

FRAME model- R (reduce rate)

A
  • Allow the patient sufficient time to process information and formulate responses.
  • Speak slowly and clearly in a normal tone of voice
  • Use short phrases and short chunks of information if patient has comprehension problems.
  • Stick to one topic at a time and emphasise key words
  • Have an attitude of patience and reassure patient it is ok to “take your time.”
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6
Q

FRAME model- A (assist patient with communication)

A
  • Be willing to “step forward” to help with communication; do not sit back and wait for the patient to fix communication breakdowns.
  • Confirm understanding and that you are communicating successfully with closed checking questions to confirm “yes” and “no”.
  • Don’t pretend to understand. If you’re having difficulty, be honest and tell your patient: “I’m sorry, I don’t understand – let’s try again.”
  • Be flexible to try different strategies until you find something that works. The same patient may need different strategies at different times.
  • Use the “least restrictive” accommodation (do not limit the patient to yes/no responses if they are able to communicate more than that).
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6
Q

FRAME model- A (assist patient with communication)

A
  • Be willing to “step forward” to help with communication; do not sit back and wait for the patient to fix communication breakdowns.
  • Confirm understanding and that you are communicating successfully with closed checking questions to confirm “yes” and “no”.
  • Don’t pretend to understand. If you’re having difficulty, be honest and tell your patient: “I’m sorry, I don’t understand – let’s try again.”
  • Be flexible to try different strategies until you find something that works. The same patient may need different strategies at different times.
  • Use the “least restrictive” accommodation (do not limit the patient to yes/no responses if they are able to communicate more than that).
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7
Q

FRAME model- M (mix communication methods)

A
  • Do not rely solely on speaking and listening; SHOW, don’t just TELL.
  • Use body language, gestures, pictures, writing, drawing to supplement spoken language.
  • Have communication aids (picture boards, alphabet boards, white boards, and pens/paper) readily available for patients; customize picture boards for the vocabulary and concepts you use in your setting.
  • Use these different modalities to help patients with comprehension problems understand what you are saying.
  • Encourage patients who have expressive impairments to use these different modalities to help them convey their messages.
  • Allow extra time for patients who use augmentative and alternative communication devices to enter messages into the device.
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8
Q

FRAME model- E (engage patients)

A
  • Speak directly with the patient; family members should help you communicate WITH the patient, but they should not communicate FOR the patient.
  • Do not “talk down” to patients—keep your tone of voice natural, respectful, and appropriate to the age of the patient.
  • Resist the urge to interrupt, finish sentences or offer words; ask the patient if it is OK to guess at what they are trying to communicate
  • Interact with the patient as you would other patients including social banter to build rapport, soliciting patient viewpoints, and providing full information and education.
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9
Q

Aphasia

A

A language disorder that results from damage to those areas of the brain that are responsible for language. It can affect understanding of spoken communication of written communication (reading), spoken production of language and written expression (writing) leaving other cognitive abilities and intellect intact. Incidence:
1) Stroke- one third are left with a communication disability

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10
Q

Types of aphasia- the Classical approach

A
  • Injury to the left hemisphere
  • Type of aphasia
  • Syndrome defined by characteristic features- fluency of speech, level of understanding, ability to repeat
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11
Q

Wernicke-Lichtheim-Geschwind model

A
  • Word meaning- distributed across cortex
  • Broca’s area- centre of motor world image
  • Output from Broca’s area to motor nuclei
  • Wernicke’s area- centre of acoustic word image
  • Input to Wernicke’s area from sub cortical regions
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12
Q

Types of aphasia

A
  • Broca’s aphasia
  • Wernicke’s aphasia
  • Damage to the arcuate fasciculus- conductive aphasia
  • Damage to Broca’s and Wernicke’s area- Global aphasia
  • Damage to the anterior superior frontal lobe of the language dominant hemisphere- transcortical motor aphasia
  • Damage to specific area of the temporal lobe- Transcortical sensory aphasia
  • Anomic aphasia
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13
Q

