Caries Risk Assessment Flashcards

1
Q

Dental Caries
(3)

A

 Multifactorial disease
 Bacterial infection, followed by acid attack
 Trends

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2
Q

 Multifactorial disease

A

 Can be altered by secondary
factors: f luoride, saliva f low,
etc.

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3
Q

 Bacterial infection, followed
by acid attack
(2)

A

 Remin/demin
 Controlled by multiple risk
factors

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4
Q

 Trends
 Decline:
 Increase:

A

fluoride
fermentable
carbohydrate

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5
Q

Caries occurs in areas where

A

plaque accumulates, undisturbed

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6
Q

 Each site has a unique
environment that influences
plaque composition, access by
(3)

A

dietary factors, saliva and anti-
caries factors

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7
Q

Site-specific Modifying Factors
(5)

A

 Pre/post-eruption fluoride exposure
 Patient’s oral hygiene practices
 Biofilm (composition varies person to person, site to
site)
 Saliva flow rate and composition
 Dietary habits

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8
Q

Changes in our Understanding
(3)

A

 Treatable vs controllable
 Fluoride results in slower
progression
 Caries process is dynamic. It can be
arrested or reversed.

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9
Q

Caries Prevention Strategies
(6)

A

Fluoride (highly effective in all forms)
Sealants (highly effective if applied correctly)
Salivary stimulation
Diet modification
Antimicrobial
Non- f luoride remineralizing strategies

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10
Q

Fluoride (highly effective in all forms)
(3)

A

 Water fluoridation
 Professionally applied
 Home delivery

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11
Q

Salivary stimulation
(1)

A

 Chewing gum

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12
Q

Diet modification
(2)

A

 Behavioral
 Protective food additives

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13
Q

Antimicrobial
(2)

A

 Non-specific
 Targeted

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14
Q

Casein phosphopeptide stabilized amorphous calcium
phosphate (Recaldent; CPP-ACP)
 Claim:
(2)

A

 CPP stabilize high concentrations of
calcium and phosphate ions, together
with f luoride ions, at the tooth
surface by binding to pellicle and
plaque
 The ions are supposedly freely bio-
available and can diffuse into enamel
subsurface lesions, thus promoting
re-mineralization

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15
Q

Evidence suggests that, under highly favorable
conditions,

A

Recaldent re-mineralizes artificial lesions
to a modest extent.

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16
Q

There is need for

A

independent, randomized,
controlled studies, under clinically relevant
conditions

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17
Q

Bottom line
 “The clinical benefits of CPP-ACP with and without
fluoride in paste form are not yet substantiated by
credible scientific evidence, and thus

A

it cannot be
recommended at this time. Topically applied fluoride
remains the standard for anti-caries effectiveness…”

18
Q

ADA Center for Evidence Based
Dentistry:
 *There is insufficient evidence from clinical trials
that use of agents containing

A

calcium and/or
phosphates with or without casein derivatives lowers
incidence of either coronal or root caries.

19
Q

Allergies
(2)

A

 These products are derived from milk products.
 Patients with dairy allergies should avoid using them.

20
Q

CAMBRA

A

CAries Management By Risk Assessment

21
Q

determining caries risk
(3)

A

Caries disease indicators
Caries risk factors
Caries protective factors

22
Q

Caries disease indicators
(3)

A

□Active caries
□Restorations within 3 years
□Areas of demineralization, including interproximal

23
Q

skipped
Caries risk factors
(9)

A

□Multiple multi-surface restorations
□Frequent snacking/sugared drinks
□Reduced saliva
□Exposed roots
□Visible, heavy plaque
□Deep pits and fissures
□Ortho
□Recreational drug use
□Physical or mental limitations

24
Q

skipped
Caries protective factors
(10)

A

□Regular dental care
□Regular professional fluoride treatments/varnish
□Sealants
□Fluoridated water
□Fluoridated toothpaste
□OTC fluoride mouthrinse (daily)
□Rx fluoride daily (5000 ppm)
□Xylitol gum
□Xylitol products
□Adequate saliva flow

