Cardio-respiratory system Flashcards
Summarise the functions of three structural components of the
cardiovascular system.
Atria-contract to push blood into
ventricles
* Ventricles- contract to push blood
into blood vessels
* Valves –prevent back flow of blood
* SA node – spreads impulse across
atria/allows electrical conductivity to next stage of conduction
* Arteries-distribute blood round the
body/ thick muscular walls, elastic fibres so withstand HIGH BP + stretch when blood passes through
* Capillaries – allow gas exchange
* Veins – return blood to the heart, thick muscular walls
* Aorta – pumps blood away from
heart
* Bicuspid/Tricuspid valves – regulates
flow/prevents back flow
* Pulmonary Vein-carries blood from
lung to heart
* Pulmonary Artery-carries blood from
heart to lungs
* Red blood cells - carry oxygen
0 Examine the chronic adaptions to the cardiovascular system experienced after
a sustained period of endurance training
Increase in maximal cardiac output (AO1)
* Enlarged heart / hypertrophy (AO1)
* An increase in blood volume (AO1)
* Increased stroke volume (increased LVEDV) (AO1)
Application to activity for example:
* Increase in capillary density/increased capillarisation of heart allows better gas exchange (AO3)
* Increased red blood cells/increased Hb allow better
oxygen carrying capacity (AO3)
* Increase gas exchange in the blood allows faster delivery of
oxygen (AO3)
* Decrease resting heart rate (bradycardia) (AO3)
Outline the mechanical process of inspiration at rest
- contraction of intercoastal muscles
- movement of ribcage up and out
- contraction of the diapgragm/flattening
- expands vol of throacic cavity
- pressure inside lungs decreases and is lower than atmospheric pressure
- air moves into lungs as gasses diffuse from high to low Pa
Describe how oxygen is transferred from alveoli to muscles at rest
- At lungs diffusion of O2 into capillaries from alveoli
- Diffusion occurs from high to low conc down conc gradient (passive)
- O2 binds to HBo carried on RBC’s
- Transport of O2 via cardiovas ss to muscles
- O2 unloaded into muscle via diffusion from high to low conc/pp02
Summarise how O2 delivery to working muscles increases during excercise
- Increased ventiliation/TV
- Increased diffusion gradient of lungs/muscles
- Increased Cardiac output/SV/HR
- Increased BP
- Vasodilation to increase BF to muscles
- Vasoconstriction of blood vessels in inactive areas Via Vascular shunting
Summarize functional adaptations to the heart as a result of aerobic training
- Increase in end diastolic vol
- Increased SV
- Increased Cardiac output when excercising
- Increased strength/force of ventricular contractions
- Bradycardia/ low resting HR
- same exercise aft aerobic training will have a lower HR
- Increase HRR
- faster recovery (HR returning to RHR)
Summarise functions of 3 charactertics of resp ss
- Nasal cavity: allows air to be inhaled
- Trachea= air flow to bronchi
- Bronchus= pathway to lungs
- Bronchiole= pathway to alevoli
- Alevoli= gas exchange of CO2 and O2
- Pulmonary capillary= allows gas exchange
Define pp02/partial pressre
- partial pressure is increased pressure that gas exertes in mixture of gases
Explain the role of pressure gradients
- Air moves from High to Low areas of pressure
- Pressure gradient allows air from outside body to diffuse inti lungs during inspiration
- Diaghrapm conctracts reducing pressure inside the lung/throacic cavity
- Diaphragm releaxes increasing pressure inside lungs- expiration
- increased pressure inside lungs forces air out
define Vo2 max
- max volume at which O2 used in 1 min
Vital capacity defintion
- VC= IRV + TV+ ERV
- max vol fully expired aft max inspiration
Define SV and cardiac output
- SV= vol of blood pumped out of heart durining 1 contraction
- CO= Vol of blood pumped out by heart in 1 min
outline 4 changes in resp value during excercise
- TV= increases
- IRV= decrease
- ERV= decrease
- VC= decrease
- Total lung capacity= decrease
explain how unhealthy lifestyle affects CV ss
- lack of regular excercise= weakens lungs, resp muscles + heart weaken, inhibits O2 delivery to muscles
- cigg smoking damages lung tissue/alveli/incrwases risk of cancer
- increased BP/HR as arteries constricted by nictotine
- imprper diet e.g high choleestrol/sodium increase BP/risk of CHD etc
- Obesity increases energy cost of moving, more stress on heart lungs, joints
- Drugs + sleep + stress + sedentary lifestyle
Karovene formula
- THR = (HRR x % HR) + RHR