Symptoms of the different types of aphasia

A
  • Anomic aphasia- good repetition, good comprehension, fluent
  • Conduction aphasia- impaired repetition, good comprehension, fluent
  • Transcortical sensory aphasia- good repetition, impaired comprehension, fluent
  • Wernicke’s aphasia- impaired repetition, impaired comprehension, fluent
  • Transcortical motor aphasia- good repetition, good comprehension, non-fluent
  • Broca’s aphasia- impaired repetition, good comprehension, non-fluent
  • Mixed transcortical aphasia- good repetition, impaired comprehension, non-fluent
  • Global aphasia- impaired repetition, impaired comprehension
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14
Q

Broca’s aphasia

A
  • Non fluent agrammatic speech= simplified sentence, nouns>verbs
  • Halting, effortful, dysprosodic production
  • Impaired non-fluent repetition
  • Good functional comprehension but asyntactic comprehension on testing
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15
Q

Wernicke’s aphasia

A
  • Fluent pragmatic speech- normal sentence structure, empty content, either English jargon and/or neologisms
  • Repetition as spontaneous speech
  • Prominent comprehension difficulties
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16
Q

Anomic aphasia

A
  • Fluent grammatical speech
  • Poor word retrieval- impaired spoken naming, naming to definition and verbal fluency. Evidence of word finding difficulties in connected speech. ‘Tip of the Tongue’ feeling.
  • Production of words with similar meaning i.e. cat instead of dog. Or production of words with similar sounds like ‘cap’ instead of ‘cat.
  • Intact repetition
  • Intact comprehension
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17
Q

Importance of aphasia

A
Language and communication is essential for participating in everyday tasks. It is the foundation of our relationship with other people. People with aphasia:
• Loss of communicative competence
• Loss of family roles
• Loss of employment and societal roles
• Report a ‘loss of self’
• Isolation
• Loss of self-confidence and self esteem
• Depression and reduced quality of life
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18
Q

Speech- helping the person to understand

A
  • Reduce background noise/distractions
  • Keeping your language clear and simple
  • Using short phrases and sentences to communicate
  • Using all forms of communication to reinforce what you are saying
  • Give the person time to take in what you say and to respond
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19
Q

Speech- helping the person to communicate

A
  • Don’t interrupt- ask if the person wants help
  • Look as well as listen- you will get information from natural gestures, facial expression and body language
  • Encourage and accept all forms of communication
  • Asking careful questions- yes or no, forced alternatives
  • If your having difficulty understanding their communication, be honest and tell them to repeat
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20
Q

Causes of stroke

A

• Ischaemic (85% cases)
• Haemorrhagic- intracerebral (rupture of blood vessel in the brain)
- Subarachnoid (rupture of blood vessel in the subarachnoid space)

21
Q

Ischaemic causes of stroke

A

Due to occlusion of arteries of cerebral circulation
• Thrombus- Atherosclerosis, Prothrombotic clotting disorders (rarer)
• Embolism- Rupture of atherosclerotic plaque, Blood clot from the heart (due to atrial fibrillation)

22
Q

Causes of Haemorrhagic stroke

A

Due to focal collection of blood from a blood vessel rupture
• Intracerebral (blood vessel in the brain)- Hypertension
• Subarachnoid (bleeding into the subarachnoid space)- Aneurysm, Vascular malformation (rare)

23
Q

Other common risk factors for a stroke

A
  • Ischaemic heart disease
  • Peripheral arterial disease
  • Carotid atherosclerosis -> carotid stenosis
  • TIA
  • Vascular heart disease and heart failure
  • Clotting disorders
  • Atrial fibrillation
24
Q

Stroke definition

A

A clinical syndrome of presumed vascular origin characterised by rapidly developing signs or focal or global disturbance of cerebral functions which lasts longer than 24 hours or leads to death.

