Cardiovascular Flashcards

1
Q

risk of stroke in non-rheumatic AF

A

CHADS2 (risk of stroke in non-rheumatic AF)

Congestive heart failure
Hypertension
Age ≥ 75
Diabetes mellitus
Prior stroke/TIA/thromboembolism

6 gives it 18.2% chance of stroke

0 = low risk; none or aspirin daily
1 = moderate; aspirin daily or warfarin (raise INR to 2.0-3.0), depending on patient preference
2+ = moderate/high; warfarin, raise INR to 2.0-3.0 unless contraindicated

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

causes of secondary hypertension

A

CHAPS (causes of secondary hypertension)

Cushing’s syndrome
Hyperaldosteronism (Conn’s)
Aorta coarctation
Phaeochromocytoma
Stenosis of renal arteries

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

sequence of elevated enzymes after MI

A

C-AST-LE “castle” (sequence of elevated enzymes after MI)

CK-MB first
AST second
LDH third
Also: can use the last ‘E’ for ESR.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Beck’s triad for cardiac tamponade

A

D3 (Beck’s triad for cardiac tamponade)

Distant heart sounds
Distended jugular veins
Decreased arterial pressure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

signs of cor pulmonale

A

Please Read His Text (signs of cor pulmonale)

Peripheral oedema
Raised JVP
Hepatomegaly
Tricuspid incompetence

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

ECG signs in RH strain or acute cor pulmonale, eg. large PE

A

S1Q3T3 (ECG signs in RH strain or acute cor pulmonale, eg. large PE)

Large S wave in Lead 1
Large Q wave in Lead 3
Inverted T wave in Lead 3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

causes of cor pulmonale

A

PuLmonary HTN (causes of cor pulmonale)

Pulmonary vascular disease
• Primary pulmonary hypertension
• Pulmonary embolism ( Acute massive PE or Chronic recurrent PE )
• Pulmonary vasculitis
• Sickle cell anemia
• ARDS
• Parasite infestation

Lung disease
• COPD
• Asthma ( Severe, Chronic)
• Bronchiectasis
• Cystic fibrosis
• Interstitial lung disease
• Loss of lung tissue following lung trauma or surgery
• Sarcoidosis
• Pneumoconiosis

Hypoventilation
• Obstructive sleep apnea
• Cerebrovascular disease
• Adenoids enlargements in children

Thoracic abnormality
• Kyphosis
• Scoliosis

Neuromuscular disease
• Polioymyelitis
• Motor neurone disease
• Myasthenia gravis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

JVP vs carotids

A

POLICE (JVP vs carotids)

Palpable (JVP isn’t)
Occludable
Location between 2 heads of SCM
Inspiration (lowers JVP)
Contours (double bump)
Erect (affects position of JVP)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

features of apex beat

A

PLS-AD-TT “please add me T_T” (features of apex beat)

Presence
Location
Size

Amplitude
Duration

Type
Thrill

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

causes of absence of apex beat

A

DR POPE (causes of absence of apex beat)

Physiological:
Dextrocardia
Apex behind a Rib (turn patient to left lateral positin)

Pathological:
Pericardial effusion
Obesity and thick chest wall
Pleural effusion (left sided)
Empyema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

treatment of ACS

A

MONA LISA (treatment of ACS)

M- morphine
O- oxygen
N- nitrates
A- aspirin

L – loop diuretic
I – IV access – for bloods, for IV GTN etc
S – streptokinase (thrombolysis) / PCI - if STEMI
A – antiplatelets (e.g. clopidogrel)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

secondary prevention in ACS

A

COBRA-A (secondary prevention in ACS)

C – Clopidogrel – antiplatelets
O – Omacar – Omega 3
B – Bisoprolol – β-blocker
R – Ramipril – ACE-i
A – Aspirin

A – Atorvastatin – very potent statin!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Tetralogy of Fallot

A

PRAVeen has heart problems (Tetralogy of Fallot)

Pulmonary infundibular stenosis
Right ventricular hypertrophy
Overriding Aorta
VSD

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Angina management through lifestyle alterations

A

Angina management through lifestyle alterations

Mnemonic: SLEW
S Smoking cessation
L Low-fat diet
E Exercise
W Weight loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Management of acute unstable angina

A

Management of acute unstable angina
Mnemonic: 2 As and BALI
A Admit, bed rest, high-flow oxygen
A Analgesia
A Aspirin and clopidogrel

B Beta blockers
A Angiography with or without angioplasty/CABG if symptoms fail to improve
L Low molecular weight heparin (LMWH)
I Infusion of nitrates

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Angina presentation

A

Angina presentation
Mnemonic: SCAR
S Sudden central pain, ʻtearingʼ in nature, may radiate to the back
C Coronary artery occlusion can lead to chest pain, MI or angina pectoris/Carotid obstruction can lead to hemiparesis, dysphasia or blackouts
A Anterior spinal artery can be affected leading to paraplegia
R Renal artery can be affected leading to anuria or renal failure

17
Q

Cardiac arrest management, Advanced Life Support (ALS)

A

Cardiac arrest management, Advanced Life Support (ALS)

Mnemonic: CDE (with A after every step)
C Cardiac monitor and defibrillator should be attached to the patient
A Assess rhythm and pulse
D Defibrillate x 3 if VF or pulseless VT, CPR for 1 min
A Assess rhythm and pulse
E EMD (no cardiac output despite ECG showing electrical activity) or asystole warrants CPR for 3 min
A Assess rhythm and pulse

18
Q

strong risk factors for atherosclerosis

A

CD MASH up of songs (strong risk factors for atherosclerosis)

Cholesterol (by far the most)
Diabetes mellitus

Male
Age
Smoking
Hypertension

19
Q

less strong risk factors for atherosclerosis

A

FLOSS your teeth for better health (less strong risk factors for atherosclerosis)

Family history
Low birth weight
Obesity
Sedentary
Socioeconomic status