25
Q

Caries risk
□Low
□Moderate
□High

A

(no disease indicators, <2 risk factors, has protective factors)
(no disease indicators, > 2 risk factors (but no caries)
(cavitated lesion(s)/disease indicators OR > 3 risk factors)

26
Q

Saliva
(4)

A

Cleansing
action
Re-
mineralization
and repair of
enamel
Dilution of
plaque acids
Antimicrobial
Properties

27
Q

Saliva testing for caries
 Salivary bacterial count
(3)

A

 Detect levels of Strep mutans and lactobacilli
 (Most) require 48-hour incubation period and follow-up
appointment to discuss results
 Chairside tests available (15 minute result)

28
Q

skipped
Saliva Flow/Buffering Capacity
(4)

A

 Insufficient salivary flow can lead to demineralization
and dental caries
 Influenced by time of day, diet, age, disease, and
medications
 Testing flow rate can aid in caries susceptibility and in
diagnosing salivary gland dysfunction
 Buffer capacity measures response to acid challenge

29
Q

skipped
Aids
(6)

A

 Saliva substitutes (Biotene products,
Oasis, Hydris)
 Prescription level fluoride (ie.
Prevident 5000 plus)
 Sugar free chewing gum (xylitol?)
 Baking soda
 Meticulous plaque control
 Professional fluoride ‘Homemade
recipe’ (1 cup water, lemon juice,
glycerine)

30
Q

skipped
Diet assessment
(4)

A

 Provide opportunity for patient to objectively
observe their dietary habits.
 Gain overall picture of types of food in patient’s diet
 To study food habits: ie. frequency and regularity of
foods eaten. (record frequency of cariogenic foods)
 Determine consistency of diet (fibrous vs sticky)

31
Q

Overall objectives:
(2)

A

 identify specific dietary behaviors that affect caries
risk (identify the high-risk behaviors)
 Enable clinician to open conversation regarding
dietary habits

32
Q

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Diet Diary
(3)

A

 Record 24 hr period –week-long
 Explain purpose
 Review form/app that you will send home with
patient

33
Q

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Review form/app that you will send home with
patient
(5)

A

 Encourage to fill out soon after eating
 Record everything eaten, including beverages and
in-between meal snacks.
 Encourage patient to be detailed
 Encourage patient to be truthful
 Consider having patient include when he/she
brushes/f losses

34
Q

Key areas to observe:
(2)

A

 Number of meals/snacks
 < 6/day (desired)
 > 6/day = increased risk

 Meal/Snack structure
 Structured (desirable)
 Unstructured/grazing = increased risk

35
Q

skipped
Considerations:
(5)

A

 Garnishes
 Sports drinks
 Chewing gum: sugarless or other?
 Canned fruit: packed in water or heavy syrup?
 Coffee/tea: with sugar?

36
Q

Sugared Beverages-what to observe
 Quantity
(3)

A

 < 12 ounces/day (desirable)
 12-20 ounces/day = moderate risk
 > 20 ounces/day = high risk

37
Q

Sugared Beverages-what to observe
 Timing
(3)

A

 With meals (desirable)
 With snacks = moderate risk
 Between meal snacks* = high risk

38
Q

Sugared Beverages-what to observe
 Frequency
(3)

A

 1 exposure/day = low risk
 2-3 exposures/day = moderate risk
 > 4 exposures/day* = high risk

39
Q

Sugared Beverages-what to observe
Length of exposure
(3)

A

 < 15 minutes = (desirable)
 15-30 minutes = moderate
 > 30 minutes = high

40
Q

Sugared Beverages-what to observe
Drinking style
(3)

A

 Straw = (desirable)
 Open container = moderate
 Swishing around in mouth = high

41
Q

skipped
Strategies for making Recommendations
(5)

A

 Delivery matters—and will improve pt’sreceptiveness
 Determine pt’sunderstanding of diet/disease
 Determine pt’smotivation
 Provide how-to advice
 Engage the patient to increase compliance

42
Q

skipped
 Provide how-to advice
(2)

A

 Include how-to advice; strategies to achieve outcome
 Provide educational resources