25
Q

TIA (transient ischaemic attack)- definition

A

Transient (less than 24 hours) neurological dysfunction caused by focal brain ischaemia without evidence of acute infarction. Tends to last less then an hour

26
Q

Oxford stroke classification

A
Based in whether it affects the anterior or pulmonary circulation
• Total anterior circulation syndrome
• Partial anterior circulation syndrome
• Lacuanar syndrome
• Posterior circulation syndrome
27
Q

Total anterior Circulation Syndrome (TACS)

A

All 3 of:
• Unilateral weakness (and/or sensory deficit) of at least 2 out of face, arm and leg
• Homonymous hemianopia (visual field defect)
• Higher cortical deficit (dysphasia or visuospatial loss)

28
Q

Partial anterior circulation syndrome (PACS)

A
  • 2 out of 3 of TACS components

* Or isolated higher cortical deficit

29
Q

Lacunar syndrome (LACS)

A
  • Pure motor (affecting >2/3 of face, arm and legs
  • Pure sensory (affecting >2/3 of face, arm and leg)
  • Mixed sensorimotor (affecting >2/3 of face, arm and leg)
30
Q

Posterior circulation syndrome (POCS)

A
  • Lesions affecting cerebellum, brainstem or occipital lobe i.e. Ipsilateral cranial nerve palsy and contralateral motor/sensory deficit
  • Bilateral sensory/motor deficit
  • Cerebellar dysfunction
  • Isolated homonymous hemianopia
31
Q

Neuroimaging

A

CT/MRI brain, this will primarily be used to rule out a bleed. Can help to exclude other lesions such as brain tumours which can mimic strokes.

32
Q

Common clinical investigation- stroke

A
  • Check blood pressure to look for hypertension
  • ECG to look for atrial fibrillation (AF)
  • Blood tests in order to check lipids, blood sugar, FBC and clotting
  • Carotid duplex ultrasound to look for atherosclerotic plaque if its an anterior circulation stroke
  • Echocardiogram to check for a clot in ventricles caused by AF
33
Q

Initial management- Strokes

A
  • Anyone with a suspected acute stroke is admitted to the hospital and a stroke ward
  • In cases of ischaemic stroke, the patient may need thrombolysis (clot busting treatment) or thrombectomy (surgical removal of clot)
  • Other treatment for infarction are antiplatelets i.e. aspirin
  • Haemorrhagic stroked may need neurosurgical interventions
34
Q

Secondary prevention Stroke- important to manage risk factors to try and prevent another stroke

A
  • Lifestyle modifications i.e. diet, exercise

* Pharmacological treatment i.e. anticoagulants (for atrial fibrillation), lipid modifications, antihypertensives

35
Q

Stroke rehabilitation

A

Important for the multi-disciplinary team to address the individual needs of the patient i.e. Speech, swallowing, mobility, continence, activities of daily living

36
Q

Stroke multi-disciplinary team

A
  • Occupational therapist
  • Physiotherapist
  • Speech and language therapist
  • Stroke specialist nurse
  • Radiologist
  • Stroke consultant
  • Social worker
37
Q

Anterior circulation of the brain

A
  • Internal carotid circulation
  • Aorta -> brachiocephalic (right only) -> common carotid (CCA) -> internal carotid (C4)
  • From both the left and right common carotid
  • Enters through the carotid canal
  • Supplies 70% of circulation
38
Q

Posterior circulation of the brain

A
  • Vertebral arteries
  • Aorta -> subclavian -> vertebral
  • Enters through the Foramen transversarium, travels up the cervical vertebra then enters through the Foramen magnum
  • Supplies 30% of circulation
39
Q

The circle of willis

A

The blood vessels are located in the subarachnoid space, between the arachnoid and pia mater. The posterior and anterior circulation join to form an arterial circle (Circle of Willis). The Circle of Willis is anterior and around the optic chiasm and the pituitary gland. The circle of Willis allows for compensation if there is an obstruction on one side. This works if there is a gradual loss of blood supply but not suddenly.

