Cardiovascular System Flashcards

1
Q

Describe the conduction system?

A
  1. The SA node generates the electrical impulse and fires it through the atria walls causing them to contract.
  2. The AV nose collects the impulse and delays it for 0.1 seconds to allow the atria to stop contracting.
  3. Bundle of his splits the impulse into two ready to be distributed to each separate ventricle
  4. The bundle branches carry the impulse to the base of each ventricle.
  5. The purkyne fibres the distribute the impulse through the ventricle causing them to contract.
  6. Once the electrical impulse journey is complete the atria and ventricle relax and the heart re-fills with blood.
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2
Q

What is the diastole phase?

A

This is the relaxation phase where the cardiac muscle fills with blood, firstly the atria then the ventricles

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3
Q

What is the systole phase?

A

This is the contraction phase of the cardiac muscle, firstly the atria then the ventricles

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4
Q

What is the cardiac cycle?

A

This is the process of cardiac muscle contraction and the movement of blood through its chambers.

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5
Q

Describe the cardiac cycle?

A
  1. Diastole phase:
  • the atria and ventricles relax, they expand drawing blood into the atria
  • the pressure in the atria increases opening the AV valves
  • blood passively enters the ventricles
  • SL valves are closed to prevent blood from leaving the heart
  1. Atrial systole phase:
  • the atria contracts forcing remaining blood into the ventricles
  1. Ventricular systole:
  • the ventricles contract increasing the pressure closing the AV valves to prevent back flow into the atria
  • SL valves are forced open as blood is ejected from the ventricles into the aorta and pulmonary artery.
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6
Q

What are the components of 3-2-1?

A

3-neutral control:

  • chemoreceptors- (located in the blood)- inform the CCC of chemical changes in blood stream such as increased levels of CO2 and lactic acid
  • Proprioceptors- (located in muscles,tendons & ligaments)- informs the CCC of motor activity

-baroreceptors- (located in blood vessel walls)-inform the CCC of increased blood pressure

2-intrinsic control

-temperature changes affect the viscosity and thickness of the blood and speed of nerve impulses

  • venous return changes will affect the stretch the of the ventricular walls, force of ventricular contraction= stroke volume

1- Hormonal control

  • adrenaline and noradrenaline are released from the adrenal gland increasing the force of ventricular contraction (SV) and distribution of electrical activity through out the heart therefore the HR
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7
Q

Based on the information provided how does the CCC cause either increase or decrease heart rate?

A

If an increase in heart rate is required the sympathetic nervous system is actioned, releasing adrenaline/ noradrenaline and sending stimulation to to the SA node via the accelerator

If a decrease in heart rate is required the parasympathetic nervousness system is actioned to inhibit these effects via the vagus nerve, lowering the heart rate.

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8
Q

What is the sympathetic nervous system?

A

Part of the autonomic nervous system responsable for increasing HR, specifically during exercise

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9
Q

What is the parasympathetic nervous system?

A

Part of the autonomic nervous system responsible decreasing HR, specifically during recovery.

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10
Q

What is the autonomic nervous system (ANS) responsibility?

A

ANS is responsible for regulating HR and determines the firing rate of the SA node

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11
Q

What is the cardiac control centre (CCC) responsable for?

A

The CCC receives information from the sensory nerves and sends direction through the motor neurones to change HR

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12
Q

What is vascular shunt?

A

It’s the redistribution of cardiac output through our the body during exercise which causes an increases percentage of blood flow to the working muscles.

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13
Q

Where do arterioles lead?

A

They lead to capillary beds which serve to bring the blood in close contact with organ and muscles cells.

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14
Q

What is the blood flow to the capillary beds controlled by?

A
  • the pre-capillary sphincters
  • they constrict= limits blood flow to blood
  • they dilate= maximises blood flow to capillary bed
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15
Q

How do the pre capillary sphincters construct or dilate?

A

This is done increased/ decreased stimulation from the sympathetic nervous system

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16
Q

What is the responsibility of the vasomotor control centre?

A

It controls the redistribution of blood

17
Q

Describe what happens to vascular shunt during exercise?

A

Pre capillary sphincters:

  • dilate @ the working muscles- this is due to decreased stimulation from the sympathetic nervous system.
  • constrict @ the vital organs- this is because of an increased stimulation from the sympathetic nervous system- decreasing HR

-arterioles:

@ working muscles they vaso dilate- because of decreased stimulation front the sympathetic nervous system- increasing

@ vital organs- they vasoconstrict- because of increased stimulation from the sympathetic nervous
system- decreasing cardiac output

18
Q

What is frank sterling’s mechanism?

A

Increased venous return ( return of blood to the heart) causes an increased stroke volume

19
Q

What is the explanation of frank Sterling mechanism?

A

-increased venous return= increased amount of deoxygenated blood to the heart

-causing the atrial and ventricular walls to contract

  • this increases the force of the ventricular contractions meaning an increased volume of blood is being ejected front the ventricle per beat= stroke volume.
20
Q

Why does frank starling mechanism cause an increase in HR?

A

Because the SA node impulses increase due to the atrial and ventricular walls stretching which causes an increase in HR

21
Q

What are the mechanics of venous return?

A
  • pocket valves- one way valves which prevent the back flow of blood
  • smooth muscle- the layer of smooth muscle in the vein wall which venoconstricts to create venomotor tone which aids the movement of blood
  • gravity- blood from the upper body is helped to be returned by gravity
  • muscle pump- during exercise skeletal muscle contract compressing the veins located between them, squeezing the blood back to the heart

-respiratory pump- during inspiration and expiration a pressure difference between the thoracic and abdominal cavity is created squeezing blood back to the heart- as exercise increases respiratory rate, respiratory pump is maximised.

22
Q

What is the explanation for the graphs for HR & Cardiac output @ sub -max intensity?

A
  1. An increase takes place because of the production of Adrenaline= anticipatory rise
  2. Sudden increase= heart need to beat more to get more oxygen to the working muscles
  3. Platue- reached steady state meaning oxygen supply= oxygen demand
  4. Sharp/ sudden decrease= heart beats less as muscles aren’t working so they don’t need large amounts of oxygen
  5. HR is higher than the rest king rate because active recovery is taking place as oxygen debt is being payed off
23
Q

What is the explanation for cardiac out put/ HR @ maximal intensity?

A

1,2,4,5= the same as sun max

3= working towards maximal intensity as heart rate won’t increase till exercise is complete

24
Q

What is the explanation for stroke volume at sub maximal intensity?

A

It’s stops increasing because there’s no more time for the heart to fill up with blood and eject more blood

25
Q

What are slow oxidative muscle fibres?

A

A type of muscle fibre rich in mitochondria, myoglobin and capillaries which produces a small amount of force over a long time.

26
Q

What are fast glycolytic muscle fibres?

A

A type of muscle fibre rich in phosphocreatine (energy compound stored in muscle cells and used as fuel) which produces maximal force over a short period of time