40
Q

Internal carotid artery branches

A
  • Post. Communicating artery (Pcom)- joins anterior to the posterior circulation
  • Ophthalmic artery (OA)- supplies the orbit and dorsum of the nose
  • Anterior choroidal artery (AChA)- supplies the deeper structures like the basal ganglia, optic tract, choroidal plexus, lateral ventricle and internal capsule
  • Middle Cerberal artery (MCA)
  • Anterior Cerebral Artery (ACA)- the two anterior cerebral arteries are joined by the Anterior communicating artery (Acom)
41
Q

Anterior cerebral artery

A
  • Moves through the longitudinal fissure, reflects back to go on the upper border of the corpus collosum
  • Supplies the medial surface of the cerebral hemisphere
  • Branch of the ICA
  • Clinical effects of an infarct- contralateral lower limb weakness and sensory loss. The ACA supplies the motor and sensory cortices that correspond to the legs
42
Q

Middle cerebral artery

A
  • Branch of the ICA
  • Separates into two branches, the upper and lower divisions
  • Supplies the majority of the lateral cerebrum
  • Passes through the lateral fissure of Sylvian
  • Clinical effect of an infarct- contralateral head, neck, trunk and arm weakness and sensory loss. MCA supplies the motor and sensory cortices corresponding to these regions
  • Aphasia-speech dysfunction: damage to Broca’s area causes expressive aphasia whilst damage to Wernicke’s area causes expressive aphasia. Normally left dominant
43
Q

Deep branches of the anterior circulation

A
  • Lenticulostriate or lateral striate arteries- branch of the MCA
  • Mediate striate or recurrent artery of Hubner- branch of the ACA
  • Anterior choroidal artery- branch of the ICA
  • Territory they cover- internal capsule= this region contains a high concentration of motor and sensory fibres projecting to and from the cortex
  • Territory they cover- Basal ganglia= subcortical nuclei involved with movement control, posture and muscle tone
  • Clinical effects of an infarct= due to the connections in this region a small stroke may have a dramatic clinical effect i.e. hemiparesis
44
Q

Posterior circulation vessels

A

• Paired Vertebral arteries= Ant.spinal artery (ASA), Post.spinal artery (PSA), Post.inf.Cerebellar artery (PICA)
• Basilar artery= Ant.Inf.Cerebellar artery (AICA), Sup.Cerebellar artery (SCA), Pontine Arteries
• Posterior Cerebral Artery (PCA)
The multiple branches come together to form the paired vertebral arteries which then form the Basilar artery which gives off branches. The Basilar artery then terminates into the two posterior Cerebral arteries

45
Q

Blood supply to the brain stem

A
  • Midbrain- PCA, SCA, Basilar
  • Pons- Basilar, AICA, SCA
  • Medulla- Vertebral, PICA, Basilar
46
Q

Blood supply to the Cerebellum

A
  • Superior- SCA

* Inferior- PICA, AICA

47
Q

Posterior cerebral artery

A
  • Branch of the basilar artery
  • Branches around the cerebral peduncle to the inferomedial temporal and occipital
  • Clinical effect of an infarct- Homonymous hemianopia due to damage to the visual cortex to the occipital lobe. Loss of the same half of the visual field in both eyes
48
Q

What are carotid and vertebral angiograms used to treat

A
  • Aneurysms
  • Vascular malformation i.e. fistula
  • Acute ischaemic stroke
49
Q

MoA of a carotid and vertebral angiogram

A

A catheter is inserted into the femoral artery and passed through the vessels until it reaches the internal carotid (ICA) or vertebral artery depending on the views required. The contrast is injected and the images taken.
• ICA- views the anterior circulation
• Vertebral- views the posterior circulation